Pages that link to "Q34450885"

The following pages link to DCM-related tropomyosin mutants E40K/E54K over-inhibit the actomyosin interaction and lead to a decrease in the number of cycling cross-bridges. (Q34450885):

Displaying 10 items.

• Structural and protein interaction effects of hypertrophic and dilated cardiomyopathic mutations in alpha-tropomyosin. (Q34608346) (← links)
• Analysis of the molecular pathogenesis of cardiomyopathy-causing cTnT mutants I79N, ΔE96, and ΔK210. (Q36824519) (← links)
• Diversity and similarity of motor function and cross-bridge kinetics in papillary muscles of transgenic mice carrying myosin regulatory light chain mutations D166V and R58Q (Q37257335) (← links)
• A study of tropomyosin's role in cardiac function and disease using thin-filament reconstituted myocardium (Q38108764) (← links)
• Alpha-tropomyosin mutations in inherited cardiomyopathies (Q38134256) (← links)
• Using baculovirus/insect cell expressed recombinant actin to study the molecular pathogenesis of HCM caused by actin mutation A331P (Q38998926) (← links)
• Noncanonical roles for Tropomyosin during myogenesis (Q47070003) (← links)
• The immediate effect of HCM causing actin mutants E99K and A230V on actin-Tm-myosin interaction in thin-filament reconstituted myocardium. (Q52783545) (← links)
• HCM and DCM cardiomyopathy-linked α-tropomyosin mutations influence off-state stability and crossbridge interaction on thin filaments. (Q53416334) (← links)
• Thin filament dysfunctions caused by mutations in tropomyosin Tpm3.12 and Tpm1.1. (Q91597359) (← links)