Pages that link to "Q35176492"
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The following pages link to Calsequestrin 2 deletion causes sinoatrial node dysfunction and atrial arrhythmias associated with altered sarcoplasmic reticulum calcium cycling and degenerative fibrosis within the mouse atrial pacemaker complex1. (Q35176492):
Displaying 46 items.
- Atrial Fibrillation and Fibrosis: Beyond the Cardiomyocyte Centric View (Q26800997) (← links)
- Fibrosis: a structural modulator of sinoatrial node physiology and dysfunction (Q26828018) (← links)
- Rationally engineered Troponin C modulates in vivo cardiac function and performance in health and disease (Q27329916) (← links)
- Inherited bradyarrhythmia: A diverse genetic background (Q28071683) (← links)
- Atrial arrhythmias in inherited arrhythmogenic disorders (Q28076679) (← links)
- The importance of Ca(2 )-dependent mechanisms for the initiation of the heartbeat (Q28085170) (← links)
- Fibroblast growth factor 23: a novel key to find hidden substrates of atrial fibrillation? (Q34061048) (← links)
- Human sinoatrial node structure: 3D microanatomy of sinoatrial conduction pathways. (Q34507987) (← links)
- Upregulation of adenosine A1 receptors facilitates sinoatrial node dysfunction in chronic canine heart failure by exacerbating nodal conduction abnormalities revealed by novel dual-sided intramural optical mapping (Q35068578) (← links)
- Potential effects of intrinsic heart pacemaker cell mechanisms on dysrhythmic cardiac action potential firing (Q35110428) (← links)
- Exposure to phthalates affects calcium handling and intercellular connectivity of human stem cell-derived cardiomyocytes (Q35208585) (← links)
- Burst pacemaker activity of the sinoatrial node in sodium-calcium exchanger knockout mice. (Q35701705) (← links)
- Graded Maximal Exercise Testing to Assess Mouse Cardio-Metabolic Phenotypes (Q35918375) (← links)
- STIM1-Ca2 signaling modulates automaticity of the mouse sinoatrial node. (Q36179318) (← links)
- Ablation of HRC alleviates cardiac arrhythmia and improves abnormal Ca handling in CASQ2 knockout mice prone to CPVT (Q36188530) (← links)
- Direct Evidence for Microdomain-Specific Localization and Remodeling of Functional L-Type Calcium Channels in Rat and Human Atrial Myocytes. (Q36398808) (← links)
- The RyR2-P2328S mutation downregulates Nav1.5 producing arrhythmic substrate in murine ventricles. (Q36688514) (← links)
- Tachycardia-bradycardia syndrome: Electrophysiological mechanisms and future therapeutic approaches (Review). (Q37713991) (← links)
- SK4 K channels are therapeutic targets for the treatment of cardiac arrhythmias (Q37734009) (← links)
- Sinus node dysfunction in catecholaminergic polymorphic ventricular tachycardia: risk factor and potential therapeutic target? (Q38238961) (← links)
- Electrophysiological and molecular mechanisms of paroxysmal atrial fibrillation (Q38834737) (← links)
- Age-dependent down-regulation of hyperpolarization-activated cyclic nucleotide-gated channel 4 causes deterioration of canine sinoatrial node function (Q38860361) (← links)
- Murine Electrophysiological Models of Cardiac Arrhythmogenesis (Q39038606) (← links)
- Evidence for a functional role of calsequestrin 2 in mouse atrium (Q39532352) (← links)
- Flecainide exerts paradoxical effects on sodium currents and atrial arrhythmia in murine RyR2-P2328S hearts. (Q41910718) (← links)
- Age-dependent atrial arrhythmic phenotype secondary to mitochondrial dysfunction in Pgc-1β deficient murine hearts (Q42650117) (← links)
- Accentuated vagal antagonism paradoxically increases ryanodine receptor calcium leak in long-term exercised Calsequestrin2 knockout mice (Q47655479) (← links)
- Contribution of small conductance K channels to sinoatrial node pacemaker activity: insights from atrial-specific Na /Ca2 exchange knockout mice. (Q48356846) (← links)
- Conditional ablation and conditional rescue models for Casq2 elucidate the role of development and of cell type specific expression of Casq2 in the CPVT2 phenotype (Q49922723) (← links)
- High-resolution Optical Mapping of the Mouse Sino-atrial Node (Q51057809) (← links)
- Remodeling of the aging heart : Sinus node dysfunction and atrial fibrillation (Q51840493) (← links)
- Alternating membrane potential/calcium interplay underlies repetitive focal activity in a genetic model of calcium-dependent atrial arrhythmias. (Q52654918) (← links)
- Ventricular pro-arrhythmic phenotype, arrhythmic substrate, ageing and mitochondrial dysfunction in peroxisome proliferator activated receptor-γ coactivator-1β deficient (Pgc-1β-/-) murine hearts. (Q55415269) (← links)
- German Cardiac Society Working Group on Cellular Electrophysiology state-of-the-art paper: impact of molecular mechanisms on clinical arrhythmia management (Q57283285) (← links)
- Gene and Protein Expression Profile of Selected Molecular Targets Mediating Electrophysiological Function in Deficient Murine Atria (Q58616505) (← links)
- Calcium-Mediated Oscillation in Membrane Potentials and Atrial-Triggered Activity in Atrial Cells of Casq2 Mutation Mice (Q59132147) (← links)
- Functional Microdomains in Heart's Pacemaker: A Step Beyond Classical Electrophysiology and Remodeling (Q59807352) (← links)
- Natriuretic Peptide Receptor-C Protects Against Angiotensin II-Mediated Sinoatrial Node Disease in Mice (Q60916085) (← links)
- The Hyperpolarization-Activated Cyclic-Nucleotide-Gated Channel Blocker Ivabradine Does Not Prevent Arrhythmias in Catecholaminergic Polymorphic Ventricular Tachycardia (Q89494185) (← links)
- Na/Ca exchange in the atrium: Role in sinoatrial node pacemaking and excitation-contraction coupling (Q89576527) (← links)
- Atropine-induced sinus tachycardia protects against exercise-induced ventricular arrhythmias in patients with catecholaminergic polymorphic ventricular tachycardia (Q89856015) (← links)
- Molecular and tissue mechanisms of catecholaminergic polymorphic ventricular tachycardia (Q89961129) (← links)
- Clinical Potential of Targeting Fibroblast Growth Factor-23 and αKlotho in the Treatment of Uremic Cardiomyopathy (Q90655716) (← links)
- Distinct mechanisms mediate pacemaker dysfunction associated with catecholaminergic polymorphic ventricular tachycardia mutations: Insights from computational modeling (Q93257610) (← links)
- Silencing miR-370-3p rescues funny current and sinus node function in heart failure (Q97537622) (← links)
- Sinoatrial Node Structure, Mechanics, Electrophysiology and the Chronotropic Response to Stretch in Rabbit and Mouse (Q98288747) (← links)