Pages that link to "Q35576523"

The following pages link to An anticholinergic reverses motor control and corticostriatal LTD deficits in Dyt1 ΔGAG knock-in mice (Q35576523):

Displaying 41 items.

• Enhanced hippocampal long-term potentiation and fear memory in Btbd9 mutant mice (Q21090923) (← links)
• Striatal cholinergic dysfunction as a unifying theme in the pathophysiology of dystonia (Q28648007) (← links)
• Cholinergic dysregulation produced by selective inactivation of the dystonia-associated protein torsinA. (Q31061478) (← links)
• In vivo imaging reveals impaired connectivity across cortical and subcortical networks in a mouse model of DYT1 dystonia (Q31114074) (← links)
• Striatal dopamine D1-like receptor binding is unchanged in primary focal dystonia (Q33863502) (← links)
• Alteration of striatal dopaminergic neurotransmission in a mouse model of DYT11 myoclonus-dystonia (Q34206423) (← links)
• Prenatal ethanol exposure alters synaptic plasticity in the dorsolateral striatum of rat offspring via changing the reactivity of dopamine receptor (Q34390849) (← links)
• Decreased dopamine receptor 1 activity and impaired motor-skill transfer in Dyt1 ΔGAG heterozygous knock-in mice. (Q34866730) (← links)
• Pre-synaptic release deficits in a DYT1 dystonia mouse model. (Q34965241) (← links)
• Minimal Change in the cytoplasmic calcium dynamics in striatal GABAergic neurons of a DYT1 dystonia knock-in mouse model (Q35049257) (← links)
• Behavioral and electrophysiological characterization of Dyt1 heterozygous knockout mice. (Q35208664) (← links)
• Mutations in HPCA cause autosomal-recessive primary isolated dystonia (Q35266327) (← links)
• Improved motor performance in Dyt1 ΔGAG heterozygous knock-in mice by cerebellar Purkinje-cell specific Dyt1 conditional knocking-out (Q35878028) (← links)
• Functional activity of the sensorimotor cortex and cerebellum relates to cervical dystonia symptoms (Q36395669) (← links)
• Functional analysis of dopaminergic systems in a DYT1 knock-in mouse model of dystonia (Q36400471) (← links)
• Subtle microstructural changes of the striatum in a DYT1 knock-in mouse model of dystonia (Q36774120) (← links)
• Engineering animal models of dystonia (Q37239952) (← links)
• Dystonia as a network disorder: what is the role of the cerebellum? (Q37592345) (← links)
• Striatal cholinergic interneurons in isolated generalized dystonia-rationale and perspectives for stem cell-derived cellular models (Q38239643) (← links)
• Probing striatal microcircuitry to understand the functional role of cholinergic interneurons (Q38557880) (← links)
• Hyperkinetic disorders and loss of synaptic downscaling (Q38847727) (← links)
• Mouse model of rare TOR1A variant found in sporadic focal dystonia impairs domains affected in DYT1 dystonia patients and animal models (Q41890579) (← links)
• Electromyographic evidence in support of a knock-in mouse model of DYT1 Dystonia (Q42755733) (← links)
• Anticholinergic drugs rescue synaptic plasticity in DYT1 dystonia: role of M1 muscarinic receptors (Q42971243) (← links)
• Enhanced mu opioid receptor-dependent opioidergic modulation of striatal cholinergic transmission in DYT1 dystonia (Q44351170) (← links)
• The Anatomical Basis for Dystonia: The Motor Network Model (Q46481280) (← links)
• Forebrain knock-out of torsinA reduces striatal free-water and impairs whole-brain functional connectivity in a symptomatic mouse model of DYT1 dystonia. (Q47837712) (← links)
• Miniature release events of glutamate from hippocampal neurons are influenced by the dystonia-associated protein torsinA. (Q48161931) (← links)
• Early structural and functional plasticity alterations in a susceptibility period of DYT1 dystonia mouse striatum (Q50420863) (← links)
• Dystonia (Q57154121) (← links)
• Structure of the Golgi apparatus is not influenced by a GAG deletion mutation in the dystonia-associated gene Tor1a (Q58601587) (← links)
• RGS9-2 rescues dopamine D2 receptor levels and signaling in dystonia mouse models (Q60954719) (← links)
• Optogenetic augmentation of the hypercholinergic endophenotype in DYT1 knock-in mice induced erratic hyperactive movements but not dystonia (Q64104410) (← links)
• Loss of the dystonia gene Thap1 leads to transcriptional deficits that converge on common pathogenic pathways in dystonic syndromes (Q64786603) (← links)
• Striatal Cholinergic Interneurons in a Knock-in Mouse Model of L-DOPA-Responsive Dystonia (Q89575934) (← links)
• Dystonia: Sparse Synapses for D2 Receptors in Striatum of a DYT1 Knock-out Mouse Model (Q89633692) (← links)
• Decreased number of striatal cholinergic interneurons and motor deficits in dopamine receptor 2-expressing-cell-specific Dyt1 conditional knockout mice (Q90747980) (← links)
• Diverse Mechanisms Lead to Common Dysfunction of Striatal Cholinergic Interneurons in Distinct Genetic Mouse Models of Dystonia (Q92000933) (← links)
• Novel mutations in KMT2B offer pathophysiological insights into childhood-onset progressive dystonia (Q92532291) (← links)
• The Role of BTBD9 in Striatum and Restless Legs Syndrome (Q92831388) (← links)
• The abnormal firing of Purkinje cells in the knockin mouse model of DYT1 dystonia (Q99723276) (← links)