Pages that link to "Q35821927"
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The following pages link to Expression of dihydropyridine receptor (Ca2 channel) and calsequestrin genes in the myocardium of patients with end-stage heart failure (Q35821927):
Displaying 50 items.
- Acidosis and ischemia increase cellular Ca2 transient alternans and repolarization alternans susceptibility in the intact rat heart (Q33418232) (← links)
- Electrical remodeling in the failing heart (Q33792957) (← links)
- Electrophysiological remodeling in heart failure (Q33887264) (← links)
- Intracellular calcium and the relationship to contractility in an avian model of heart failure (Q34093037) (← links)
- Electrophysiological modeling of cardiac ventricular function: from cell to organ (Q34433124) (← links)
- Pharmacological support for children with myocardial dysfunction (Q34534627) (← links)
- Electrical and structural remodeling of the failing ventricle (Q34578568) (← links)
- Targeting calcium cycling proteins in heart failure through gene transfer (Q35037546) (← links)
- The long QT interval is not only inherited but is also linked to cardiac hypertrophy (Q35128926) (← links)
- Interleukin-1 beta modulates the growth and phenotype of neonatal rat cardiac myocytes (Q35753280) (← links)
- Molecular determinants of altered contractility in heart failure (Q35791489) (← links)
- Current perspectives of electrical remodeling and its therapeutic implications (Q35862167) (← links)
- Mechanisms of ventricular arrhythmias: a dynamical systems-based perspective (Q36042518) (← links)
- Ion channels as novel therapeutic targets in heart failure (Q36132294) (← links)
- Regulation of calcium channel expression in neonatal myocytes by catecholamines (Q36796737) (← links)
- In vivo and in vitro cardiac responses to beta-adrenergic stimulation in volume-overload heart failure (Q36968582) (← links)
- Electrophysiological consequences of dyssynchronous heart failure and its restoration by resynchronization therapy (Q37245080) (← links)
- Overexpression of the rat sarcoplasmic reticulum Ca2 ATPase gene in the heart of transgenic mice accelerates calcium transients and cardiac relaxation (Q37370078) (← links)
- Modification of cardiovascular ion channels by gene therapy (Q37578556) (← links)
- Electrical remodeling in dyssynchrony and resynchronization (Q37978197) (← links)
- Ca2 –calmodulin‐dependent protein kinase II represses cardiac transcription of the L‐type calcium channel α1C‐subunit gene (Cacna1c) by DREAM translocation (Q39559824) (← links)
- Single-channel properties of L-type calcium channels from failing human ventricle (Q40856643) (← links)
- Calcium transport proteins in the nonfailing and failing heart: gene expression and function (Q40958592) (← links)
- Calcium cycling proteins and force-frequency relationship in heart failure (Q41259282) (← links)
- Molecular and Cellular Mechanisms of Myocardial Failure (Q41669341) (← links)
- Diminished Ca 2 and Ba 2 Currents in Myocytes Surviving in the Epicardial Border Zone of the 5-Day Infarcted Canine Heart (Q41671949) (← links)
- The force—interval relationship in human myocardium (Q41675592) (← links)
- Reduced coronary vasodilator responses to amlodipine in pacing-induced heart failure in conscious dogs: role of nitric oxide (Q41806964) (← links)
- Changes in L-type calcium channel abundance and function during the transition to pacing-induced congestive heart failure (Q42456202) (← links)
- Impaired oxidative metabolism and calcium mishandling underlie cardiac dysfunction in a rat model of post-acute isoproterenol-induced cardiomyopathy. (Q42470577) (← links)
- Regulation of DHP receptor expression by elements in the 5'-flanking sequence (Q42486058) (← links)
- Cellular and molecular determinants of altered Ca2 handling in the failing rabbit heart: primary defects in SR Ca2 uptake and release mechanisms. (Q42535579) (← links)
- Myocardial phenotype changes in heart failure: cellular and subcellular adaptations and their functional significance (Q42578708) (← links)
- Doxycycline inducible expression of SERCA2a improves calcium handling and reverts cardiac dysfunction in pressure overload-induced cardiac hypertrophy (Q43892725) (← links)
- Calcium dynamics in the failing heart: restoration by beta-adrenergic receptor blockade (Q44370057) (← links)
- Tissue-specific expression of two human Ca(v)1.2 isoforms under the control of distinct 5' flanking regulatory elements (Q44495053) (← links)
- Sarcoplasmic reticulum Ca2 -ATPase overexpression by adenovirus mediated gene transfer and in transgenic mice (Q46112930) (← links)
- Modeling the cellular basis of altered excitation-contraction coupling in heart failure (Q46378454) (← links)
- Defective intracellular Ca2 homeostasis contributes to myocyte dysfunction during ventricular remodelling induced by chronic volume overload in rats (Q46697305) (← links)
- Human SERCA2a levels correlate inversely with age in senescent human myocardium (Q47671855) (← links)
- Calcineurin—the missing link in cardiac hypertrophy (Q47982927) (← links)
- Sarcoplasmic reticulum in heart failure: central player or bystander? (Q48008693) (← links)
- Alterations of calcium-regulatory proteins in heart failure (Q48008757) (← links)
- Uniformity of calcium channel number and isometric contraction in human right and left ventricular myocardium. (Q48165302) (← links)
- Effect of Glucose on 3D Cardiac Microtissues Derived from Human Induced Pluripotent Stem Cells (Q48332216) (← links)
- Developmental regulation of the L-type calcium channel alpha1C subunit expression in heart (Q52168216) (← links)
- Expression of calcium channel subunits in the normal and diseased human myocardium. (Q53007895) (← links)
- Calcium currents in postinfarction rat cardiac myocytes. (Q53999573) (← links)
- Early changes in excitation-contraction coupling: transition from compensated hypertrophy to failure in Dahl salt-sensitive rat myocytes (Q54135977) (← links)
- Calcium channels and cation transport ATPases in cardiac hypertrophy induced by aortic constriction in newborn rats (Q71918733) (← links)