Pages that link to "Q37813545"
Jump to navigation
Jump to search
The following pages link to Pathogenic role of TGF-β in the progression of podocyte diseases (Q37813545):
Displaying 14 items.
- A Systems Biology Overview on Human Diabetic Nephropathy: From Genetic Susceptibility to Post-Transcriptional and Post-Translational Modifications (Q26769678) (← links)
- Mechanisms and consequences of TGF-ß overexpression by podocytes in progressive podocyte disease (Q35649310) (← links)
- Gα12 activation in podocytes leads to cumulative changes in glomerular collagen expression, proteinuria and glomerulosclerosis. (Q35918667) (← links)
- Sugar, sex, and TGF-β in diabetic nephropathy (Q36234919) (← links)
- Protein Array-Based Detection of Proteins in Kidney Tissues from Patients with Membranous Nephropathy (Q37699835) (← links)
- The role of cell–extracellular matrix interactions in glomerular injury (Q37993661) (← links)
- Renal biopsy: use of biomarkers as a tool for the diagnosis of focal segmental glomerulosclerosis (Q38203501) (← links)
- Combined blockade of angiotensin II and prorenin receptors ameliorates podocytic apoptosis induced by IgA-activated mesangial cells (Q38895253) (← links)
- Role of Epigenetic Histone Modifications in Diabetic Kidney Disease Involving Renal Fibrosis (Q39427404) (← links)
- Kidney-targeting Smad7 gene transfer inhibits renal TGF-β/MAD homologue (SMAD) and nuclear factor κB (NF-κB) signalling pathways, and improves diabetic nephropathy in mice (Q45875016) (← links)
- Transforming growth factor-β in stem cells and tissue homeostasis (Q50104779) (← links)
- Panax notoginseng saponins protect kidney from diabetes by up-regulating silent information regulator 1 and activating antioxidant proteins in rats (Q50206045) (← links)
- Severe intraglomerular detachment of podocytes in a Gitelman syndrome patient (Q83831996) (← links)
- Transforming Growth Factor-Beta1 in Diabetic Kidney Disease (Q91714322) (← links)