Pages that link to "Q36423404"

The following pages link to Dioxin-mediated tumor progression through activation of mitochondria-to-nucleus stress signaling (Q36423404):

Displaying 43 items.

• Dioxins and cardiovascular disease mortality (Q23918980) (← links)
• Dual localization of glutathione S-transferase in the cytosol and mitochondria: implications in oxidative stress, toxicity and disease (Q24633323) (← links)
• Mitochondrial DNA damage and its consequences for mitochondrial gene expression (Q27001150) (← links)
• Dioxins, the aryl hydrocarbon receptor and the central regulation of energy balance (Q28287699) (← links)
• The impact of low-dose carcinogens and environmental disruptors on tissue invasion and metastasis (Q28388257) (← links)
• Exactly the Same but Different: Promiscuity and Diversity in the Molecular Mechanisms of Action of the Aryl Hydrocarbon (Dioxin) Receptor (Q28392826) (← links)
• Dynamic zebrafish interactome reveals transcriptional mechanisms of dioxin toxicity (Q28395284) (← links)
• Extension of the mitochondria dysfunction hypothesis of metabolic syndrome to atherosclerosis with emphasis on the endocrine-disrupting chemicals and biophysical laws (Q33603449) (← links)
• TCDD promotes lung tumors via attenuation of apoptosis through activation of the Akt and ERK1/2 signaling pathways. (Q33753495) (← links)
• Chloramphenicol causes mitochondrial stress, decreases ATP biosynthesis, induces matrix metalloproteinase-13 expression, and solid-tumor cell invasion (Q33917938) (← links)
• Moesin is a biomarker for the assessment of genotoxic carcinogens in mouse lymphoma (Q34167507) (← links)
• Strong associations between the pesticide hexachlorocyclohexane and type 2 diabetes in Saudi adults (Q34348073) (← links)
• Association between type 2 diabetes and exposure to persistent organic pollutants (Q35179930) (← links)
• DDT and its metabolites are linked to increased risk of type 2 diabetes among Saudi adults: a cross-sectional study (Q35215894) (← links)
• Circulating persistent organic pollutants and body fat distribution: Evidence from NHANES 1999-2004. (Q36001568) (← links)
• 2,3,7,8-Tetrachlorodibenzo-p-dioxin-mediated production of reactive oxygen species is an essential step in the mechanism of action to accelerate human keratinocyte differentiation (Q36620467) (← links)
• Mitochondria as a target of environmental toxicants (Q36956066) (← links)
• Ah Receptor Activation by Dioxin Disrupts Activin, BMP, and WNT Signals During the Early Differentiation of Mouse Embryonic Stem Cells and Inhibits Cardiomyocyte Functions (Q36987476) (← links)
• Mitochondrial retrograde signaling at the crossroads of tumor bioenergetics, genetics and epigenetics (Q37320456) (← links)
• Heterogeneous nuclear ribonucleoprotein A2 is a common transcriptional coactivator in the nuclear transcription response to mitochondrial respiratory stress (Q37346019) (← links)
• Mutations in mitochondrial DNA polymerase-gamma promote breast tumorigenesis (Q37434648) (← links)
• Identification of EBP50 as a specific biomarker for carcinogens via the analysis of mouse lymphoma cellular proteome (Q37461483) (← links)
• Implications of mitochondrial DNA mutations and mitochondrial dysfunction in tumorigenesis (Q37521873) (← links)
• Fish oil rich in eicosapentaenoic acid protects against oxidative stress-related renal dysfunction induced by TCDD in Wistar rats (Q37695222) (← links)
• Dioxin toxicity, aryl hydrocarbon receptor signaling, and apoptosis—Persistent pollutants affect programmed cell death (Q37842582) (← links)
• The use of α-fetoprotein for the delivery of cytotoxic payloads to cancer cells (Q38261958) (← links)
• Classical and Novel TSPO Ligands for the Mitochondrial TSPO Can Modulate Nuclear Gene Expression: Implications for Mitochondrial Retrograde Signaling (Q38708545) (← links)
• Mitochondrial-targeted aryl hydrocarbon receptor and the impact of 2,3,7,8-tetrachlorodibenzo-p-dioxin on cellular respiration and the mitochondrial proteome (Q38776225) (← links)
• International regulatory needs for development of an IATA for non-genotoxic carcinogenic chemical substances (Q38800245) (← links)
• Mitochondrial dysfunction and mitochondrial dynamics-The cancer connection (Q39011563) (← links)
• Release of targeted p53 from the mitochondrion as an early signal during mitochondrial dysfunction. (Q39117989) (← links)
• Role of calcineurin, hnRNPA2 and Akt in mitochondrial respiratory stress-mediated transcription activation of nuclear gene targets (Q39740063) (← links)
• Synergistic cellular effects including mitochondrial destabilization, autophagy and apoptosis following low-level exposure to a mixture of lipophilic persistent organic pollutants (Q40956845) (← links)
• A distinctive physiological role for IkappaBbeta in the propagation of mitochondrial respiratory stress signaling (Q42014943) (← links)
• β-Naphthoflavone-Induced Mitochondrial Respiratory Damage in Cyp1 Knockout Mouse and in Cell Culture Systems: Attenuation by Resveratrol Treatment. (Q43372982) (← links)
• Metabolomic analysis of liver and skeletal muscle tissues in C57BL/6J and DBA/2J mice exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin. (Q45962023) (← links)
• Dioxin-sensitive proteins in differentiating osteoblasts: effects on bone formation in vitro (Q46129106) (← links)
• Actein alleviates 2,3,7,8-tetrachlorodibenzo-p-dioxin-mediated cellular dysfunction in osteoblastic MC3T3-E1 cells. (Q46316992) (← links)
• Early Life Exposure to Low Levels of AHR Agonist PCB126 (3,3',4,4',5-Pentachlorobiphenyl) Reprograms Gene Expression in Adult Brain (Q48035326) (← links)
• From dioxin to dioxin congeners: understanding the differences in hydrophobic aggregation in water and absorption into lipid membranes by means of atomistic simulations. (Q53042859) (← links)
• hnRNPA2 mediated acetylation reduces telomere length in response to mitochondrial dysfunction (Q59807501) (← links)
• Nuclear localization of the mitochondrial ncRNAs in normal and cancer cells (Q83468964) (← links)
• Dysregulation of RyR Calcium Channel Causes the Onset of Mitochondrial Retrograde Signaling (Q98185959) (← links)