Pages that link to "Q36687958"

The following pages link to Analysis of murine coronavirus surface glycoprotein functions by using monoclonal antibodies. (Q36687958):

Displaying 27 items.

• Two-step conformational changes in a coronavirus envelope glycoprotein mediated by receptor binding and proteolysis (Q30436297) (← links)
• Virus-neutralizing monoclonal antibody expressed in milk of transgenic mice provides full protection against virus-induced encephalitis. (Q33865777) (← links)
• Monoclonal antibodies targeting the HR2 domain and the region immediately upstream of the HR2 of the S protein neutralize in vitro infection of severe acute respiratory syndrome coronavirus. (Q34301708) (← links)
• Receptor-induced conformational changes of murine coronavirus spike protein (Q34360420) (← links)
• N-terminal domain of the murine coronavirus receptor CEACAM1 is responsible for fusogenic activation and conformational changes of the spike protein (Q34554080) (← links)
• Receptor-independent infection of murine coronavirus: analysis by spinoculation (Q34647808) (← links)
• Characterization of a human coronavirus (strain 229E) 3C-like proteinase activity (Q35841589) (← links)
• The S2 subunit of the murine coronavirus spike protein is not involved in receptor binding. (Q35851315) (← links)
• Identification of spike protein residues of murine coronavirus responsible for receptor-binding activity by use of soluble receptor-resistant mutants (Q36548568) (← links)
• Localization of neutralizing epitopes and the receptor-binding site within the amino-terminal 330 amino acids of the murine coronavirus spike protein (Q36634787) (← links)
• Identification of an immunodominant linear neutralization domain on the S2 portion of the murine coronavirus spike glycoprotein and evidence that it forms part of complex tridimensional structure (Q36642677) (← links)
• Fusion formation by the uncleaved spike protein of murine coronavirus JHMV variant cl-2. (Q36642701) (← links)
• Organ-specific attenuation of murine hepatitis virus strain A59 by replacement of catalytic residues in the putative viral cyclic phosphodiesterase ns2. (Q37145292) (← links)
• Receptor-independent infection of mouse hepatitis virus: analysis by spinoculation (Q40220816) (← links)
• Communication between S1N330 and a region in S2 of murine coronavirus spike protein is important for virus entry into cells expressing CEACAM1b receptor (Q40729276) (← links)
• A review of feline infectious peritonitis virus: molecular biology, immunopathogenesis, clinical aspects, and vaccination (Q40778595) (← links)
• MHV S peplomer protein expressed by a recombinant vaccinia virus vector exhibits IgG Fc-receptor activity (Q41082112) (← links)
• Pathogenicity of neutralization escape mutants of mouse hepatitis virus: correlation with T- and B-cell depletions (Q41446550) (← links)
• Expression of a Recombinant Monoclonal Antibody From a Bicistronic mRNA (Q42668477) (← links)
• Identification of epitopes associated with different biological activities on the glycoprotein of vesicular stomatitis virus by use of monoclonal antibodies (Q43690904) (← links)
• SJL/J resistance to mouse hepatitis virus-JHM-induced neurologic disease can be partially overcome by viral variants of S and host immunosuppression (Q45876574) (← links)
• Tectonic conformational changes of a coronavirus spike glycoprotein promote membrane fusion. (Q47407834) (← links)
• Sequence analysis of the spike protein gene of murine coronavirus variants: study of genetic sites affecting neuropathogenicity. (Q54277024) (← links)
• Evidence for variable rates of recombination in the MHV genome (Q67489834) (← links)
• Carbohydrate-induced conformational changes strongly modulate the antigenicity of coronavirus TGEV glycoproteins S and M (Q68056872) (← links)
• Targeting Glycoproteins as a therapeutic strategy for diabetes mellitus and its complications (Q89477264) (← links)
• The molecular biology of coronaviruses (Q93899306) (← links)