Pages that link to "Q35083218"

The following pages link to Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. (Q35083218):

Displaying 24 items.

• Targeting metabolism in breast cancer: How far we can go? (Q26767022) (← links)
• New developments in the pathogenesis and therapeutic targeting of the IDH1 mutation in glioma (Q26863394) (← links)
• Targeting mitochondria metabolism for cancer therapy (Q27027219) (← links)
• Silencing of GLS and overexpression of GLS2 genes cooperate in decreasing the proliferation and viability of glioblastoma cells (Q28299851) (← links)
• Kidney-type glutaminase (GLS1) is a biomarker for pathologic diagnosis and prognosis of hepatocellular carcinoma (Q35781367) (← links)
• Glutaminolysis Was Induced by TGF-β1 through PP2Ac Regulated Raf-MEK-ERK Signaling in Endothelial Cells (Q36128637) (← links)
• Key Roles of Glutamine Pathways in Reprogramming the Cancer Metabolism (Q36260036) (← links)
• Glutaminase inhibitor compound 968 inhibits cell proliferation and sensitizes paclitaxel in ovarian cancer (Q37393426) (← links)
• Glutaminolysis as a target for cancer therapy (Q38642387) (← links)
• A tale of two glutaminases: homologous enzymes with distinct roles in tumorigenesis (Q38767151) (← links)
• Glutamine Addiction In Gliomas. (Q39172398) (← links)
• MiR-513c suppresses neuroblastoma cell migration, invasion, and proliferation through direct targeting glutaminase (GLS). (Q48157973) (← links)
• Glutamine Metabolism in Gliomas (Q48417468) (← links)
• Expression of Gls and Gls2 glutaminase isoforms in astrocytes (Q48492368) (← links)
• Downregulation of GLS2 in glioblastoma cells is related to DNA hypermethylation but not to the p53 status. (Q55459904) (← links)
• Transfection with GLS2 Glutaminase (GAB) Sensitizes Human Glioblastoma Cell Lines to Oxidative Stress by a Common Mechanism Involving Suppression of the PI3K/AKT Pathway (Q61812918) (← links)
• Glutaminases in slowly proliferating gastroenteropancreatic neuroendocrine neoplasms/tumors (GEP-NETs): Selective overexpression of mRNA coding for the KGA isoform (Q86725890) (← links)
• Targeting Glutamine Addiction in Gliomas (Q89510879) (← links)
• Nuclear Translocation of Glutaminase GLS2 in Human Cancer Cells Associates with Proliferation Arrest and Differentiation (Q89637305) (← links)
• Targeting glutaminase 1 attenuates stemness properties in hepatocellular carcinoma by increasing reactive oxygen species and suppressing Wnt/beta-catenin pathway (Q90568402) (← links)
• GLS1 promotes proliferation in hepatocellular carcinoma cells via AKT/GSK3β/CyclinD1 pathway (Q91759159) (← links)
• A comparative pharmaco-metabolomic study of glutaminase inhibitors in glioma stem-like cells confirms biological effectiveness but reveals differences in target-specificity (Q93240382) (← links)
• Combined blockade of EGFR and glutamine metabolism in preclinical models of colorectal cancer (Q97540425) (← links)
• Circ_0016418 promotes melanoma development and glutamine catabolism by regulating the miR-605-5p/GLS axis (Q98386561) (← links)