Pages that link to "Q36459963"

The following pages link to Expression of Fused in sarcoma mutations in mice recapitulates the neuropathology of FUS proteinopathies and provides insight into disease pathogenesis (Q36459963):

Displaying 27 items.

• Mechanisms of FUS mutations in familial amyotrophic lateral sclerosis (Q26749169) (← links)
• Microglia centered pathogenesis in ALS: insights in cell interconnectivity (Q27009559) (← links)
• Pathogenesis of FUS-associated ALS and FTD: insights from rodent models (Q28067147) (← links)
• FUS Interacts with HSP60 to Promote Mitochondrial Damage (Q28547727) (← links)
• Motor neuron intrinsic and extrinsic mechanisms contribute to the pathogenesis of FUS-associated amyotrophic lateral sclerosis (Q30848999) (← links)
• Neuropathology of Amyotrophic Lateral Sclerosis and Its Variants (Q33361835) (← links)
• FUS is phosphorylated by DNA-PK and accumulates in the cytoplasm after DNA damage (Q33707858) (← links)
• The ALS gene FUS regulates synaptic transmission at the Drosophila neuromuscular junction. (Q33784500) (← links)
• Capsid serotype and timing of injection determines AAV transduction in the neonatal mice brain (Q34795088) (← links)
• Severe respiratory changes at end stage in a FUS-induced disease state in adult rats (Q36177447) (← links)
• Fused in sarcoma (FUS) protein lacking nuclear localization signal (NLS) and major RNA binding motifs triggers proteinopathy and severe motor phenotype in transgenic mice (Q37132099) (← links)
• Composition of Rosenthal Fibers, the Protein Aggregate Hallmark of Alexander Disease (Q37284065) (← links)
• FUS/TLS acts as an aggregation-dependent modifier of polyglutamine disease model mice (Q37339987) (← links)
• Protein aggregation in amyotrophic lateral sclerosis. (Q38106862) (← links)
• The role of FUS gene variants in neurodegenerative diseases (Q38212812) (← links)
• Worming forward: amyotrophic lateral sclerosis toxicity mechanisms and genetic interactions in Caenorhabditis elegans (Q38214677) (← links)
• Advances in the Development of Disease-Modifying Treatments for Amyotrophic Lateral Sclerosis (Q38741121) (← links)
• PINK1 and Parkin are genetic modifiers for FUS-induced neurodegeneration. (Q39235670) (← links)
• ALS/FTD-associated FUS activates GSK-3β to disrupt the VAPB-PTPIP51 interaction and ER-mitochondria associations. (Q40991422) (← links)
• Adenoviral expression of TDP-43 and FUS genes and shRNAs for protein degradation pathways in rodent motoneurons in vitro and in vivo (Q42277252) (← links)
• Humanized mutant FUS drives progressive motor neuron degeneration without aggregation in 'FUSDelta14' knockin mice. (Q47623937) (← links)
• ALS/FTD-Linked Mutation in FUS Suppresses Intra-axonal Protein Synthesis and Drives Disease Without Nuclear Loss-of-Function of FUS (Q57813809) (← links)
• From Mouse Models to Human Disease: An Approach for Amyotrophic Lateral Sclerosis (Q58765919) (← links)
• FUS causes synaptic hyperexcitability in Drosophila dendritic arborization neurons (Q88292829) (← links)
• rAAV-based brain slice culture models of Alzheimer's and Parkinson's disease inclusion pathologies (Q91632938) (← links)
• ALS Genetics, Mechanisms, and Therapeutics: Where Are We Now? (Q92187628) (← links)
• The Missing Heritability of Sporadic Frontotemporal Dementia: New Insights from Rare Variants in Neurodegenerative Candidate Genes (Q92570889) (← links)