Pages that link to "Q37965921"

The following pages link to Immunological mechanisms of contact hypersensitivity in mice (Q37965921):

Displaying 47 items.

• Toluene diisocyanate (TDI) disposition and co-localization of immune cells in hair follicles (Q23909608) (← links)
• Hapten-induced contact hypersensitivity, autoimmune reactions, and tumor regression: plausibility of mediating antitumor immunity (Q28387629) (← links)
• Anti-inflammatory potential of Capparis spinosa L. in vivo in mice through inhibition of cell infiltration and cytokine gene expression (Q28817812) (← links)
• Basophils are required for the induction of Th2 immunity to haptens and peptide antigens. (Q30539528) (← links)
• Enhanced scratching elicited by a pruritogen and an algogen in a mouse model of contact hypersensitivity (Q34067012) (← links)
• AHAPS-functionalized silica nanoparticles do not modulate allergic contact dermatitis in mice (Q34254990) (← links)
• Suppression of antigen-specific adaptive immunity by IL-37 via induction of tolerogenic dendritic cells (Q34408962) (← links)
• The role of neutrophils and G-CSF in DNFB-induced contact hypersensitivity in mice (Q34454707) (← links)
• Sphingosine 1-phosphate modulates antigen capture by murine Langerhans cells via the S1P2 receptor subtype (Q34474499) (← links)
• Afadin requirement for cytokine expressions in keratinocytes during chemically induced inflammation in mice (Q34507244) (← links)
• Contact hypersensitivity to oxazolone provokes vulvar mechanical hyperalgesia in mice (Q35036636) (← links)
• Oxazolone-induced contact hypersensitivity reduces lymphatic drainage but enhances the induction of adaptive immunity. (Q35184089) (← links)
• The NF-κB regulator Bcl-3 modulates inflammation during contact hypersensitivity reactions in radioresistant cells (Q35609977) (← links)
• CXCR3 chemokine receptor signaling mediates itch in experimental allergic contact dermatitis (Q35985416) (← links)
• Common clonal origin of central and resident memory T cells following skin immunization (Q36246184) (← links)
• Dermal γδ T Cells Do Not Freely Re-Circulate Out of Skin and Produce IL-17 to Promote Neutrophil Infiltration during Primary Contact Hypersensitivity (Q36247489) (← links)
• IL-34 is a tissue-restricted ligand of CSF1R required for the development of Langerhans cells and microglia (Q37615661) (← links)
• Enhanced excitability of MRGPRA3- and MRGPRD-positive nociceptors in a model of inflammatory itch and pain (Q37647764) (← links)
• ASK1 promotes the contact hypersensitivity response through IL-17 production (Q37705602) (← links)
• Contact dermatitis: a comparative and translational review of the literature (Q38549107) (← links)
• ELR( ) chemokine-mediated neutrophil recruitment is involved in 2,4,6-trinitrochlorobenzene-induced contact hypersensitivity (Q38649819) (← links)
• Potential applications of keratinocytes derived from human embryonic stem cells. (Q38668504) (← links)
• Human embryonic stem cells derived keratinocyte as an in vitro research model for the study of immune response. (Q39184806) (← links)
• Construction and phenotypic analysis of mice carrying a duplication of the major histocompatibility class I (MHC-I) locus. (Q39294873) (← links)
• Allergic Contact Dermatitis: A Model of Inflammatory Itch and Pain in Human and Mouse (Q41811724) (← links)
• A generally applicable translational strategy identifies S100A4 as a candidate gene in allergy. (Q42112839) (← links)
• Aquaporin-9-expressing neutrophils are required for the establishment of contact hypersensitivity (Q43207291) (← links)
• Biological clock dysfunction exacerbates contact hypersensitivity in mice (Q44544254) (← links)
• Topical administration of the pan-Src kinase inhibitors, dasatinib and LCB 03-0110, prevents allergic contact dermatitis in mice. (Q45764606) (← links)
• Local and systemic effects of co-stimulatory blockade using cytotoxic T lymphocyte antigen-4-immunoglobulin in dinitrofluorobenzene- and oxazolone-induced contact hypersensitivity in mice (Q46614633) (← links)
• Sensory TRP channels contribute differentially to skin inflammation and persistent itch. (Q47132169) (← links)
• The Cooperative Role of CD326 and CD11b Dendritic Cell Subsets for a Hapten-Induced Th2 Differentiation (Q47709675) (← links)
• The indane diastereoisomers, PH2 and PH5: divergence between their effects in delayed-type hypersensitivity models and a model of colitis (Q47745802) (← links)
• Local Hyperthermia Affects Murine Contact Hypersensitivity around Elicitation Phase (Q49294273) (← links)
• Curcumin inhibits epigen and amphiregulin upregulated by 2,4,6-trinitrochlorobenzene associated with attenuation of skin swelling. (Q52777064) (← links)
• The balance between pro- and anti-inflammatory cytokines is crucial in human allergic contact dermatitis pathogenesis: the role of IL-1 family members. (Q54688502) (← links)
• Anti-inflammatory Effects of Brassica oleracea Var. capitata L. (Cabbage) Methanol Extract in Mice with Contact Dermatitis. (Q55317497) (← links)
• Depletion of regulatory T cells in a hapten-induced inflammation model results in prolonged and increased inflammation driven by T cells (Q56896192) (← links)
• Mrgprs activation is required for chronic itch conditions in mice (Q57134299) (← links)
• Stat5 gene dosage in T cells modulates CD8 T-cell homeostasis and attenuates contact hypersensitivity response in mice (Q58455445) (← links)
• Multi-walled carbon nanotube oxidation dependent keratinocyte cytotoxicity and skin inflammation (Q60917814) (← links)
• IL-24 contributes to skin inflammation in Para-Phenylenediamine-induced contact hypersensitivity (Q61796786) (← links)
• β-arrestin 2 inhibits proinflammatory chemokine production and attenuates contact allergic inflammation in the skin (Q87374064) (← links)
• Maternal ω3 docosapentaenoic acid inhibits infant allergic dermatitis through TRAIL-expressing plasmacytoid dendritic cells in mice (Q89571135) (← links)
• The regulatory B cell-mediated peripheral tolerance maintained by mast cell IL-5 suppresses oxazolone-induced contact hypersensitivity (Q92060275) (← links)
• TAK-242 ameliorates contact dermatitis exacerbated by IL-36 receptor antagonist deficiency (Q92783002) (← links)
• Quercitrin, the Main Compound in Wikstroemia indica, Mitigates Skin Lesions in a Mouse Model of 2,4-Dinitrochlorobenzene-Induced Contact Hypersensitivity (Q97644735) (← links)