Pages that link to "Q35782114"

The following pages link to Interleukin-4-induced macrophage fusion is prevented by inhibitors of mannose receptor activity (Q35782114):

Displaying 32 items.

• Foreign body-type multinucleated giant cell formation is potently induced by alpha-tocopherol and prevented by the diacylglycerol kinase inhibitor R59022 (Q24685285) (← links)
• Future challenges in the in vitro and in vivo evaluation of biomaterial biocompatibility (Q26752105) (← links)
• Gender differences in murine pulmonary responses elicited by cellulose nanocrystals (Q28396338) (← links)
• Inhalation of rod-like carbon nanotubes causes unconventional allergic airway inflammation (Q28396866) (← links)
• Macrophage fusion leading to foreign body giant cell formation persists under phagocytic stimulation by microspheres in vitro and in vivo in mouse models (Q33679249) (← links)
• Characterization of topographical effects on macrophage behavior in a foreign body response model (Q33721768) (← links)
• Characterization of Mannose Receptor-Dependent Phagocytosis Mediated byMycobacterium tuberculosisLipoarabinomannan (Q33754868) (← links)
• Alternative activation of tumor-associated macrophages by IL-4: priming for protumoral functions (Q34619781) (← links)
• Phenotypic expression in human monocyte-derived interleukin-4-induced foreign body giant cells and macrophages in vitro: dependence on material surface properties (Q34978459) (← links)
• Macrophage immunoregulatory pathways in tuberculosis (Q35039679) (← links)
• A Novel in vitro Human Macrophage Model to Study the Persistence of Mycobacterium tuberculosis Using Vitamin D(3) and Retinoic Acid Activated THP-1 Macrophages (Q35084568) (← links)
• The CC chemokine ligand, CCL2/MCP1, participates in macrophage fusion and foreign body giant cell formation (Q35103605) (← links)
• NF-κB signaling participates in both RANKL- and IL-4-induced macrophage fusion: receptor cross-talk leads to alterations in NF-κB pathways. (Q35147834) (← links)
• Immunoblot analysis of proteins associated with self‐assembled monolayer surfaces of defined chemistries (Q35163420) (← links)
• Profiles of carbohydrate ligands associated with adsorbed proteins on self-assembled monolayers of defined chemistries (Q35678201) (← links)
• Beta1 and beta2 integrins mediate adhesion during macrophage fusion and multinucleated foreign body giant cell formation (Q35747364) (← links)
• Extended culture of macrophages from different sources and maturation results in a common M2 phenotype. (Q35904647) (← links)
• Mannose receptor regulates myoblast motility and muscle growth (Q36118366) (← links)
• Foreign body-type multinucleated giant cell formation requires protein kinase C beta, delta, and zeta (Q36509305) (← links)
• Foreign body reaction to biomaterials (Q36575306) (← links)
• Macrophages in tuberculosis: friend or foe. (Q37147624) (← links)
• The foreign body reaction in T-cell-deficient mice (Q37396054) (← links)
• Quantitative in vivo cytokine analysis at synthetic biomaterial implant sites (Q37396113) (← links)
• Exploitation of the Macrophage Mannose Receptor (CD206) in Infectious Disease Diagnostics and Therapeutics. (Q37659134) (← links)
• The role of macrophage phenotype in vascularization of tissue engineering scaffolds (Q38978638) (← links)
• IL-4 abrogates osteoclastogenesis through STAT6-dependent inhibition of NF-kappaB (Q39946703) (← links)
• Macrophages, Foreign Body Giant Cells and Their Response to Implantable Biomaterials (Q41029580) (← links)
• Temporal and spatial distribution of macrophage phenotype markers in the foreign body response to glutaraldehyde-crosslinked gelatin hydrogels (Q42569323) (← links)
• Multi-nucleated giant cell formation from human cord blood monocytes in vitro, in comparison with adult peripheral blood monocytes (Q43279653) (← links)
• Protein-mediated macrophage adhesion and activation on biomaterials: a model for modulating cell behavior (Q51622731) (← links)
• Monocyte, macrophage and foreign body giant cell interactions with molecularly engineered surfaces (Q80527472) (← links)
• Electrospun Nanofiber Meshes With Endometrial MSCs Modulate Foreign Body Response by Increased Angiogenesis, Matrix Synthesis, and Anti-Inflammatory Gene Expression in Mice: Implication in Pelvic Floor (Q91710534) (← links)