Pages that link to "Q35850550"
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The following pages link to Herpes simplex virus VP16 rescues viral mRNA from destruction by the virion host shutoff function (Q35850550):
Displaying 50 items.
- Molecular biology of pseudorabies virus: impact on neurovirology and veterinary medicine (Q24531303) (← links)
- An activation domain in the C-terminal subunit of HCF-1 is important for transactivation by VP16 and LZIP. (Q24536035) (← links)
- Herpes simplex virus virion host shutoff protein is stimulated by translation initiation factors eIF4B and eIF4H (Q24623749) (← links)
- Association of the herpes simplex virus type 1 Us11 gene product with the cellular kinesin light-chain-related protein PAT1 results in the redistribution of both polypeptides (Q24683333) (← links)
- Shutoff of Host Gene Expression in Influenza A Virus and Herpesviruses: Similar Mechanisms and Common Themes (Q26752361) (← links)
- Tegument Assembly and Secondary Envelopment of Alphaherpesviruses (Q26782247) (← links)
- Ocular Herpes Simplex Virus: How are Latency, Reactivation, Recurrent Disease and Therapy Interrelated? (Q27021069) (← links)
- The herpes simplex virus type 1 vhs-UL41 gene secures viral replication by temporarily evading apoptotic cellular response to infection: Vhs-UL41 activity might require interactions with elements of cellular mRNA degradation machinery (Q33229421) (← links)
- Transcriptomic analysis of the dialogue between Pseudorabies virus and porcine epithelial cells during infection (Q33323240) (← links)
- De novo synthesis of VP16 coordinates the exit from HSV latency in vivo (Q33422983) (← links)
- The herpes simplex virus vhs protein induces endoribonucleolytic cleavage of target RNAs in cell extracts (Q33648831) (← links)
- Construction and properties of a herpes simplex virus 2 dl5-29 vaccine candidate strain encoding an HSV-1 virion host shutoff protein (Q33724497) (← links)
- Analysis of HCF, the cellular cofactor of VP16, in herpes simplex virus-infected cells (Q33794386) (← links)
- Evidence that herpes simplex virus VP16 is required for viral egress downstream of the initial envelopment event (Q33807388) (← links)
- Site-directed mutagenesis of the virion host shutoff gene (UL41) of herpes simplex virus (HSV): analysis of functional differences between HSV type 1 (HSV-1) and HSV-2 alleles. (Q33822743) (← links)
- Picornavirus internal ribosome entry site elements target RNA cleavage events induced by the herpes simplex virus virion host shutoff protein (Q33822939) (← links)
- Truncation of the C-terminal acidic transcriptional activation domain of herpes simplex virus VP16 renders expression of the immediate-early genes almost entirely dependent on ICP0 (Q33824256) (← links)
- Herpes simplex virus triggers and then disarms a host antiviral response (Q33835458) (← links)
- Herpes simplex virus virion host shutoff protein requires a mammalian factor for efficient in vitro endoribonuclease activity. (Q33836046) (← links)
- Translational control of viral gene expression in eukaryotes (Q33935121) (← links)
- Role of herpes simplex virus ICP27 in the degradation of mRNA by virion host shutoff RNase (Q34120868) (← links)
- Cell surface major histocompatibility complex class II proteins are regulated by the products of the gamma(1)34.5 and U(L)41 genes of herpes simplex virus 1. (Q34341979) (← links)
- Varicella-zoster virus (VZV) ORF17 protein induces RNA cleavage and is critical for replication of VZV at 37 degrees C but not 33 degrees C. (Q34349183) (← links)
- The patterns of accumulation of cellular RNAs in cells infected with a wild-type and a mutant herpes simplex virus 1 lacking the virion host shutoff gene (Q34430595) (← links)
- Analysis of virion-incorporated host proteins required for herpes simplex virus type 1 infection through a RNA interference screen (Q34542419) (← links)
- ICP0 is not required for efficient stress-induced reactivation of herpes simplex virus type 1 from cultured quiescently infected neuronal cells (Q34545788) (← links)
- A single amino acid substitution in herpes simplex virus type 1 VP16 inhibits binding to the virion host shutoff protein and is incompatible with virus growth (Q34758139) (← links)
- Herpes simplex virus ICP27 is required for virus-induced stabilization of the ARE-containing IEX-1 mRNA encoded by the human IER3 gene (Q35101393) (← links)
- Phosphorylation of the VP16 transcriptional activator protein during herpes simplex virus infection and mutational analysis of putative phosphorylation sites (Q35253876) (← links)
- Attenuation of herpes simplex virus neurovirulence with picornavirus cis-acting genetic elements (Q35635064) (← links)
- Packaging of the virion host shutoff (Vhs) protein of herpes simplex virus: two forms of the Vhs polypeptide are associated with intranuclear B and C capsids, but only one is associated with enveloped virions (Q35635097) (← links)
- Expression and function of the equine herpesvirus 1 virion-associated host shutoff homolog (Q35873762) (← links)
- Truncation of the C-terminal acidic transcriptional activation domain of herpes simplex virus VP16 produces a phenotype similar to that of the in1814 linker insertion mutation (Q35890478) (← links)
- Mutational analysis of the virion host shutoff gene (UL41) of herpes simplex virus (HSV): characterization of HSV type 1 (HSV-1)/HSV-2 chimeras (Q35894630) (← links)
- Interaction of herpes simplex virus RNase with VP16 and VP22 is required for the accumulation of the protein but not for accumulation of mRNA (Q35901199) (← links)
- HSV-2 inhibits type-I interferon signaling via multiple complementary and compensatory STAT2-associated mechanisms (Q36098919) (← links)
- Replication-competent herpes simplex virus 1 isolates selected from cells transfected with a bacterial artificial chromosome DNA lacking only the UL49 gene vary with respect to the defect in the UL41 gene encoding host shutoff RNase. (Q36099301) (← links)
- Deletion of the herpes simplex virus 1 UL49 gene results in mRNA and protein translation defects that are complemented by secondary mutations in UL41. (Q36363780) (← links)
- Translocation and colocalization of ICP4 and ICP0 in cells infected with herpes simplex virus 1 mutants lacking glycoprotein E, glycoprotein I, or the virion host shutoff product of the UL41 gene (Q36483357) (← links)
- Selective ablation of virion host shutoff protein RNase activity attenuates herpes simplex virus 2 in mice (Q36498155) (← links)
- Messenger RNA turnover and its regulation in herpesviral infection (Q36550916) (← links)
- Disruption of the U(L)41 gene in the herpes simplex virus 2 dl5-29 mutant increases its immunogenicity and protective capacity in a murine model of genital herpes (Q36570266) (← links)
- C-terminal trans-activation sub-region of VP16 is uniquely required for forskolin-induced herpes simplex virus type 1 reactivation from quiescently infected-PC12 cells but not for replication in neuronally differentiated-PC12 cells (Q36597915) (← links)
- The herpes simplex virus host shutoff RNase degrades cellular and viral mRNAs made before infection but not viral mRNA made after infection (Q36760073) (← links)
- Selective degradation of mRNAs by the HSV host shutoff RNase is regulated by the UL47 tegument protein (Q36820009) (← links)
- VP22 of herpes simplex virus 1 promotes protein synthesis at late times in infection and accumulation of a subset of viral mRNAs at early times in infection (Q37033322) (← links)
- The herpes simplex virus 1 UL41 gene-dependent destabilization of cellular RNAs is selective and may be sequence-specific (Q37074416) (← links)
- Mode of virus rescue determines the acquisition of VHS mutations in VP22-negative herpes simplex virus 1. (Q37123122) (← links)
- Type I IFN response to Papiine herpesvirus 2 (Herpesvirus papio 2; HVP2) determines neuropathogenicity in mice (Q37311931) (← links)
- The nuclear-cytoplasmic shuttling of virion host shutoff RNase is enabled by pUL47 and an embedded nuclear export signal and defines the sites of degradation of AU-rich and stable cellular mRNAs (Q37336666) (← links)