Pages that link to "Q36456672"

The following pages link to Mass spectrometry-based identification of native cardiac Nav1.5 channel α subunit phosphorylation sites. (Q36456672):

Displaying 23 items.

• Counteracting Protein Kinase Activity in the Heart: The Multiple Roles of Protein Phosphatases (Q26776182) (← links)
• The multi-faceted aspects of the complex cardiac Nav1.5 protein in membrane function and pathophysiology (Q26799744) (← links)
• CaMKII-dependent regulation of cardiac Na( ) homeostasis (Q26997372) (← links)
• An update on transcriptional and post-translational regulation of brain voltage-gated sodium channels (Q28087661) (← links)
• Post-translational modifications of the cardiac Na channel: contribution of CaMKII-dependent phosphorylation to acquired arrhythmias (Q28292930) (← links)
• Voltage-Gated Sodium Channel Phosphorylation at Ser571 Regulates Late Current, Arrhythmia, and Cardiac Function In Vivo (Q30973910) (← links)
• Increased persistent sodium current due to decreased PI3K signaling contributes to QT prolongation in the diabetic heart (Q37333469) (← links)
• Neuronal Na Channels Are Integral Components of Pro-arrhythmic Na /Ca2 Signaling Nanodomain That Promotes Cardiac Arrhythmias During β-adrenergic Stimulation (Q37341328) (← links)
• CaMKII Phosphorylation of Na(V)1.5: Novel in Vitro Sites Identified by Mass Spectrometry and Reduced S516 Phosphorylation in Human Heart Failure (Q37601736) (← links)
• Modulatory mechanisms and multiple functions of somatodendritic A-type K ( ) channel auxiliary subunits (Q37681403) (← links)
• Function and regulation of serine/threonine phosphatases in the healthy and diseased heart. (Q38139729) (← links)
• Control of cardiac repolarization by phosphoinositide 3-kinase signaling to ion channels (Q38304968) (← links)
• Role of CaMKII in cardiac arrhythmias (Q38313769) (← links)
• Regulation of the cardiac Na channel NaV1.5 by post-translational modifications. (Q38369684) (← links)
• Neuronal sodium channels: emerging components of the nano-machinery of cardiac calcium cycling (Q39134795) (← links)
• C-terminal phosphorylation of NaV1.5 impairs FGF13-dependent regulation of channel inactivation. (Q46697702) (← links)
• Voltage-gated sodium channels assemble and gate as dimers (Q47145207) (← links)
• Posttranslational Modification of Sodium Channels (Q50086104) (← links)
• Blockade of CaMKII depresses conduction preferentially in the right ventricular outflow tract and promotes ischemic ventricular fibrillation in the rabbit heart (Q51165286) (← links)
• Late sodium current dysregulation as a causal factor in arrhythmia (Q51517055) (← links)
• Identification of N-terminal protein acetylation and arginine methylation of the voltage-gated sodium channel in end-stage heart failure human heart (Q57707814) (← links)
• Phosphorylation of cardiac voltage-gated sodium channel: potential players with multiple dimensions (Q58604019) (← links)
• "Heart Oddity": Intrinsically Reduced Excitability in the Right Ventricle Requires Compensation by Regionally Specific Stress Kinase Function (Q90054837) (← links)