Pages that link to "Q36653717"
Jump to navigation
Jump to search
The following pages link to The RR1 gene of herpes simplex virus type 1 is uniquely trans activated by ICP0 during infection (Q36653717):
Displaying 36 items.
- ICP0 dismantles microtubule networks in herpes simplex virus-infected cells (Q21136348) (← links)
- The Herpes Simplex Virus Type 1 Cleavage/Packaging Protein, UL32, Is Involved in Efficient Localization of Capsids to Replication Compartments (Q30453534) (← links)
- The PK domain of the large subunit of herpes simplex virus type 2 ribonucleotide reductase (ICP10) is required for immediate-early gene expression and virus growth (Q33785400) (← links)
- Attenuated, replication-competent herpes simplex virus type 1 mutant G207: safety evaluation in mice (Q33802747) (← links)
- Early expression of herpes simplex virus (HSV) proteins and reactivation of latent infection (Q34152914) (← links)
- The cyclin-dependent kinase inhibitor roscovitine inhibits the transactivating activity and alters the posttranslational modification of herpes simplex virus type 1 ICP0. (Q34330457) (← links)
- Evaluation of colocalization interactions between the IE110, IE175, and IE63 transactivator proteins of herpes simplex virus within subcellular punctate structures (Q35830151) (← links)
- Herpes simplex virus immediate-early protein ICP22 is required for viral modification of host RNA polymerase II and establishment of the normal viral transcription program (Q35845807) (← links)
- Herpes simplex ICP27 mutant viruses exhibit reduced expression of specific DNA replication genes (Q35857639) (← links)
- Attenuation of DNA-dependent protein kinase activity and its catalytic subunit by the herpes simplex virus type 1 transactivator ICP0. (Q35871672) (← links)
- Herpes simplex virus immediate-early proteins ICP0 and ICP4 activate the endogenous human alpha-globin gene in nonerythroid cells (Q35877736) (← links)
- The herpes simplex virus immediate-early protein ICP0 affects transcription from the viral genome and infected-cell survival in the absence of ICP4 and ICP27 (Q35886888) (← links)
- Lytic Promoters Express Protein during Herpes Simplex Virus Latency. (Q36062519) (← links)
- Competitive quantitative PCR analysis of herpes simplex virus type 1 DNA and latency-associated transcript RNA in latently infected cells of the rat brain. (Q36630170) (← links)
- PCR-based analysis of herpes simplex virus type 1 latency in the rat trigeminal ganglion established with a ribonucleotide reductase-deficient mutant. (Q36636904) (← links)
- The Differential Requirement for Cyclin-Dependent Kinase Activities Distinguishes Two Functions of Herpes Simplex Virus Type 1 ICP0 (Q37060135) (← links)
- Alternatively spliced mRNAs predicted to yield frame-shift proteins and stable intron 1 RNAs of the herpes simplex virus 1 regulatory gene alpha 0 accumulate in the cytoplasm of infected cells (Q37242656) (← links)
- Ras-GAP binding and phosphorylation by herpes simplex virus type 2 RR1 PK (ICP10) and activation of the Ras/MEK/MAPK mitogenic pathway are required for timely onset of virus growth (Q39585343) (← links)
- Hexamethylene bisacetamide leads to reduced helper virus-free HSV-1 amplicon expression titers via suppression of ICP0. (Q39601616) (← links)
- The herpes simplex virus type 2 R1 protein kinase (ICP10 PK) blocks apoptosis in hippocampal neurons, involving activation of the MEK/MAPK survival pathway (Q39682246) (← links)
- Characterization of the novel protein kinase activity present in the R1 subunit of herpes simplex virus ribonucleotide reductase (Q39871239) (← links)
- Point mutations in the herpes simplex virus type 1 Vmw110 RING finger helix affect activation of gene expression, viral growth, and interaction with PML-containing nuclear structures. (Q39872517) (← links)
- Detection of herpes simplex virus type 1 latency-associated transcript expression in trigeminal ganglia by in situ reverse transcriptase PCR. (Q39876313) (← links)
- Induction of Transcription by a Viral Regulatory Protein Depends on the Relative Strengths of Functional TATA Boxes (Q40017145) (← links)
- Analysis of Herpes Simplex Virus ICP0 Promoter Function in Sensory Neurons during Acute Infection, Establishment of Latency, and Reactivation In Vivo (Q40132856) (← links)
- Transcription of the Herpes Simplex Virus Genome during Productive and Latent Infection (Q40473966) (← links)
- Multiple immediate-early gene-deficient herpes simplex virus vectors allowing efficient gene delivery to neurons in culture and widespread gene delivery to the central nervous system in vivo (Q40814855) (← links)
- HSV as a gene transfer vector for the nervous system (Q40922461) (← links)
- Regulation of synthesis of ribonucleotide reductase and relationship to DNA replication in various systems (Q40998388) (← links)
- Overexpression of transfected human ribonucleotide reductase M2 subunit in human cancer cells enhances their invasive potential (Q41058506) (← links)
- A phase I pharmacodynamic study of GTI-2040, an antisense oligonucleotide against ribonuclotide reductase, in acute leukemias: a California Cancer Consortium study (Q41652561) (← links)
- Progress in development of herpes simplex virus gene vectors for treatment of rheumatoid arthritis (Q43524051) (← links)
- The Large Subunit of Herpes Simplex Virus Type 2 Ribonucleotide Reductase (ICP10) Is Associated with the Virion Tegument and Has PK Activity (Q45760993) (← links)
- Attenuated Herpes Simplex Virus 1 (HSV-1) Expressing a Mutant Form of ICP6 Stimulates a Strong Immune Response That Protects Mice against HSV-1-Induced Corneal Disease (Q59358017) (← links)
- Engineering Herpes Simplex (Q62971299) (← links)
- A Conserved Mechanism of APOBEC3 Relocalization by Herpesviral Ribonucleotide Reductase Large Subunits (Q90167784) (← links)