Pages that link to "Q35233491"
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The following pages link to The Rel family: models for transcriptional regulation and oncogenic transformation (Q35233491):
Displaying 42 items.
- A novel interferon regulatory factor (IRF), IRF-10, has a unique role in immune defense and is induced by the v-Rel oncoprotein (Q24537405) (← links)
- Mutational analysis of the v-Rel dimerization interface reveals a critical role for v-Rel homodimers in transformation (Q24537610) (← links)
- Interaction of the v-Rel oncoprotein with cellular transcription factor Sp1 (Q24645607) (← links)
- The suppression of SH3BGRL is important for v-Rel-mediated transformation (Q28274150) (← links)
- Phosphorylation of I kappa B alpha precedes but is not sufficient for its dissociation from NF-kappa B (Q28615823) (← links)
- AP-1 factors play an important role in transformation induced by the v-rel oncogene (Q33786606) (← links)
- Avian reticuloendotheliosis virus strain A and spleen necrosis virus do not infect human cells (Q33795485) (← links)
- Multiple mutations contribute to the oncogenicity of the retroviral oncoprotein v-Rel (Q33797168) (← links)
- Mapping of a serine-rich domain essential for the transcriptional, antiapoptotic, and transforming activities of the v-Rel oncoprotein (Q33957101) (← links)
- Differential regulation of the inhibitor of apoptosis ch-IAP1 by v-rel and the proto-oncogene c-rel. (Q34386316) (← links)
- A novel PRD I and TG binding activity involved in virus-induced transcription of IFN-A genes. (Q34785077) (← links)
- GAL4-I kappa B alpha and GAL4-I kappa B gamma activate transcription by different mechanisms (Q34982290) (← links)
- The relocalization of v-Rel from the nucleus to the cytoplasm coincides with induction of expression of Ikba and nfkb1 and stabilization of I kappa B-alpha (Q35829918) (← links)
- Constitutive and inducible kappa B binding activities in the cytosol of v-Rel-transformed lymphoid cells. (Q35835885) (← links)
- Mutations in the DNA-binding and dimerization domains of v-Rel are responsible for altered kappa B DNA-binding complexes in transformed cells (Q35839572) (← links)
- Avian I kappa B alpha is transcriptionally induced by c-Rel and v-Rel with different kinetics (Q35845620) (← links)
- Moloney murine leukemia virus activates NF-kappa B (Q35862437) (← links)
- v-rel Induces ectopic expression of an adhesion molecule, DM-GRASP, during B-lymphoma development (Q36550173) (← links)
- Interaction of the v-Rel oncoprotein with NF-kappaB and IkappaB proteins: heterodimers of a transformation-defective v-Rel mutant and NF-2 are functional in vitro and in vivo (Q36557574) (← links)
- ch-IAP1, a Member of the Inhibitor-of-Apoptosis Protein Family, Is a Mediator of the Antiapoptotic Activity of the v-Rel Oncoprotein (Q36574102) (← links)
- v-rel induces expression of three avian immunoregulatory surface receptors more efficiently than c-rel. (Q36619719) (← links)
- Evolution of the oncogenic potential of v-rel: rel-induced expression of immunoregulatory receptors correlates with tumor development and in vitro transformation (Q36630907) (← links)
- Transformation of avian fibroblasts overexpressing the c-rel proto-oncogene and a variant of c-rel lacking 40 C-terminal amino acids. (Q36631040) (← links)
- In vivo evolution of c-rel oncogenic potential (Q36631973) (← links)
- Chronic human immunodeficiency virus type 1 infection stimulates distinct NF-kappa B/rel DNA binding activities in myelomonoblastic cells. (Q36651776) (← links)
- Temperature-sensitive transforming mutants of the v-rel oncogene (Q36654705) (← links)
- NF-kappa B p100 is one of the high-molecular-weight proteins complexed with the v-Rel oncoprotein in transformed chicken spleen cells. (Q36655517) (← links)
- Effect of CD28 signal transduction on c-Rel in human peripheral blood T cells (Q36671402) (← links)
- Functional interaction of the v-Rel and c-Rel oncoproteins with the TATA-binding protein and association with transcription factor IIB. (Q36824974) (← links)
- A mutant v-rel with increased ability to transform B lymphocytes (Q38300329) (← links)
- The v-rel oncogene promotes malignant T-cell leukemia/lymphoma in transgenic mice (Q38355384) (← links)
- Characterization of elements determining the dimerization properties of RelB and p50. (Q40016375) (← links)
- Overexpression of RelA in transgenic mouse thymocytes: specific increase in levels of the inhibitor protein I kappa B alpha (Q40016532) (← links)
- Activation of gga-miR-155 by reticuloendotheliosis virus T strain and its contribution to transformation (Q40361877) (← links)
- Differential interactions of Rel-NF-kappa B complexes with I kappa B alpha determine pools of constitutive and inducible NF-kappa B activity (Q40793381) (← links)
- Multiple hemopoietic defects and lymphoid hyperplasia in mice lacking the transcriptional activation domain of the c-Rel protein (Q41043956) (← links)
- Activation of the NF-kappaB transcription factor in a T-lymphocytic cell line by hypochlorous acid (Q41130879) (← links)
- IkappaBalpha overexpression delays tumor formation in v-rel transgenic mice. (Q41855462) (← links)
- Downregulation of RelA (p65) by Rapamycin Inhibits Murine Adipocyte Differentiation and Reduces Fat Mass of C57BL/6J Mice despite High Fat Diet (Q41868317) (← links)
- Increased expression of p50-NF-kappaB and constitutive activation of NF-kappaB transcription factors during mouse skin carcinogenesis (Q42481352) (← links)
- Developmental expression of the mouse c-rel proto-oncogene in hematopoietic organs (Q52214145) (← links)
- Mice lacking the transcription factor subunit Rel can clear an influenza infection and have functional anti-viral cytotoxic T cells but do not develop an optimal antibody response (Q57607842) (← links)