Pages that link to "Q36435215"
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The following pages link to Hyperpolarization-activated, cyclic nucleotide-gated cation channels in Aplysia: Contribution to classical conditioning (Q36435215):
Displaying 6 items.
- Decreased hyperpolarization-activated cyclic nucleotide-gated channels are involved in bladder dysfunction associated with spinal cord injury (Q50047888) (← links)
- Inhibition of GluR Current in Microvilli of Sensory Neurons via Na -Microdomain Coupling Among GluR, HCN Channel, and Na /K Pump. (Q55090037) (← links)
- Persistent effects of cyclic adenosine monophosphate are directly responsible for maintaining a neural network state. (Q64993079) (← links)
- Comparison of the ionic currents modulated during activity-dependent and normal presynaptic facilitation (Q90729862) (← links)
- Hyperpolarization-Activated Cyclic Nucleotide-Gated Ion (HCN) Channels Regulate PC12 Cell Differentiation Toward Sympathetic Neuron (Q90732187) (← links)
- Intermediate-term memory in Aplysia involves neurotrophin signaling, transcription, and DNA methylation (Q93160125) (← links)