Pages that link to "Q35546584"
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The following pages link to Monogenic forms of insulin resistance: apertures that expose the common metabolic syndrome (Q35546584):
Displaying 25 items.
- Peroxisomal proliferator activated receptor-gamma deficiency in a Canadian kindred with familial partial lipodystrophy type 3 (FPLD3) (Q25257726) (← links)
- New insights from monogenic diabetes for "common" type 2 diabetes (Q26795455) (← links)
- New opportunities: harnessing induced pluripotency for discovery in diabetes and metabolism (Q26995562) (← links)
- The metabolic syndrome (Q29616309) (← links)
- Thematic review series: Adipocyte Biology. Lipodystrophies: windows on adipose biology and metabolism (Q34610713) (← links)
- Brain insulin controls adipose tissue lipolysis and lipogenesis (Q34701526) (← links)
- 11β-Hydroxysteroid dehydrogenase type 1 shRNA ameliorates glucocorticoid-induced insulin resistance and lipolysis in mouse abdominal adipose tissue (Q34800976) (← links)
- Yin and Yang of hypothalamic insulin and leptin signaling in regulating white adipose tissue metabolism (Q35659559) (← links)
- The metabolic syndrome among patients with cardiovascular disease in Accra, Ghana (Q35767966) (← links)
- Unbuckling lipodystrophy from insulin resistance and hypertension (Q35835637) (← links)
- Genetic and physiological insights into the metabolic syndrome (Q36125352) (← links)
- Sp1 transcription factor interaction with accumulated prelamin a impairs adipose lineage differentiation in human mesenchymal stem cells: essential role of sp1 in the integrity of lipid vesicles (Q36861511) (← links)
- Genetic forms of the cardiometabolic syndrome: what can they tell the clinician? (Q36904431) (← links)
- Adipokines mediate inflammation and insulin resistance (Q36920864) (← links)
- Long-term efficacy of leptin replacement in patients with Dunnigan-type familial partial lipodystrophy (Q36998109) (← links)
- Genetics of metabolic syndrome: is there a role for phenomics? (Q37167771) (← links)
- The metabolic syndrome as an endocrine disease: is there an effective pharmacotherapeutic strategy optimally targeting the pathogenesis? (Q37362674) (← links)
- Insulin Resistance in Obesity as the Underlying Cause for the Metabolic Syndrome (Q37801630) (← links)
- Lipin1 is a key factor for the maturation and maintenance of adipocytes in the regulatory network with CCAAT/enhancer-binding protein alpha and peroxisome proliferator-activated receptor gamma 2. (Q38359945) (← links)
- Peroxisome proliferator activating receptor-γ and the podocyte. (Q38815188) (← links)
- Obstructive sleep apnea in 2 women with familial partial lipodystrophy due to a heterozygous LMNA R482Q mutation (Q42374759) (← links)
- Altered pre-lamin A processing is a common mechanism leading to lipodystrophy. (Q42815664) (← links)
- A case of Dunnigan-type familial partial lipodystrophy (FPLD) due to lamin A/C (LMNA) mutations complicated by end-stage renal disease (Q46244222) (← links)
- Prioritization of candidate disease genes for metabolic syndrome by computational analysis of its defining phenotypes (Q46494646) (← links)
- Mechanisms of the components of the metabolic syndrome that predispose to diabetes and atherosclerotic CVD. (Q51476172) (← links)