Pages that link to "Q35091760"

The following pages link to Quantitative effect of luxS gene inactivation on the fitness of Helicobacter pylori (Q35091760):

Displaying 13 items.

• Lack of genomic evidence of AI-2 receptors suggests a non-quorum sensing role for luxS in most bacteria (Q21263071) (← links)
• Enzyme-ligand interactions that drive active site rearrangements in the Helicobacter pylori 5′-methylthioadenosine/S-adenosylhomocysteine nucleosidase (Q27665126) (← links)
• In Helicobacter pylori, LuxS is a key enzyme in cysteine provision through a reverse transsulfuration pathway. (Q33648743) (← links)
• In Helicobacter pylori auto-inducer-2, but not LuxS/MccAB catalysed reverse transsulphuration, regulates motility through modulation of flagellar gene transcription (Q33651670) (← links)
• Analysis of protein expression regulated by the Helicobacter pylori ArsRS two-component signal transduction system (Q33769136) (← links)
• Helicobacter pylori perceives the quorum-sensing molecule AI-2 as a chemorepellent via the chemoreceptor TlpB (Q35959831) (← links)
• Quorum sensing dependent phenotypes and their molecular mechanisms in Campylobacterales (Q38194557) (← links)
• Helicobacter pylori HopQ outer membrane protein attenuates bacterial adherence to gastric epithelial cells. (Q39907550) (← links)
• Biofilm Formation by Helicobacter pylori and Its Involvement for Antibiotic Resistance (Q41158525) (← links)
• Autoinducer-2 production in Campylobacter jejuni contributes to chicken colonization (Q41783749) (← links)
• Functional analysis of luxS in the probiotic strain Lactobacillus rhamnosus GG reveals a central metabolic role important for growth and biofilm formation (Q42125989) (← links)
• The quorum-sensing molecule autoinducer 2 regulates motility and flagellar morphogenesis in Helicobacter pylori. (Q42412402) (← links)
• Regulatory Mechanisms of the LuxS/AI-2 System and Bacterial Resistance (Q92429989) (← links)