Pages that link to "Q35196491"

The following pages link to Age and α-synuclein expression interact to reveal a dependence of dopaminergic axons on endogenous Akt/PKB signaling. (Q35196491):

Displaying 10 items.

• Inhibition of rho kinase enhances survival of dopaminergic neurons and attenuates axonal loss in a mouse model of Parkinson's disease (Q24609725) (← links)
• The glucagon-like peptide 1 (GLP) receptor as a therapeutic target in Parkinson's disease: mechanisms of action (Q26768527) (← links)
• The loss of glucose-regulated protein 78 (GRP78) during normal aging or from siRNA knockdown augments human alpha-synuclein (α-syn) toxicity to rat nigral neurons (Q35612163) (← links)
• α-Synuclein elevation in human neurodegenerative diseases: experimental, pathogenetic, and therapeutic implications. (Q38040282) (← links)
• Review: Axon pathology in age-related neurodegenerative disorders (Q38050513) (← links)
• The role of the dysfunctional akt-related pathway in cancer: establishment and maintenance of a malignant cell phenotype, resistance to therapy, and future strategies for drug development (Q38175044) (← links)
• Neurotrophic effects of serum- and glucocorticoid-inducible kinase on adult murine mesencephalic dopamine neurons (Q42322825) (← links)
• Age-dependent alpha-synuclein accumulation is correlated with elevation of mitochondrial TRPC3 in the brains of monkeys and mice (Q48410950) (← links)
• Utility of Neuronal-Derived Exosomes to Examine Molecular Mechanisms That Affect Motor Function in Patients With Parkinson Disease: A Secondary Analysis of the Exenatide-PD Trial (Q64785443) (← links)
• Diabetes, a Contemporary Risk for Parkinson's Disease: Epidemiological and Cellular Evidences (Q91583464) (← links)