Pages that link to "Q39241268"

The following pages link to Involvement of ATP-sensitive potassium (K(ATP)) channels in the loss of beta-cell function induced by human islet amyloid polypeptide. (Q39241268):

Displaying 12 items.

• On the Environmental Factors Affecting the Structural and Cytotoxic Properties of IAPP Peptides (Q27022467) (← links)
• Chaperones ameliorate beta cell dysfunction associated with human islet amyloid polypeptide overexpression (Q28540631) (← links)
• A gut-brain neural circuit controlled by intestinal gluconeogenesis is crucial in metabolic health (Q35040550) (← links)
• Stress and the inflammatory process: a major cause of pancreatic cell death in type 2 diabetes. (Q37568237) (← links)
• Does the small tyrosine kinase inhibitor Imatinib mesylate counteract diabetes by affecting pancreatic islet amyloidosis and fibrosis? (Q38045474) (← links)
• Intracellular and extracellular adenosine triphosphate in regulation of insulin secretion from pancreatic β cells (β). (Q38153423) (← links)
• Amyloid-induced β-cell dysfunction and islet inflammation are ameliorated by 4-phenylbutyrate (PBA) treatment (Q47933235) (← links)
• BACE2 suppression promotes β-cell survival and function in a model of type 2 diabetes induced by human islet amyloid polypeptide overexpression. (Q48247783) (← links)
• Conformational switching within dynamic oligomers underpins toxic gain-of-function by diabetes-associated amyloid. (Q52608994) (← links)
• Pancreatic β cells overexpressing hIAPP impaired mitophagy and unbalanced mitochondrial dynamics. (Q53821054) (← links)
• Membranes as modulators of amyloid protein misfolding and target of toxicity (Q57636158) (← links)
• A Novel Pentapeptide Inhibitor Reduces Amyloid Deposit Formation by Direct Interaction with hIAPP (Q64242160) (← links)