Pages that link to "Q36246858"
Jump to navigation
Jump to search
The following pages link to Alzheimer's β-secretase (BACE1) regulates the cAMP/PKA/CREB pathway independently of β-amyloid (Q36246858):
Displaying 46 items.
- Diminished CRE-Induced Plasticity is Linked to Memory Deficits in Familial Alzheimer's Disease Mice (Q27333843) (← links)
- Role of Drosophila Amyloid Precursor Protein in Memory Formation (Q28072008) (← links)
- Hsp90 chaperone inhibitor 17-AAG attenuates Aβ-induced synaptic toxicity and memory impairment (Q28565257) (← links)
- Cross-Sectional and Longitudinal Effects of CREB1 Genotypes on Individual Differences in Memory and Executive Function: Findings from the BLSA (Q33689729) (← links)
- Dexibuprofen prevents neurodegeneration and cognitive decline in APPswe/PS1dE9 through multiple signaling pathways. (Q33825472) (← links)
- Knock-in of human BACE1 cleaves murine APP and reiterates Alzheimer-like phenotypes (Q34006919) (← links)
- PERK mediates eIF2α phosphorylation responsible for BACE1 elevation, CREB dysfunction and neurodegeneration in a mouse model of Alzheimer's disease (Q34024468) (← links)
- RTN3 Is a Novel Cold-Induced Protein and Mediates Neuroprotective Effects of RBM3. (Q34552112) (← links)
- Protein kinase A rescues microtubule affinity-regulating kinase 2-induced microtubule instability and neurite disruption by phosphorylating serine 409. (Q35048976) (← links)
- Synaptic therapy in Alzheimer's disease: a CREB-centric approach (Q35064948) (← links)
- Postsynaptic target specific synaptic dysfunctions in the CA3 area of BACE1 knockout mice (Q35122722) (← links)
- A novel triple repeat mutant tau transgenic model that mimics aspects of pick's disease and fronto-temporal tauopathies (Q35213879) (← links)
- Amyloid-β Peptide Exacerbates the Memory Deficit Caused by Amyloid Precursor Protein Loss-of-Function in Drosophila (Q35746174) (← links)
- A brain-targeted, modified neurosin (kallikrein-6) reduces α-synuclein accumulation in a mouse model of multiple system atrophy (Q36086721) (← links)
- BACE1 is at the crossroad of a toxic vicious cycle involving cellular stress and β-amyloid production in Alzheimer's disease (Q36427048) (← links)
- α-Synuclein interferes with the ESCRT-III complex contributing to the pathogenesis of Lewy body disease (Q36608855) (← links)
- Reduced pCREB in Alzheimer's disease prefrontal cortex is reflected in peripheral blood mononuclear cells (Q37199042) (← links)
- CREB overexpression in dorsal CA1 ameliorates long-term memory deficits in aged rats. (Q37586398) (← links)
- Mitochondria: a kinase anchoring protein 1, a signaling platform for mitochondrial form and function (Q37613398) (← links)
- Genetic Deletion of Prostacyclin IP Receptor Exacerbates Transient Global Cerebral Ischemia in Aging Mice (Q37630989) (← links)
- Roles of eIF2α kinases in the pathogenesis of Alzheimer's disease (Q37718976) (← links)
- MicroRNAs in the brain: it's regulatory role in neuroinflammation (Q38073708) (← links)
- Accumulation of oligomer-prone α-synuclein exacerbates synaptic and neuronal degeneration in vivo (Q38966914) (← links)
- Amyloid precursor protein and endosomal-lysosomal dysfunction in Alzheimer's disease: inseparable partners in a multifactorial disease (Q39405747) (← links)
- BACE1 Deficiency Causes Abnormal Neuronal Clustering in the Dentate Gyrus. (Q41019229) (← links)
- Derailed intraneuronal signalling drives pathogenesis in sporadic and familial Alzheimer's disease. (Q42872730) (← links)
- The Past, Present, and Future of Phosphodiesterase-4 Modulation for Age-Induced Memory Loss (Q45068631) (← links)
- Cyclic nucleotide signaling changes associated with normal aging and age-related diseases of the brain. (Q45305673) (← links)
- Amyloid-β dimers in the absence of plaque pathology impair learning and synaptic plasticity. (Q46625235) (← links)
- Centrally Delivered BACE1 Inhibitor Activates Microglia, and Reverses Amyloid Pathology and Cognitive Deficit in Aged Tg2576 Mice. (Q47707395) (← links)
- Chronic Sleep Restriction Induces Cognitive Deficits and Cortical Beta-Amyloid Deposition in Mice via BACE1-Antisense Activation (Q47768829) (← links)
- cAMP/PKA signaling pathway contributes to neuronal apoptosis via regulating IDE expression in a mixed model of type 2 diabetes and Alzheimer's disease. (Q47945449) (← links)
- Secretases-related miRNAs in Alzheimer's disease: new approach for biomarker discovery (Q47974946) (← links)
- Early Preclinical Changes in Hippocampal CREB-Binding Protein Expression in a Mouse Model of Familial Alzheimer's Disease. (Q48018264) (← links)
- AMPK Plays a Dual Role in Regulation of CREB/BDNF Pathway in Mouse Primary Hippocampal Cells (Q48347195) (← links)
- Brain Amyloid-β Plays an Initiating Role in the Pathophysiological Process of the PS1V97L-Tg Mouse Model of Alzheimer's Disease (Q50655925) (← links)
- Inhibition of endothelial nitric oxide synthase reverses the effect of exercise on improving cognitive function in hypertensive rats. (Q52641741) (← links)
- Effects of CREB1 gene silencing on cognitive dysfunction by mediating PKA-CREB signaling pathway in mice with vascular dementia. (Q55450411) (← links)
- Adenosine Augmentation Evoked by an ENT1 Inhibitor Improves Memory Impairment and Neuronal Plasticity in the APP/PS1 Mouse Model of Alzheimer’s Disease (Q57896371) (← links)
- MicroRNA-98 reduces amyloid β-protein production and improves oxidative stress and mitochondrial dysfunction through the Notch signaling pathway via HEY2 in Alzheimer's disease mice (Q58567557) (← links)
- Molecular Targets of Bis (7)-Cognitin and Its Relevance in Neurological Disorders: A Systematic Review (Q64260940) (← links)
- Electroacupuncture Mitigates Hippocampal Cognitive Impairments by Reducing BACE1 Deposition and Activating PKA in APP/PS1 Double Transgenic Mice. (Q64911191) (← links)
- A dual GLP-1 and Gcg receptor agonist rescues spatial memory and synaptic plasticity in APP/PS1 transgenic mice (Q91440655) (← links)
- Pharmacotherapy of Alzheimer's Disease: Seeking Clarity in a Time of Uncertainty (Q91710367) (← links)
- Synaptic and memory dysfunction induced by tau oligomers is rescued by up-regulation of the nitric oxide cascade (Q93083881) (← links)
- Phosphodiesterase Inhibitors for Alzheimer's Disease: A Systematic Review of Clinical Trials and Epidemiology with a Mechanistic Rationale (Q97887377) (← links)