Pages that link to "Q36845491"
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The following pages link to Induction of monocyte chemoattractant protein-1 in the small veins of the ischemic and reperfused canine myocardium. (Q36845491):
Displaying 50 items.
- Differential signaling mechanisms regulate expression of CC chemokine receptor-2 during monocyte maturation (Q24815569) (← links)
- Inflammation and Inflammatory Cells in Myocardial Infarction and Reperfusion Injury: A Double-Edged Sword (Q26747421) (← links)
- Origin of developmental precursors dictates the pathophysiologic role of cardiac fibroblasts (Q26863633) (← links)
- Reactive oxygen intermediates induce monocyte chemotactic protein-1 in vascular endothelium after brief ischemia (Q28360935) (← links)
- Myocardial infarction and remodeling in mice: effect of reperfusion (Q33178763) (← links)
- Macrophage roles following myocardial infarction (Q33802153) (← links)
- Evaluation of Cardiac Function by Transthoracic Echocardiography in Subjects with ST-Segment Elevation Myocardial Infarction Following Primary Percutaneous Coronary Intervention according to Valsartan Dose: The Valsartan One Center Trial (Q34201349) (← links)
- The role of chemokines in human cardiovascular pathology: enhanced biological insights (Q34470222) (← links)
- Monocyte chemoattractant protein-1 and coronary artery disease (Q34631140) (← links)
- Chemokines in health and disease (Q34635703) (← links)
- Primary Monocytes Regulate Endothelial Cell Survival Through Secretion of Angiopoietin-1 and Activation of Endothelial Tie2 (Q35013388) (← links)
- Of mice and dogs: species-specific differences in the inflammatory response following myocardial infarction. (Q35083426) (← links)
- The inflammatory response in myocardial injury, repair, and remodelling. (Q35529656) (← links)
- Chemokines and their receptors as markers of allograft rejection and targets for immunosuppression (Q35538137) (← links)
- Chemokines and the inflammatory response following cardiopulmonary bypass – a new target for therapeutic intervention? – a review (Q35554997) (← links)
- Chemokine receptor‐dependent alloresponses (Q35583497) (← links)
- Increased expression of monocyte chemotactic protein-1 during active hepatic fibrogenesis: correlation with monocyte infiltration (Q35763752) (← links)
- Evidence for an active inflammatory process in the hibernating human myocardium (Q35788998) (← links)
- Dexrazoxane Shows No Protective Effect in the Acute Phase of Reperfusion during Myocardial Infarction in Pigs (Q36230443) (← links)
- The role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction. (Q36399896) (← links)
- Mast cell tryptase may modulate endothelial cell phenotype in healing myocardial infarcts (Q36509613) (← links)
- Microglia and ischemic stroke: a double-edged sword (Q36893695) (← links)
- Inhibition of Janus activated kinase-3 protects against myocardial ischemia and reperfusion injury in mice (Q36907206) (← links)
- The immune system and cardiac repair. (Q37096652) (← links)
- Targeting inflammatory pathways in myocardial infarction. (Q37100838) (← links)
- Induction of early biomarkers in a thrombus-induced sheep model of ischemic heart failure (Q37369965) (← links)
- Chemokines and cardiac fibrosis. (Q37502208) (← links)
- Role of MCP-1 in cardiovascular disease: molecular mechanisms and clinical implications (Q37536067) (← links)
- The inflammatory response and cardiac repair after myocardial infarction (Q37641794) (← links)
- Role of monocyte chemoattractant protein-1 in myocardial infarction (Q38106962) (← links)
- Targeting the chemokines in cardiac repair. (Q38120799) (← links)
- Inflammation as a therapeutic target in myocardial infarction: learning from past failures to meet future challenges. (Q38560476) (← links)
- Cellular recruitment in myocardial ischaemia/reperfusion injury. (Q38811208) (← links)
- The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis. (Q38875774) (← links)
- Association between the monocyte chemoattractant protein-1 −2518G/A gene polymorphism and acute myocardial infarction patients among Egyptian population (Q38985073) (← links)
- Anti-inflammatory drugs as promising cardiovascular treatments. (Q39051784) (← links)
- The role of transforming growth factor (TGF)-β in the infarcted myocardium (Q39265526) (← links)
- The use of MCP-1 and soluble CD40 ligand in a pathophysiology-guided approach to the management of coronary artery disease (Q40393639) (← links)
- Monocyte‐endothelial cell interaction in atherogenesis and thrombosis (Q41706091) (← links)
- Complement inhibitors in myocardial ischemia/reperfusion injury (Q41707687) (← links)
- Contrasting Inflammation Resolution during Atherosclerosis and Post Myocardial Infarction at the Level of Monocyte/Macrophage Phagocytic Clearance (Q42144621) (← links)
- Mechanisms of cell death in acute myocardial infarction: pathophysiological implications for treatment (Q42332637) (← links)
- Brief murine myocardial I/R induces chemokines in a TNF-alpha-independent manner: role of oxygen radicals (Q43800019) (← links)
- Retroinfusion of NFkappaB decoy oligonucleotide extends cardioprotection achieved by CD18 inhibition in a preclinical study of myocardial ischemia and retroinfusion in pigs (Q45885035) (← links)
- Endothelin-1 and monocyte chemoattractant protein-1 modulation in ischemia and human brain-derived endothelial cell cultures (Q46105583) (← links)
- Inflammation following acute myocardial infarction: Multiple players, dynamic roles, and novel therapeutic opportunities (Q47734765) (← links)
- Inflammatory responses after percutaneous coronary intervention in patients with acute myocardial infarction or stable angina pectoris (Q47981043) (← links)
- In vivo labelling of resting monocytes in the reticuloendothelial system with fluorescent iron oxide nanoparticles prior to injury reveals that they are mobilized to infarcted myocardium (Q48828850) (← links)
- Monocyte chemoattractant protein-1 expressed in neurons and astrocytes during focal ischemia in mice (Q48862259) (← links)
- Failure to down-regulate intragraft cytokine mRNA expression shortly after clinical heart transplantation is associated with high incidence of acute rejection (Q48884337) (← links)