Pages that link to "Q35549266"
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The following pages link to Rac1 GTPase-deficient mouse lens exhibits defects in shape, suture formation, fiber cell migration and survival (Q35549266):
Displaying 30 items.
- Mutations in SIPA1L3 cause eye defects through disruption of cell polarity and cytoskeleton organization (Q28118377) (← links)
- Beta-1 integrin is important for the structural maintenance and homeostasis of differentiating fiber cells. (Q33792743) (← links)
- Loss of Sip1 leads to migration defects and retention of ectodermal markers during lens development. (Q34090015) (← links)
- Molecular mechanism of formation of cortical opacity in CRYAAN101D transgenic mice (Q34341725) (← links)
- L-type calcium channels play a critical role in maintaining lens transparency by regulating phosphorylation of aquaporin-0 and myosin light chain and expression of connexins (Q34755622) (← links)
- The cellular and molecular mechanisms of vertebrate lens development (Q34999320) (← links)
- Hemizygous Le-Cre transgenic mice have severe eye abnormalities on some genetic backgrounds in the absence of LoxP sites (Q35291610) (← links)
- Rap1 GTPase is required for mouse lens epithelial maintenance and morphogenesis (Q36103500) (← links)
- Uterine RAC1 via Pak1-ERM signaling directs normal luminal epithelial integrity conducive to on-time embryo implantation in mice (Q36751296) (← links)
- The lens actin filament cytoskeleton: Diverse structures for complex functions (Q38772098) (← links)
- The molecular mechanisms underlying lens fiber elongation (Q38789258) (← links)
- A GAL4-inducible transgenic tool kit for the in vivo modulation of Rho GTPase activity in zebrafish (Q40785765) (← links)
- Ionizing radiation response of primary normal human lens epithelial cells (Q41136921) (← links)
- Functional non-coding polymorphism in an EPHA2 promoter PAX2 binding site modifies expression and alters the MAPK and AKT pathways. (Q41565096) (← links)
- Intact and N- or C-terminal end truncated AQP0 function as open water channels and cell-to-cell adhesion proteins: end truncation could be a prelude for adjusting the refractive index of the lens to prevent spherical aberration (Q41819336) (← links)
- The neural cell adhesion molecule (NCAM) associates with and signals through p21-activated kinase 1 (Pak1). (Q44970664) (← links)
- Induction of Immune Surveillance of the Dysmorphogenic Lens. (Q47122994) (← links)
- Targeted deletion of RIC8A in mouse neural precursor cells interferes with the development of the brain, eyes, and muscles. (Q47832358) (← links)
- Signaling and Gene Regulatory Networks in Mammalian Lens Development (Q47863726) (← links)
- Crk proteins transduce FGF signaling to promote lens fiber cell elongation (Q48185910) (← links)
- Functional role for stable microtubules in lens fiber cell elongation (Q49663380) (← links)
- Sprouty gain of function disrupts lens cellular processes and growth by restricting RTK signaling. (Q50561111) (← links)
- AP-2α is required after lens vesicle formation to maintain lens integrity (Q50668949) (← links)
- N-cadherin regulates signaling mechanisms required for lens fiber cell elongation and lens morphogenesis (Q50974374) (← links)
- Upregulation of multiple signaling pathways by Dock5 deletion in epithelial cells. (Q54976938) (← links)
- Proteome-transcriptome analysis and proteome remodeling in mouse lens epithelium and fibers (Q57802761) (← links)
- FHL2 mediates podocyte Rac1 activation and foot process effacement in hypertensive nephropathy (Q64080545) (← links)
- Ankyrin-G regulated epithelial phenotype is required for mouse lens morphogenesis and growth (Q90615307) (← links)
- CB1R regulates CDK5 signaling and epigenetically controls Rac1 expression contributing to neurobehavioral abnormalities in mice postnatally exposed to ethanol (Q91133801) (← links)
- Calponin-3 deficiency augments contractile activity, plasticity, fibrogenic response and Yap/Taz transcriptional activation in lens epithelial cells and explants (Q93012770) (← links)