Pages that link to "Q35675089"
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The following pages link to The biology of CML blast crisis (Q35675089):
Displaying 50 items.
- Regulation of myeloid leukaemia by the cell-fate determinant Musashi (Q24634359) (← links)
- Impaired DNA replication within progenitor cell pools promotes leukemogenesis. (Q24816866) (← links)
- New Developments in Chronic Myeloid Leukemia: Implications for Therapy (Q26745669) (← links)
- The non-genomic loss of function of tumor suppressors: an essential role in the pathogenesis of chronic myeloid leukemia chronic phase (Q26749343) (← links)
- Management of Chronic Myeloid Leukemia Patients Resistant to Tyrosine Kinase Inhibitors Treatment (Q26766299) (← links)
- Transformation of Canine Lymphoma/Leukemia to More Aggressive Diseases: Anecdotes or Reality? (Q26776071) (← links)
- Functions of heterogeneous nuclear ribonucleoproteins in stem cell potency and differentiation (Q26849397) (← links)
- Role of the transcription factor Bcl11b in development and lymphomagenesis (Q27014084) (← links)
- Effect of cellular quiescence on the success of targeted CML therapy (Q28469312) (← links)
- Combination of two but not three current targeted drugs can improve therapy of chronic myeloid leukemia (Q28474737) (← links)
- Optimizing Combination Therapies with Existing and Future CML Drugs (Q28475255) (← links)
- Icaritin Shows Potent Anti-Leukemia Activity on Chronic Myeloid Leukemia In Vitro and In Vivo by Regulating MAPK/ERK/JNK and JAK2/STAT3 /AKT Signalings (Q28741022) (← links)
- Misfolding, Aggregation, and Disordered Segments in c-Abl and p53 in Human Cancer. (Q30374671) (← links)
- Requirement of c-Myb for p210(BCR/ABL)-dependent transformation of hematopoietic progenitors and leukemogenesis (Q30439716) (← links)
- Exosomes released by K562 chronic myeloid leukemia cells promote angiogenesis in a src-dependent fashion (Q30537455) (← links)
- Lis1 regulates asymmetric division in hematopoietic stem cells and in leukemia (Q30608477) (← links)
- A phase 2 trial of ponatinib in Philadelphia chromosome-positive leukemias (Q33411250) (← links)
- BCR/ABL modifies the kinetics and fidelity of DNA double-strand breaks repair in hematopoietic cells (Q33796384) (← links)
- Ceramide promotes apoptosis in chronic myelogenous leukemia-derived K562 cells by a mechanism involving caspase-8 and JNK (Q33810952) (← links)
- Effective targeting of quiescent chronic myelogenous leukemia stem cells by histone deacetylase inhibitors in combination with imatinib mesylate (Q33866597) (← links)
- Genomic Profiling of Chronic Myelogenous Leukemia: Basic and Clinical Approach (Q33888064) (← links)
- Drug resistance in cancer: principles of emergence and prevention (Q33892278) (← links)
- Methylation status of DDIT3 gene in chronic myeloid leukemia (Q33933648) (← links)
- p53 loss promotes acute myeloid leukemia by enabling aberrant self-renewal (Q33953631) (← links)
- Chronic myeloid leukemia: mechanisms of blastic transformation (Q33968037) (← links)
- Mechanisms of primary and secondary resistance to imatinib in chronic myeloid leukemia (Q34016866) (← links)
- GTPase regulator associated with the focal adhesion kinase (GRAF) transcript was down-regulated in patients with myeloid malignancies (Q34081636) (← links)
- Destabilization of Bcr-Abl/Jak2 Network by a Jak2/Abl Kinase Inhibitor ON044580 Overcomes Drug Resistance in Blast Crisis Chronic Myelogenous Leukemia (CML) (Q34082540) (← links)
- Cancer stem cells: a stride towards cancer cure? (Q34086837) (← links)
- The B cell mutator AID promotes B lymphoid blast crisis and drug resistance in chronic myeloid leukemia (Q34099472) (← links)
- Metabolism considerations for kinase inhibitors in cancer treatment (Q34134390) (← links)
- miR-153 sensitized the K562 cells to As2O3-induced apoptosis (Q34160845) (← links)
- The elusive chronic myeloid leukemia stem cell: does it matter and how do we eliminate it? (Q34167708) (← links)
- del(15q) is a recurrent minor-route cytogenetic abnormality in the clonal evolution of chronic myelogenous leukemia (Q34204486) (← links)
- Tyrosine kinase inhibition: a therapeutic target for the management of chronic-phase chronic myeloid leukemia (Q34272962) (← links)
- The proteolytic activity of separase in BCR-ABL-positive cells is increased by imatinib (Q34369606) (← links)
- Cancer stem cell plasticity and tumor hierarchy (Q34459650) (← links)
- Gene expression changes associated with progression and response in chronic myeloid leukemia (Q34479561) (← links)
- Blastic phase of chronic myelogenous leukemia (Q34514042) (← links)
- Expression and activity of Fyn mediate proliferation and blastic features of chronic myelogenous leukemia (Q34532048) (← links)
- Chronic myeloid leukaemia as a model of disease evolution in human cancer (Q34631354) (← links)
- Mutational spectrum of myeloid malignancies with inv(3)/t(3;3) reveals a predominant involvement of RAS/RTK signaling pathways (Q35102301) (← links)
- The molecular basis of myeloid malignancies (Q35103708) (← links)
- Surface morphology of leukemic cells from chronic myeloid leukemia under atomic force microscopy (Q35159204) (← links)
- The overwhelmingly positive response to Dasatinib of a patient with multiple blast crisis of chronic myeloid leukemia (Q35173962) (← links)
- BCL6-mediated repression of p53 is critical for leukemia stem cell survival in chronic myeloid leukemia (Q35441406) (← links)
- Stem cell and kinase activity-independent pathway in resistance of leukaemia to BCR-ABL kinase inhibitors (Q35459748) (← links)
- Characterization of miRNomes in acute and chronic myeloid leukemia cell lines (Q35543478) (← links)
- Clonal Evolution and Blast Crisis Correlate with Enhanced Proteolytic Activity of Separase in BCR-ABL b3a2 Fusion Type CML under Imatinib Therapy (Q35667438) (← links)
- Bmi1 reprograms CML B-lymphoid progenitors to become B-ALL–initiating cells (Q35669231) (← links)