Pages that link to "Q38520897"

The following pages link to The neuropsychology of Down syndrome: evidence for hippocampal dysfunction (Q38520897):

Displaying 50 items.

• The development of associate learning in school age children (Q21131993) (← links)
• Gene-dosage effects in Down syndrome and trisomic mouse models (Q21194863) (← links)
• Down syndrome: searching for the genetic culprits (Q22241596) (← links)
• On the promise of pharmacotherapies targeted at cognitive and neurodegenerative components of Down syndrome (Q22241936) (← links)
• Systematic review of cognitive development across childhood in Down syndrome: implications for treatment interventions (Q22242622) (← links)
• Prospects for Improving Brain Function in Individuals with Down Syndrome (Q22252566) (← links)
• Emerging Pharmacotherapies for Neurodevelopmental Disorders (Q24619170) (← links)
• The functional nature of synaptic circuitry is altered in area CA3 of the hippocampus in a mouse model of Down's syndrome (Q24679420) (← links)
• DYRK1A, a Dosage-Sensitive Gene Involved in Neurodevelopmental Disorders, Is a Target for Drug Development in Down Syndrome (Q26745413) (← links)
• The importance of understanding individual differences in Down syndrome (Q26750601) (← links)
• Building an adaptive brain across development: targets for neurorehabilitation must begin in infancy (Q26781609) (← links)
• Effects of neonatal neural progenitor cell implantation on adult neuroanatomy and cognition in the Ts65Dn model of Down syndrome (Q27303804) (← links)
• Hippocampal circuit dysfunction in the Tc1 mouse model of Down syndrome (Q27312111) (← links)
• A critical literature review of the effectiveness of various instruments in the diagnosis of dementia in adults with intellectual disabilities (Q28067327) (← links)
• Chromosome 21 and down syndrome: from genomics to pathophysiology (Q28290257) (← links)
• Characterization of PTZ-induced seizure susceptibility in a down syndrome mouse model that overexpresses CSTB (Q28478194) (← links)
• The LonDownS adult cognitive assessment to study cognitive abilities and decline in Down syndrome (Q28585473) (← links)
• Violence: heightened brain attentional network response is selectively muted in Down syndrome (Q28647773) (← links)
• Possible functional links among brain- and skull-related genes selected in modern humans (Q28648399) (← links)
• The NIH Toolbox Cognitive Battery for intellectual disabilities: three preliminary studies and future directions (Q29109561) (← links)
• Comprehensive behavioral phenotyping of Ts65Dn mouse model of Down Syndrome: Activation of β1-adrenergic receptor by xamoterol as a potential cognitive enhancer (Q30459302) (← links)
• Verbal short-term memory deficits in Down syndrome: phonological, semantic, or both? (Q30473695) (← links)
• The use of mouse models to understand and improve cognitive deficits in Down syndrome (Q30474023) (← links)
• Brain plasticity and disease: a matter of inhibition (Q30474810) (← links)
• Behavioral validation of the Ts65Dn mouse model for Down syndrome of a genetic background free of the retinal degeneration mutation Pde6b(rd1). (Q30477664) (← links)
• A mouse model of Down syndrome trisomic for all human chromosome 21 syntenic regions (Q30495192) (← links)
• Effects of individual segmental trisomies of human chromosome 21 syntenic regions on hippocampal long-term potentiation and cognitive behaviors in mice. (Q30497997) (← links)
• Lithium rescues synaptic plasticity and memory in Down syndrome mice (Q30530902) (← links)
• Trisomy for the Down syndrome 'critical region' is necessary but not sufficient for brain phenotypes of trisomic mice. (Q31104439) (← links)
• Impairments in motor coordination without major changes in cerebellar plasticity in the Tc1 mouse model of Down syndrome (Q33404261) (← links)
• Hedgehog agonist therapy corrects structural and cognitive deficits in a Down syndrome mouse model (Q33552991) (← links)
• Molecular basis of pharmacotherapies for cognition in Down syndrome (Q33625613) (← links)
• Visuo-spatial ability in individuals with Down syndrome: is it really a strength? (Q33699171) (← links)
• The mouse model of Down syndrome Ts65Dn presents visual deficits as assessed by pattern visual evoked potentials (Q33867210) (← links)
• Remembering things without context: development matters (Q33934650) (← links)
• Developmentally altered inhibition in Ts65Dn, a mouse model of Down syndrome (Q33966522) (← links)
• Brain phenotype of transgenic mice overexpressing cystathionine β-synthase (Q34130338) (← links)
• Abnormal MicroRNA Expression in Ts65Dn Hippocampus and Whole Blood: Contributions to Down Syndrome Phenotypes (Q34229190) (← links)
• Over-expression of RCAN1 causes Down syndrome-like hippocampal deficits that alter learning and memory. (Q34268889) (← links)
• Genotype-phenotype correlations in Down syndrome identified by array CGH in 30 cases of partial trisomy and partial monosomy chromosome 21 (Q34325983) (← links)
• Defective cerebellar response to mitogenic Hedgehog signaling in Down [corrected] syndrome mice (Q34335155) (← links)
• Increased male reproductive success in Ts65Dn “Down syndrome” mice (Q34407263) (← links)
• Acute Exercise Improves Prefrontal Cortex but not Hippocampal Function in Healthy Adults (Q34502126) (← links)
• Development and validation of the Arizona Cognitive Test Battery for Down syndrome (Q34517102) (← links)
• Neuropsychological components of intellectual disability: the contributions of immediate, working, and associative memory (Q34939918) (← links)
• Unravelling genes and pathways implicated in working memory of schizophrenia in Han Chinese (Q35016729) (← links)
• Safety and efficacy of rivastigmine in adolescents with Down syndrome: a preliminary 20-week, open-label study (Q35087290) (← links)
• Allocentric spatial learning and memory deficits in Down syndrome (Q35087311) (← links)
• Down's syndrome: a genetic disorder in biobehavioral perspective (Q35204303) (← links)
• Chronic Treatment with a Promnesiant GABA-A α5-Selective Inverse Agonist Increases Immediate Early Genes Expression during Memory Processing in Mice and Rectifies Their Expression Levels in a Down Syndrome Mouse Model (Q35418044) (← links)