Pages that link to "Q35675287"
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The following pages link to Increased efficiency of the GABAA and GABAB receptor-mediated neurotransmission in the Ts65Dn mouse model of Down syndrome (Q35675287):
Displaying 44 items.
- Prospects for Improving Brain Function in Individuals with Down Syndrome (Q22252566) (← links)
- Mouse models of Down syndrome as a tool to unravel the causes of mental disabilities (Q26863005) (← links)
- Down Syndrome Cognitive Phenotypes Modeled in Mice Trisomic for All HSA 21 Homologues (Q27300997) (← links)
- Hippocampal Transcriptome Profile of Persistent Memory Rescue in a Mouse Model of THRA1 Mutation-Mediated Resistance to Thyroid Hormone. (Q27303040) (← links)
- Monoacylglycerol lipase inhibitor JZL184 improves behavior and neural properties in Ts65Dn mice, a model of down syndrome (Q28542515) (← links)
- GABAA receptor subtypes: Therapeutic potential in Down syndrome, affective disorders, schizophrenia, and autism (Q30421260) (← links)
- Modulation of GABAergic transmission in development and neurodevelopmental disorders: investigating physiology and pathology to gain therapeutic perspectives (Q33659830) (← links)
- A 'cure' for Down syndrome: what do parents want? (Q33747513) (← links)
- Environmental enrichment decreases GABAergic inhibition and improves cognitive abilities, synaptic plasticity, and visual functions in a mouse model of Down syndrome (Q35632680) (← links)
- Increased excitability and altered action potential waveform in cerebellar granule neurons of the Ts65Dn mouse model of Down syndrome (Q36075008) (← links)
- Molecular and cellular alterations in Down syndrome: toward the identification of targets for therapeutics (Q36111546) (← links)
- From abnormal hippocampal synaptic plasticity in down syndrome mouse models to cognitive disability in down syndrome (Q36111747) (← links)
- Adult-onset fluoxetine treatment does not improve behavioral impairments and may have adverse effects on the Ts65Dn mouse model of Down syndrome (Q36119092) (← links)
- Deficits in cognition and synaptic plasticity in a mouse model of Down syndrome ameliorated by GABAB receptor antagonists. (Q36138353) (← links)
- Propofol differentially inhibits the release of glutamate, γ-aminobutyric acid and glycine in the spinal dorsal horn of rats (Q36249350) (← links)
- Inter-Dependent Mechanisms Behind Cognitive Dysfunction, Vascular Biology and Alzheimer's Dementia in Down Syndrome: Multi-Faceted Roles of APP (Q36332872) (← links)
- Increased cortical synaptic activation of TrkB and downstream signaling markers in a mouse model of Down Syndrome (Q36399166) (← links)
- Weaker control of the electrical properties of cerebellar granule cells by tonically active GABAA receptors in the Ts65Dn mouse model of Down's syndrome. (Q37045414) (← links)
- Size does not always matter: Ts65Dn Down syndrome mice show cerebellum-dependent motor learning deficits that cannot be rescued by postnatal SAG treatment (Q37381873) (← links)
- The GABAergic Hypothesis for Cognitive Disabilities in Down Syndrome (Q37683060) (← links)
- New innovations: therapeutic opportunities for intellectual disabilities (Q38137883) (← links)
- GABAergic dysfunction in pediatric neuro-developmental disorders (Q38175971) (← links)
- Altered intrinsic and network properties of neocortical neurons in the Ts65Dn mouse model of Down syndrome (Q38565706) (← links)
- New Perspectives for the Rescue of Cognitive Disability in Down Syndrome (Q38607518) (← links)
- Improving Memory and Cognition in Individuals with Down Syndrome (Q38858096) (← links)
- Altered Intrinsic Pyramidal Neuron Properties and Pathway-Specific Synaptic Dysfunction Underlie Aberrant Hippocampal Network Function in a Mouse Model of Tauopathy. (Q38921687) (← links)
- Dentate gyrus mediates cognitive function in the Ts65Dn/DnJ mouse model of Down syndrome (Q39294662) (← links)
- GABAergic hyperinnervation of dentate granule cells in the Ts65Dn mouse model of down syndrome: Exploring the role of App. (Q39324113) (← links)
- GluN2B antagonism affects interneurons and leads to immediate and persistent changes in synaptic plasticity, oscillations, and behavior (Q40778694) (← links)
- Cognitive and pharmacological insights from the Ts65Dn mouse model of Down syndrome (Q41811378) (← links)
- Rodent models in Down syndrome research: impact and future opportunities. (Q46193918) (← links)
- Evidence that increased Kcnj6 gene dose is necessary for deficits in behavior and dentate gyrus synaptic plasticity in the Ts65Dn mouse model of Down syndrome (Q46652242) (← links)
- Aerobic exercise and a BDNF-mimetic therapy rescue learning and memory in a mouse model of Down syndrome (Q47155189) (← links)
- Decreasing the Expression of GABAA α5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome. (Q47666263) (← links)
- The medial temporal memory system in Down syndrome: Translating animal models of hippocampal compromise. (Q47765503) (← links)
- Reversing excitatory GABAAR signaling restores synaptic plasticity and memory in a mouse model of Down syndrome. (Q48278664) (← links)
- Differential Brain, Cognitive and Motor Profiles Associated with Partial Trisomy. Modeling Down Syndrome in Mice. (Q48306556) (← links)
- Altered distribution of hippocampal interneurons in the murine Down Syndrome model Ts65Dn (Q48455685) (← links)
- In vivo 1H MRS study in microlitre voxels in the hippocampus of a mouse model of Down syndrome at 11.7 T. (Q48622660) (← links)
- Reduced cortical neurotransmitter receptor complex levels in fetal Down syndrome brain (Q50211157) (← links)
- Human Models Are Needed for Studying Human Neurodevelopmental Disorders. (Q64927158) (← links)
- Unbalanced dendritic inhibition of CA1 neurons drives spatial-memory deficits in the Ts2Cje Down syndrome model (Q91062464) (← links)
- Timing of Treatment with the Flavonoid 7,8-DHF Critically Impacts on Its Effects on Learning and Memory in the Ts65Dn Mouse (Q92588893) (← links)
- Alterations of specific cortical GABAergic circuits underlie abnormal network activity in a mouse model of Down syndrome (Q98388079) (← links)