Pages that link to "Q35844884"
Jump to navigation
Jump to search
The following pages link to Overlapping functions between XLF repair protein and 53BP1 DNA damage response factor in end joining and lymphocyte development (Q35844884):
Displaying 28 items.
- The proteasomal de-ubiquitinating enzyme POH1 promotes the double-strand DNA break response (Q24297147) (← links)
- Functional redundancy between the XLF and DNA-PKcs DNA repair factors in V(D)J recombination and nonhomologous DNA end joining (Q24624166) (← links)
- Functional intersection of ATM and DNA-dependent protein kinase catalytic subunit in coding end joining during V(D)J recombination (Q28508673) (← links)
- Structural insights into NHEJ: building up an integrated picture of the dynamic DSB repair super complex, one component and interaction at a time (Q33914715) (← links)
- Hematopoietic stem cell dysfunction underlies the progressive lymphocytopenia in XLF/Cernunnos deficiency (Q34137010) (← links)
- XRCC4 and XLF form long helical protein filaments suitable for DNA end protection and alignment to facilitate DNA double strand break repair (Q34329896) (← links)
- XRCC4/XLF Interaction Is Variably Required for DNA Repair and Is Not Required for Ligase IV Stimulation (Q35917467) (← links)
- Lymphocyte development: integration of DNA damage response signaling (Q36846366) (← links)
- Loss of p53-mediated cell-cycle arrest, senescence and apoptosis promotes genomic instability and premature aging (Q37022020) (← links)
- PAXX and XLF DNA repair factors are functionally redundant in joining DNA breaks in a G1-arrested progenitor B-cell line (Q37281378) (← links)
- Peripheral subnuclear positioning suppresses Tcrb recombination and segregates Tcrb alleles from RAG2. (Q37353134) (← links)
- Synthetic lethality between PAXX and XLF in mammalian development (Q37383233) (← links)
- PAXX promotes KU accumulation at DNA breaks and is essential for end-joining in XLF-deficient mice (Q37562697) (← links)
- A role for XLF in DNA repair and recombination in human somatic cells (Q37628142) (← links)
- Mechanisms of programmed DNA lesions and genomic instability in the immune system. (Q38078491) (← links)
- Functional overlaps between XLF and the ATM-dependent DNA double strand break response (Q38199848) (← links)
- XLF/Cernunnos: An important but puzzling participant in the nonhomologous end joining DNA repair pathway. (Q41918614) (← links)
- Deficiency of XLF and PAXX prevents DNA double-strand break repair by non-homologous end joining in lymphocytes (Q43141606) (← links)
- Role of Protein Linked DNA Breaks in Cancer (Q46243206) (← links)
- Synthetic lethality between murine DNA repair factors XLF and DNA-PKcs is rescued by inactivation of Ku70. (Q48008495) (← links)
- Normal development of mice lacking PAXX, the paralogue of XRCC4 and XLF. (Q52670821) (← links)
- Robust DNA repair in PAXX-deficient mammalian cells. (Q53839339) (← links)
- C-NHEJ without indels is robust and requires synergistic function of distinct XLF domains. (Q55349308) (← links)
- Kinase-dead ATR differs from ATR loss by limiting the dynamic exchange of ATR and RPA (Q60044506) (← links)
- MRI Is a DNA Damage Response Adaptor during Classical Non-homologous End Joining (Q64389222) (← links)
- Generation of a Mouse Model Lacking the Non-Homologous End-Joining Factor Mri/Cyren (Q91643258) (← links)
- CtIP is essential for early B cell proliferation and development in mice (Q92088877) (← links)
- Mediator of DNA Damage Checkpoint Protein 1 Facilitates V(D)J Recombination in Cells Lacking DNA Repair Factor XLF (Q92443688) (← links)