Pages that link to "Q35748944"
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The following pages link to Single atom substitution in mouse protein kinase G eliminates oxidant sensing to cause hypertension (Q35748944):
Displaying 50 items.
- Reciprocal Control of the Circadian Clock and Cellular Redox State - a Critical Appraisal (Q26774796) (← links)
- Ca(2 ) signaling in the myocardium by (redox) regulation of PKA/CaMKII (Q26796230) (← links)
- Redox regulation of cGMP-dependent protein kinase Iα in the cardiovascular system (Q26800060) (← links)
- Nitric oxide synthases in heart failure (Q26830146) (← links)
- Abnormal Ca(2 ) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balance (Q26852587) (← links)
- The vascular Ca2 -sensing receptor regulates blood vessel tone and blood pressure (Q30835393) (← links)
- Biochemical methods for monitoring protein thiol redox states in biological systems (Q33857887) (← links)
- Oxidant-induced activation of cGMP-dependent protein kinase Iα mediates neuropathic pain after peripheral nerve injury. (Q34154543) (← links)
- Oxidative stress in atrial fibrillation: an emerging role of NADPH oxidase (Q34651371) (← links)
- Soluble guanylate cyclase is required for systemic vasodilation but not positive inotropy induced by nitroxyl in the mouse. (Q34892722) (← links)
- Prevention of PKG1α oxidation augments cardioprotection in the stressed heart. (Q35836147) (← links)
- Redox signaling and splicing dependent change in myosin phosphatase underlie early versus late changes in NO vasodilator reserve in a mouse LPS model of sepsis. (Q36000019) (← links)
- Endothelium-Derived Hyperpolarization and Coronary Vasodilation: Diverse and Integrated Roles of Epoxyeicosatrienoic Acids, Hydrogen Peroxide, and Gap Junctions. (Q36450226) (← links)
- Pathophysiology of hypertension in the absence of nitric oxide/cyclic GMP signaling (Q36532454) (← links)
- Hydrogen peroxide sensing and signaling by protein kinases in the cardiovascular system. (Q36596615) (← links)
- Structures of cGMP-Dependent Protein Kinase (PKG) Iα Leucine Zippers Reveal an Interchain Disulfide Bond Important for Dimer Stability (Q36818087) (← links)
- Diverse Functions of Endothelial NO Synthases System: NO and EDH. (Q36891648) (← links)
- Protein kinase G oxidation is a major cause of injury during sepsis. (Q36932174) (← links)
- Old dog, new tricks: novel cardiac targets and stress regulation by protein kinase G. (Q37075920) (← links)
- Roles for cytosolic NADPH redox in regulating pulmonary artery relaxation by thiol oxidation-elicited subunit dimerization of protein kinase G1α (Q37093715) (← links)
- Disulfide-activated protein kinase G Iα regulates cardiac diastolic relaxation and fine-tunes the Frank-Starling response. (Q37393164) (← links)
- Pressure-induced oxidative activation of PKG enables vasoregulation by Ca2 sparks and BK channels. (Q37499325) (← links)
- A key role for tetrahydrobiopterin-dependent endothelial NOS regulation in resistance arteries: studies in endothelial cell tetrahydrobiopterin-deficient mice (Q37723167) (← links)
- cGMP becomes a drug target (Q37980659) (← links)
- Vasodilatory mechanisms of beta receptor blockade (Q38013691) (← links)
- Contribution of oxidative stress to endothelial dysfunction in hypertension (Q38066208) (← links)
- Dual roles of vascular-derived reactive oxygen species--with a special reference to hydrogen peroxide and cyclophilin A. (Q38177120) (← links)
- Meeting highlights from the 2013 European Society of Cardiology Heart Failure Association Winter Meeting on Translational Heart Failure Research (Q38181036) (← links)
- Role of 8-nitro-cGMP and its redox regulation in cardiovascular electrophilic signaling. (Q38188356) (← links)
- cGMP: transition from bench to bedside: a report of the 6th International Conference on cGMP Generators, Effectors and Therapeutic Implications (Q38220091) (← links)
- Mitochondrial oxidative metabolism and uncoupling proteins in the failing heart (Q38246367) (← links)
- Endothelial dysfunction and vascular disease - a 30th anniversary update (Q38680112) (← links)
- The activity of cGMP-dependent protein kinase Iα is not directly regulated by oxidation-induced disulfide formation at cysteine 43. (Q38709543) (← links)
- Heme Oxygenases in Cardiovascular Health and Disease (Q38823562) (← links)
- Oxidative Stress and Hypertensive Diseases (Q39018896) (← links)
- Responses to reductive stress in the cardiovascular system (Q39035367) (← links)
- Reactive oxygen species: key regulators in vascular health and diseases. (Q39254617) (← links)
- Nitroglycerin fails to lower blood pressure in redox-dead Cys42Ser PKG1α knock-in mouse (Q39626999) (← links)
- Role of the renal sympathetic nerve in renal glucose metabolism during the development of type 2 diabetes in rats. (Q40450518) (← links)
- Phosphodiesterase 5 Inhibition Limits Doxorubicin-induced Heart Failure by Attenuating Protein Kinase G Iα Oxidation. (Q41048633) (← links)
- Examining a role for PKG Iα oxidation in the pathogenesis of cardiovascular dysfunction during diet-induced obesity (Q41231498) (← links)
- Proof of Principle for a Novel Class of Antihypertensives That Target the Oxidative Activation of PKG Iα (Protein Kinase G Iα). (Q41305323) (← links)
- Oxidant sensor in the cGMP-binding pocket of PKGIα regulates nitroxyl-mediated kinase activity. (Q41565976) (← links)
- Increased hydrogen peroxide impairs angiotensin II contractions of afferent arterioles in mice after renal ischaemia-reperfusion injury (Q41628503) (← links)
- Dehydroepiandrosterone promotes pulmonary artery relaxation by NADPH oxidation-elicited subunit dimerization of protein kinase G 1α. (Q41943788) (← links)
- Novel role for protein kinase g oxidative activation in the vasodilator and antihypertensive actions of hydrogen sulfide (Q43004748) (← links)
- Cardiac Aging - Benefits of Exercise, Nrf2 Activation and Antioxidant Signaling (Q46288550) (← links)
- Endothelium-derived hyperpolarizing factor and hypertension (Q47729492) (← links)
- PGC-1α (Peroxisome Proliferator-Activated Receptor γ Coactivator 1-α) Overexpression in Coronary Artery Disease Recruits NO and Hydrogen Peroxide During Flow-Mediated Dilation and Protects Against Increased Intraluminal Pressure (Q47835770) (← links)
- Meeting report of the 8th International Conference on cGMP "cGMP: generators, effectors, and therapeutic implications" at Bamberg, Germany, from June 23 to 25, 2017. (Q47904506) (← links)