Pages that link to "Q35026044"
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The following pages link to Olfactory deficits in mice overexpressing human wildtype alpha-synuclein (Q35026044):
Displaying 50 items.
- Highly pathogenic H5N1 influenza virus can enter the central nervous system and induce neuroinflammation and neurodegeneration (Q24649627) (← links)
- Genetically engineered mouse models of Parkinson's disease (Q26852964) (← links)
- A progressive mouse model of Parkinson's disease: the Thy1-aSyn ("Line 61") mice (Q26859072) (← links)
- The Progressive BSSG Rat Model of Parkinson's: Recapitulating Multiple Key Features of the Human Disease (Q27315090) (← links)
- The contribution of alpha synuclein to neuronal survival and function - Implications for Parkinson's disease (Q28068464) (← links)
- Circadian dysfunction may be a key component of the non-motor symptoms of Parkinson's disease: insights from a transgenic mouse model (Q28384426) (← links)
- Vanadium exposure induces olfactory dysfunction in an animal model of metal neurotoxicity (Q28388187) (← links)
- Olfactory deficits in an alpha-synuclein fly model of Parkinson's disease (Q28539195) (← links)
- Activation of inflammatory signaling by lipopolysaccharide produces a prolonged increase of voluntary alcohol intake in mice. (Q30500334) (← links)
- Holocranohistochemistry enables the visualization of α-synuclein expression in the murine olfactory system and discovery of its systemic anti-microbial effects (Q33734084) (← links)
- Lack of TRPM5-Expressing Microvillous Cells in Mouse Main Olfactory Epithelium Leads to Impaired Odor-Evoked Responses and Olfactory-Guided Behavior in a Challenging Chemical Environment (Q33787086) (← links)
- Behavioral phenotyping of mouse models of Parkinson's disease (Q33820609) (← links)
- Diagnosing premotor Parkinson's disease using a two-step approach combining olfactory testing and DAT SPECT imaging (Q34020893) (← links)
- Chronic administration of cholesterol oximes in mice increases transcription of cytoprotective genes and improves transcriptome alterations induced by alpha-synuclein overexpression in nigrostriatal dopaminergic neurons (Q34056025) (← links)
- A mouse model of non-motor symptoms in Parkinson's disease: focus on pharmacological interventions targeting affective dysfunctions (Q34094065) (← links)
- Intranasal NAP (davunetide) decreases tau hyperphosphorylation and moderately improves behavioral deficits in mice overexpressing α-synuclein (Q34292532) (← links)
- Inducible dopaminergic glutathione depletion in an alpha-synuclein transgenic mouse model results in age-related olfactory dysfunction (Q34441722) (← links)
- A pilot trial of the microtubule-interacting peptide (NAP) in mice overexpressing alpha-synuclein shows improvement in motor function and reduction of alpha-synuclein inclusions (Q34612993) (← links)
- Rasagiline ameliorates olfactory deficits in an alpha-synuclein mouse model of Parkinson's disease (Q34662060) (← links)
- Impairment of mitochondria in adult mouse brain overexpressing predominantly full-length, N-terminally acetylated human α-synuclein (Q34718257) (← links)
- Intact olfaction in a mouse model of multiple system atrophy. (Q34732028) (← links)
- Non-motor and motor features in LRRK2 transgenic mice. (Q34922393) (← links)
- Olfaction in three genetic and two MPTP-induced Parkinson's disease mouse models (Q35035322) (← links)
- Olfactory dysfunction and neurotransmitter disturbance in olfactory bulb of transgenic mice expressing human A53T mutant α-synuclein (Q35208334) (← links)
- A GCase chaperone improves motor function in a mouse model of synucleinopathy (Q35340158) (← links)
- The temporal expression pattern of alpha-synuclein modulates olfactory neurogenesis in transgenic mice (Q35593656) (← links)
- Analysis of striatal transcriptome in mice overexpressing human wild-type alpha-synuclein supports synaptic dysfunction and suggests mechanisms of neuroprotection for striatal neurons (Q35728813) (← links)
- α-Synuclein overexpressing transgenic mice show internal organ pathology and autonomic deficits (Q36034871) (← links)
- Impaired sense of smell and altered olfactory system in RAG-1(-∕-) immunodeficient mice (Q36038434) (← links)
- α-Synuclein induces alterations in adult neurogenesis in Parkinson disease models via p53-mediated repression of Notch1. (Q36234694) (← links)
- Animal models of the non-motor features of Parkinson's disease (Q36235578) (← links)
- Early start of progressive motor deficits in Line 61 α-synuclein transgenic mice (Q36265332) (← links)
- Aβ alters the connectivity of olfactory neurons in the absence of amyloid plaques in vivo (Q36486453) (← links)
- Elevated tonic extracellular dopamine concentration and altered dopamine modulation of synaptic activity precede dopamine loss in the striatum of mice overexpressing human α‐synuclein (Q36585297) (← links)
- Olfactory assays for mouse models of neurodegenerative disease (Q36787949) (← links)
- Neuropsychiatric disease in murine lupus is dependent on the TWEAK/Fn14 pathway. (Q36902003) (← links)
- Alpha-synuclein overexpression in mice alters synaptic communication in the corticostriatal pathway (Q36999607) (← links)
- Research on the premotor symptoms of Parkinson's disease: clinical and etiological implications (Q37374203) (← links)
- Drug-induced Parkinson's disease modulates protein kinase A and Olfactory Marker Protein in the mouse olfactory bulb. (Q37607727) (← links)
- Cognitive deficits in a mouse model of pre-manifest Parkinson's disease (Q37669038) (← links)
- Genetic animal models of Parkinson's disease (Q37765030) (← links)
- Genetic mouse models of Parkinson's disease The state of the art. (Q37796009) (← links)
- Parkinson's disease mouse models in translational research (Q37874160) (← links)
- Parkinson's disease, proteins, and prions: milestones (Q37882593) (← links)
- Modelling of Parkinson's disease in mice (Q37957494) (← links)
- α-Synuclein elevation in human neurodegenerative diseases: experimental, pathogenetic, and therapeutic implications. (Q38040282) (← links)
- The function of α-synuclein (Q38139561) (← links)
- Olfactory bulb involvement in neurodegenerative diseases. (Q38189727) (← links)
- Harnessing neurogenesis for the possible treatment of Parkinson's disease (Q38203839) (← links)
- Neural stem cells in Parkinson's disease: a role for neurogenesis defects in onset and progression (Q38268886) (← links)