Pages that link to "Q36587818"

The following pages link to The PML-retinoic acid receptor alpha translocation converts the receptor from an inhibitor to a retinoic acid-dependent activator of transcription factor AP-1 (Q36587818):

Displaying 34 items.

• A novel member of the BTB/POZ family, PATZ, associates with the RNF4 RING finger protein and acts as a transcriptional repressor (Q22253326) (← links)
• PML/RARalpha targets promoter regions containing PU.1 consensus and RARE half sites in acute promyelocytic leukemia (Q24299511) (← links)
• PML, a growth suppressor disrupted in acute promyelocytic leukemia (Q24324168) (← links)
• Modulation of CREB binding protein function by the promyelocytic (PML) oncoprotein suggests a role for nuclear bodies in hormone signaling (Q24674185) (← links)
• The autoimmune regulator protein has transcriptional transactivating properties and interacts with the common coactivator CREB-binding protein (Q28140689) (← links)
• SUMO-1 protease-1 regulates gene transcription through PML (Q28507462) (← links)
• HIF factors cooperate with PML-RARα to promote acute promyelocytic leukemia progression and relapse (Q33623363) (← links)
• PML/RARalpha fusion protein transactivates the tissue factor promoter through a GAGC-containing element without direct DNA association (Q33734488) (← links)
• Acute promyelocytic leukemia: from treatment to genetics and back (Q33739256) (← links)
• Disruption of PML Subnuclear Domains by the Acidic IE1 Protein of Human Cytomegalovirus Is Mediated through Interaction with PML and May Modulate a RING Finger-Dependent Cryptic Transactivator Function of PML (Q33776312) (← links)
• The promyelocytic (PML) nuclear compartment and transcription control (Q34047268) (← links)
• Cross-talk between glucocorticoid receptor and AP-1. (Q34279367) (← links)
• Transcriptional regulation in acute promyelocytic leukemia. (Q34435531) (← links)
• Classical and novel retinoids: their targets in cancer therapy (Q34605217) (← links)
• The promyelocytic leukemia nuclear body: sites of activity? (Q34746892) (← links)
• Variations on a theme: the alternate translocations in APL. (Q34926608) (← links)
• Transcriptional activation by TAL1 and FUS-CHOP proteins expressed in acute malignancies as a result of chromosomal abnormalities. (Q35669591) (← links)
• Genome-wide functions of PML-RARα in acute promyelocytic leukaemia (Q37829905) (← links)
• Computational identification of the normal and perturbed genetic networks involved in myeloid differentiation and acute promyelocytic leukemia (Q38275870) (← links)
• Retinoid receptors and keratinocytes (Q41005226) (← links)
• A retrovirus carrying the promyelocyte-retinoic acid receptor PML-RARalpha fusion gene transforms haematopoietic progenitors in vitro and induces acute leukaemias (Q41065219) (← links)
• Transcriptional Repression by the Promyelocytic Leukemia Protein, PML (Q41071228) (← links)
• PML-containing nuclear bodies: their spatial distribution in relation to other nuclear components (Q41149744) (← links)
• Nuclear DNA helicase II is recruited to IFN-alpha-activated transcription sites at PML nuclear bodies. (Q41965526) (← links)
• Coordinated regulation of the immunoproteasome subunits by PML/RARα and PU.1 in acute promyelocytic leukemia (Q42440049) (← links)
• Pro-proliferative function of the long isoform of PML-RARalpha involved in acute promyelocytic leukemia (Q42805877) (← links)
• The t(8;21) fusion protein, AML1/ETO, transforms NIH3T3 cells and activates AP-1. (Q42816817) (← links)
• Treatment by design in leukemia, a meeting report, Philadelphia, Pennsylvania, December 2002. (Q44619783) (← links)
• The short isoform of PML-RARα activates the NRF2/HO-1 pathway through a direct interaction with NRF2. (Q47990678) (← links)
• Overview of Genetic Diagnosis in Cancer. (Q54365695) (← links)
• PML-RARα interferes with erythropoiesis by repressing LMO2 in acute promyelocytic leukaemia (Q57463561) (← links)
• RNF8 is responsible for ATRA resistance in variant acute promyelocytic leukemia with GTF2I/RARA fusion, and inhibition of the ubiquitin-proteasome pathway contributes to the reversion of ATRA resistance (Q64075543) (← links)
• Leukemia: the sophisticated subversion of hematopoiesis by nuclear receptor oncoproteins (Q78169779) (← links)
• KDM3B suppresses APL progression by restricting chromatin accessibility and facilitating the ATRA-mediated degradation of PML/RARα (Q90581670) (← links)