Pages that link to "Q37429069"
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The following pages link to Loss of parity between IL-2 and IL-21 in the NOD Idd3 locus (Q37429069):
Displaying 35 items.
- Cellular mechanisms of restored β-cell tolerance mediated by protective alleles of Idd3 and Idd5 (Q28253641) (← links)
- Use of nonobese diabetic mice to understand human type 1 diabetes (Q34078163) (← links)
- Distinct genetic control of autoimmune neuropathy and diabetes in the non-obese diabetic background (Q34142507) (← links)
- IL-21 Deficiency Influences CD8 T Cell Quality and Recall Responses following an Acute Viral Infection (Q34179921) (← links)
- A recombination hotspot leads to sequence variability within a novel gene (AK005651) and contributes to type 1 diabetes susceptibility (Q34341601) (← links)
- Interleukin-21 is critically required in autoimmune and allogeneic responses to islet tissue in murine models. (Q34615453) (← links)
- Fine-mapping and transethnic genotyping establish IL2/IL21 genetic association with lupus and localize this genetic effect to IL21 (Q35019514) (← links)
- Autoreactive Cytotoxic T Lymphocytes Acquire Higher Expression of Cytotoxic Effector Markers in the Islets of NOD Mice after Priming in Pancreatic Lymph Nodes (Q35070682) (← links)
- Premature CD4 T cell aging and its contribution to lymphopenia-induced proliferation of memory cells in autoimmune-prone non-obese diabetic mice (Q35107187) (← links)
- A T cell extrinsic mechanism by which IL-2 dampens Th17 differentiation (Q35554348) (← links)
- Clonal Deletion Prunes but Does Not Eliminate Self-Specific αβ CD8( ) T Lymphocytes (Q35683291) (← links)
- Sleeping Beauty Transposon Mutagenesis as a Tool for Gene Discovery in the NOD Mouse Model of Type 1 Diabetes (Q36383217) (← links)
- Genetic interactions among Idd3, Idd5.1, Idd5.2, and Idd5.3 protective loci in the nonobese diabetic mouse model of type 1 diabetes (Q36718178) (← links)
- Innate pro-B-cell progenitors protect against type 1 diabetes by regulating autoimmune effector T cells (Q36932371) (← links)
- Interleukin-21 plays a critical role in the pathogenesis and severity of type I autoimmune hepatitis (Q37017406) (← links)
- Pathogenic mechanisms in type 1 diabetes: the islet is both target and driver of disease (Q37088180) (← links)
- Comparative genetics: synergizing human and NOD mouse studies for identifying genetic causation of type 1 diabetes (Q37088185) (← links)
- Reduced IL‐2 expression in NOD mice leads to a temporal increase in CD62LloFoxP3 CD4 T cells with limited suppressor activity (Q37248276) (← links)
- β-cell-specific IL-2 therapy increases islet Foxp3 Treg and suppresses type 1 diabetes in NOD mice (Q37251067) (← links)
- Implication of IL-2/IL-21 region in systemic sclerosis genetic susceptibility (Q37460609) (← links)
- IL-21 restricts T follicular regulatory T cell proliferation through Bcl-6 mediated inhibition of responsiveness to IL-2. (Q37710748) (← links)
- Mouse models for the study of autoimmune type 1 diabetes: a NOD to similarities and differences to human disease (Q37737987) (← links)
- Targeting interleukin-21 in inflammatory diseases (Q37851522) (← links)
- Interleukin-21 in chronic inflammatory diseases (Q38095870) (← links)
- Advances in our understanding of the pathophysiology of Type 1 diabetes: lessons from the NOD mouse (Q38135862) (← links)
- Expression of IL-2 in β cells by AAV8 gene transfer in pre-diabetic NOD mice prevents diabetes through activation of FoxP3-positive regulatory T cells (Q38449217) (← links)
- CD4 T cell differentiation in type 1 diabetes. (Q38535279) (← links)
- Circadian rhythm-related genes: implication in autoimmunity and type 1 diabetes. (Q38579236) (← links)
- Congenic mice reveal genetic epistasis and overlapping disease loci for autoimmune diabetes and listeriosis (Q42211687) (← links)
- Altered homeostasis and development of regulatory T cell subsets represent an IL-2R-dependent risk for diabetes in NOD mice. (Q47562474) (← links)
- The Common Cytokine Receptor γ Chain Family of Cytokines (Q50077079) (← links)
- Induction of autoimmune diabetes in non-obese diabetic mice requires interleukin-21-dependent activation of autoreactive CD8⁺ T cells (Q51052754) (← links)
- The Role of NOD Mice in Type 1 Diabetes Research: Lessons from the Past and Recommendations for the Future (Q51149244) (← links)
- The circadian gene Arntl2 on distal mouse chromosome 6 controls thymocyte apoptosis (Q51152901) (← links)
- Thymic B Cell-Mediated Attack of Thymic Stroma Precedes Type 1 Diabetes Development. (Q55279860) (← links)