Pages that link to "Q34705973"
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The following pages link to S-nitrosation of mitochondrial complex I depends on its structural conformation (Q34705973):
Displaying 50 items.
- Regulation of cardiovascular cellular processes by S-nitrosylation (Q24300987) (← links)
- Pivotal Importance of STAT3 in Protecting the Heart from Acute and Chronic Stress: New Advancement and Unresolved Issues (Q26776101) (← links)
- Antioxidant responses and cellular adjustments to oxidative stress (Q26800229) (← links)
- The redox interplay between nitrite and nitric oxide: From the gut to the brain (Q26996490) (← links)
- Ischemic A/D transition of mitochondrial complex I and its role in ROS generation (Q28074253) (← links)
- Protein S-nitrosylation and cardioprotection (Q30385401) (← links)
- Cardioprotection by S-nitrosation of a cysteine switch on mitochondrial complex I. (Q33606564) (← links)
- Redox regulation of antioxidants, autophagy, and the response to stress: implications for electrophile therapeutics. (Q33702016) (← links)
- On the mechanism(s) of membrane permeability transition in liver mitochondria of lamprey, Lampetra fluviatilis L.: insights from cadmium (Q33786799) (← links)
- Identification of Potential Protein Targets of Isothiocyanates by Proteomics (Q33991759) (← links)
- Regulation of mitochondrial processes by protein S-nitrosylation (Q34170269) (← links)
- NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats (Q34570408) (← links)
- Dietary nitrate increases arginine availability and protects mitochondrial complex I and energetics in the hypoxic rat heart. (Q34619501) (← links)
- The many roads to mitochondrial dysfunction in neuroimmune and neuropsychiatric disorders (Q35246024) (← links)
- Redox signalling and cardioprotection: translatability and mechanism (Q35286322) (← links)
- Selective inhibition of deactivated mitochondrial complex I by biguanides (Q35633876) (← links)
- OXPHOS mutations and neurodegeneration (Q36533623) (← links)
- Complex I within oxidatively stressed bovine heart mitochondria is glutathionylated on Cys-531 and Cys-704 of the 75-kDa subunit: potential role of CYS residues in decreasing oxidative damage (Q36861498) (← links)
- A mitochondria-targeted S-nitrosothiol modulates respiration, nitrosates thiols, and protects against ischemia-reperfusion injury. (Q37227938) (← links)
- Conformational change of mitochondrial complex I increases ROS sensitivity during ischemia (Q37231724) (← links)
- Challenges in elucidating structure and mechanism of proton pumping NADH:ubiquinone oxidoreductase (complex I). (Q37316694) (← links)
- Mitochondria as metabolizers and targets of nitrite (Q37605280) (← links)
- Gelsolin as therapeutic target in Alzheimer's disease (Q37740141) (← links)
- Nitrosylation and nitration of mitochondrial complex I in Parkinson's disease. (Q37785104) (← links)
- Mitochondrial mechanisms of neuroglobin's neuroprotection (Q38103249) (← links)
- Characterisation of the active/de-active transition of mitochondrial complex I. (Q38191082) (← links)
- Protein S-nitrosylation in preconditioning and postconditioning (Q38199441) (← links)
- Chemical modifications of respiratory complex I for structural and functional studies (Q38226104) (← links)
- Cysteine-mediated redox signalling in the mitochondria. (Q38292320) (← links)
- Caveolae and signalling in cancer (Q38388286) (← links)
- GSNOR Deficiency Enhances In Situ Skeletal Muscle Strength, Fatigue Resistance, and RyR1 S-Nitrosylation Without Impacting Mitochondrial Content and Activity (Q38412593) (← links)
- Cardiovascular Mitochondrial Dysfunction Induced by Cocaine: Biomarkers and Possible Beneficial Effects of Modulators of Oxidative Stress (Q38737300) (← links)
- Moving Forwards by Blocking Back-Flow: The Yin and Yang of MI Therapy (Q38760245) (← links)
- Molecular mechanism and physiological role of active-deactive transition of mitochondrial complex I. (Q38809592) (← links)
- Nitrosative Stress, Hypernitrosylation, and Autoimmune Responses to Nitrosylated Proteins: New Pathways in Neuroprogressive Disorders Including Depression and Chronic Fatigue Syndrome (Q38875536) (← links)
- NO control of mitochondrial function in normal and transformed cells (Q39143236) (← links)
- Modulation of the conformational state of mitochondrial complex I as a target for therapeutic intervention (Q39223384) (← links)
- Post-Translational Oxidative Modifications of Mitochondrial Complex I (NADH: Ubiquinone Oxidoreductase): Implications for Pathogenesis and Therapeutics in Human Diseases (Q39352425) (← links)
- Respiratory substrates regulate S-nitrosylation of mitochondrial proteins through a thiol-dependent pathway (Q39939581) (← links)
- Sulforaphane Induced Apoptosis via Promotion of Mitochondrial Fusion and ERK1/2-Mediated 26S Proteasome Degradation of Novel Pro-survival Bim and Upregulation of Bax in Human Non-Small Cell Lung Cancer Cells (Q41243369) (← links)
- ND3, ND1 and 39kDa subunits are more exposed in the de-active form of bovine mitochondrial complex I. (Q41875296) (← links)
- The deactive form of respiratory complex I from mammalian mitochondria is a Na /H antiporter (Q42021655) (← links)
- Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury? (Q42044272) (← links)
- The Chemical Interplay between Nitric Oxide and Mitochondrial Cytochrome c Oxidase: Reactions, Effectors and Pathophysiology (Q42234432) (← links)
- Measuring mitochondrial protein thiol redox state (Q42977519) (← links)
- Nitrosation-modulating effect of ascorbate in a model dynamic system of coexisting nitric oxide and superoxide. (Q43148203) (← links)
- Effect of monovalent cations on the kinetics of hypoxic conformational change of mitochondrial complex I. (Q43217778) (← links)
- 'SNO'-Storms Compromise Protein Activity and Mitochondrial Metabolism in Neurodegenerative Disorders (Q46183247) (← links)
- Physiological Levels of Nitric Oxide Diminish Mitochondrial Superoxide. Potential Role of Mitochondrial Dinitrosyl Iron Complexes and Nitrosothiols (Q46184389) (← links)
- Mitochondrial complex I dysfunction and altered NAD(P)H kinetics in rat myocardium in cardiac right ventricular hypertrophy and failure. (Q51492667) (← links)