Pages that link to "Q35572691"
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The following pages link to Oxidized CaMKII causes cardiac sinus node dysfunction in mice. (Q35572691):
Displaying 50 items.
- Regulation of CaMKII signaling in cardiovascular disease (Q26785302) (← links)
- Atrial Fibrillation and Fibrosis: Beyond the Cardiomyocyte Centric View (Q26800997) (← links)
- Fibrosis: a structural modulator of sinoatrial node physiology and dysfunction (Q26828018) (← links)
- Ion Channels in the Heart (Q26829681) (← links)
- Regulation of thrombosis and vascular function by protein methionine oxidation (Q26865448) (← links)
- CaMKII oxidative activation and the pathogenesis of cardiac disease (Q26998988) (← links)
- Calmodulin-dependent protein kinase II: linking heart failure and arrhythmias (Q26999872) (← links)
- NADPH oxidases in heart failure: poachers or gamekeepers? (Q27001094) (← links)
- Mechanisms of altered Ca²⁺ handling in heart failure (Q27012758) (← links)
- New therapeutic targets in cardiology: arrhythmias and Ca2 /calmodulin-dependent kinase II (CaMKII) (Q27016402) (← links)
- Protein methionine oxidation augments reperfusion injury in acute ischemic stroke (Q27333918) (← links)
- Atrial Fibrillation: Epidemiology, Pathophysiology, and Clinical Outcomes (Q30234626) (← links)
- Diabetes increases mortality after myocardial infarction by oxidizing CaMKII (Q30540314) (← links)
- Microdomain-Specific Modulation of L-Type Calcium Channels Leads to Triggered Ventricular Arrhythmia in Heart Failure. (Q30817882) (← links)
- Role of RyR2 phosphorylation in heart failure and arrhythmias: protein kinase A-mediated hyperphosphorylation of the ryanodine receptor at serine 2808 does not alter cardiac contractility or cause heart failure and arrhythmias (Q33693920) (← links)
- Toward a hierarchy of mechanisms in CaMKII-mediated arrhythmia (Q33775402) (← links)
- Three ways to die suddenly: do they all require calcium calmodulin-dependent protein kinase II? (Q33957736) (← links)
- Mathematical modeling of physiological systems: an essential tool for discovery. (Q34089490) (← links)
- Atrial fibrillation and sinus node dysfunction in human ankyrin-B syndrome: a computational analysis (Q34329504) (← links)
- Oxidized Ca(2 )/calmodulin-dependent protein kinase II triggers atrial fibrillation. (Q34370565) (← links)
- Human sinoatrial node structure: 3D microanatomy of sinoatrial conduction pathways. (Q34507987) (← links)
- Role of sinoatrial node architecture in maintaining a balanced source-sink relationship and synchronous cardiac pacemaking (Q34574809) (← links)
- Oxidative stress in atrial fibrillation: an emerging role of NADPH oxidase (Q34651371) (← links)
- The multifunctional Ca²⁺/calmodulin-dependent kinase IIδ (CaMKIIδ) regulates arteriogenesis in a mouse model of flow-mediated remodeling (Q34949842) (← links)
- CaMKII is essential for the proasthmatic effects of oxidation (Q35091137) (← links)
- Cycle length restitution in sinoatrial node cells: a theory for understanding spontaneous action potential dynamics (Q35095636) (← links)
- Mitochondrial reprogramming induced by CaMKIIδ mediates hypertrophy decompensation (Q35132584) (← links)
- Calsequestrin 2 deletion causes sinoatrial node dysfunction and atrial arrhythmias associated with altered sarcoplasmic reticulum calcium cycling and degenerative fibrosis within the mouse atrial pacemaker complex1. (Q35176492) (← links)
- Mitochondrial-targeted antioxidant therapy decreases transforming growth factor-β-mediated collagen production in a murine asthma model (Q35207685) (← links)
- The mitochondrial uniporter controls fight or flight heart rate increases. (Q35438927) (← links)
- Function and dysfunction of human sinoatrial node (Q35658401) (← links)
- Burst pacemaker activity of the sinoatrial node in sodium-calcium exchanger knockout mice. (Q35701705) (← links)
- MyD88 mediated inflammatory signaling leads to CaMKII oxidation, cardiac hypertrophy and death after myocardial infarction. (Q35892464) (← links)
- Oxidation of CaMKII determines the cardiotoxic effects of aldosterone. (Q35903753) (← links)
- Modern perspectives on numerical modeling of cardiac pacemaker cell (Q35934714) (← links)
- The multifunctional Ca2 /calmodulin-dependent kinase II regulates vascular smooth muscle migration through matrix metalloproteinase 9. (Q35994104) (← links)
- Calmodulin 2 Mutation N98S Is Associated with Unexplained Cardiac Arrest in Infants Due to Low Clinical Penetrance Electrical Disorders (Q35995795) (← links)
- Accelerated junctional rhythm and nonalternans repolarization lability precede ventricular tachycardia in Casq2-/- mice. (Q36381286) (← links)
- Protective effects of kaempferol against cardiac sinus node dysfunction via CaMKII deoxidization (Q36431198) (← links)
- Molecular mechanisms underlying cardiac protein phosphatase 2A regulation in heart (Q36526222) (← links)
- Genetic inhibition of Na -Ca2 exchanger current disables fight or flight sinoatrial node activity without affecting resting heart rate (Q36582471) (← links)
- A Single Protein Kinase A or Calmodulin Kinase II Site Does Not Control the Cardiac Pacemaker Ca2 Clock (Q36584849) (← links)
- Atrial remodelling in atrial fibrillation: CaMKII as a nodal proarrhythmic signal (Q36650585) (← links)
- Two-Pore K Channel TREK-1 Regulates Sinoatrial Node Membrane Excitability. (Q36877219) (← links)
- Oxidant stress promotes disease by activating CaMKII. (Q37358999) (← links)
- Modeling CaMKII in cardiac physiology: from molecule to tissue (Q37550292) (← links)
- Oxidized CaMKII promotes asthma through the activation of mast cells (Q37558095) (← links)
- CaMKII in sinoatrial node physiology and dysfunction (Q37642573) (← links)
- Numerical Modeling Calcium and CaMKII Effects in the SA Node (Q37689500) (← links)
- Heart failure modulates electropharmacological characteristics of sinoatrial nodes. (Q37697674) (← links)