Pages that link to "Q34426387"

The following pages link to mTOR attenuates the inflammatory response in cardiomyocytes and prevents cardiac dysfunction in pathological hypertrophy (Q34426387):

Displaying 50 items.

• Crosstalk between the heart and peripheral organs in heart failure (Q26766491) (← links)
• Deletion of MLIP (muscle-enriched A-type lamin-interacting protein) leads to cardiac hyperactivation of Akt/mammalian target of rapamycin (mTOR) and impaired cardiac adaptation (Q28267272) (← links)
• Leucine-enriched essential amino acids attenuate inflammation in rat muscle and enhance muscle repair after eccentric contraction (Q28386920) (← links)
• Impairment of Excitation-Contraction Coupling in Right Ventricular Hypertrophied Muscle with Fibrosis Induced by Pulmonary Artery Banding (Q28469354) (← links)
• Swimming exercise training-induced left ventricular hypertrophy involves microRNAs and synergistic regulation of the PI3K/AKT/mTOR signaling pathway (Q29568312) (← links)
• Deficiency of cardiac Acyl-CoA synthetase-1 induces diastolic dysfunction, but pathologic hypertrophy is reversed by rapamycin (Q33719493) (← links)
• Cardio-Oncology: Cancer Therapy-related Cardiovascular Complications in a Molecular Targeted Era: New Concepts and Perspectives (Q33807542) (← links)
• Late-life rapamycin treatment reverses age-related heart dysfunction. (Q33903651) (← links)
• Mechanistic target of rapamycin complex 2 protects the heart from ischemic damage (Q34039918) (← links)
• BET bromodomains mediate transcriptional pause release in heart failure (Q34361491) (← links)
• Exercise protects against chronic β-adrenergic remodeling of the heart by activation of endothelial nitric oxide synthase (Q35165245) (← links)
• New insights into insulin resistance in the diabetic heart (Q35243945) (← links)
• Cardiac Insulin Resistance and MicroRNA Modulators (Q35251097) (← links)
• Caloric restriction and chronic inflammatory diseases (Q35353852) (← links)
• A novel cardioprotective p38-MAPK/mTOR pathway (Q35549070) (← links)
• Cardiac mTOR rescues the detrimental effects of diet-induced obesity in the heart after ischemia-reperfusion (Q35749631) (← links)
• Adaptive mechanisms to compensate for overnutrition-induced cardiovascular abnormalities (Q35789287) (← links)
• The antioxidant compound tert-butylhydroquinone activates Akt in myocardium, suppresses apoptosis and ameliorates pressure overload-induced cardiac dysfunction (Q35936057) (← links)
• PTPN11-associated mutations in the heart: has LEOPARD changed Its RASpots? (Q36025665) (← links)
• Cardiac mTOR protects the heart against ischemia-reperfusion injury (Q36115340) (← links)
• Loss of long-chain acyl-CoA synthetase isoform 1 impairs cardiac autophagy and mitochondrial structure through mechanistic target of rapamycin complex 1 activation. (Q36171264) (← links)
• Targets, trafficking, and timing of cardiac autophagy (Q36413143) (← links)
• AMPK attenuates microtubule proliferation in cardiac hypertrophy (Q36700031) (← links)
• Cardiac Stim1 Silencing Impairs Adaptive Hypertrophy and Promotes Heart Failure Through Inactivation of mTORC2/Akt Signaling (Q36791996) (← links)
• Sarcospan Regulates Cardiac Isoproterenol Response and Prevents Duchenne Muscular Dystrophy-Associated Cardiomyopathy (Q36836386) (← links)
• Regulation of fatty acid metabolism by mTOR in adult murine hearts occurs independently of changes in PGC-1α (Q37054943) (← links)
• Metabolomics reveals the mechanisms for the cardiotoxicity of Pinelliae Rhizoma and the toxicity-reducing effect of processing (Q37307148) (← links)
• The long noncoding RNA Chaer defines an epigenetic checkpoint in cardiac hypertrophy (Q37317831) (← links)
• Mammalian target of rapamycin signaling in cardiac physiology and disease. (Q37715817) (← links)
• Phosphoinositide-3 Kinase Signaling in Cardiac Hypertrophy and Heart Failure (Q37883418) (← links)
• Target of rapamycin (TOR)-based therapy for cardiomyopathy: evidence from zebrafish and human studies (Q38030250) (← links)
• Mechanisms and consequences of inflammatory signaling in the myocardium (Q38044254) (← links)
• Myocyte signalling in leucocyte recruitment to the heart (Q38185647) (← links)
• A Review of the Molecular Mechanisms Underlying the Development and Progression of Cardiac Remodeling. (Q38656290) (← links)
• Fuel availability and fate in cardiac metabolism: A tale of two substrates (Q38781072) (← links)
• Autophagy in cardiac metabolic control: Novel mechanisms for cardiovascular disorders (Q38837178) (← links)
• DDiT4L promotes autophagy and inhibits pathological cardiac hypertrophy in response to stress. (Q38936784) (← links)
• The alteration of protein prenylation induces cardiomyocyte hypertrophy through Rheb-mTORC1 signalling and leads to chronic heart failure. (Q38941373) (← links)
• The mTOR Signaling Pathway in Myocardial Dysfunction in Type 2 Diabetes Mellitus (Q39256900) (← links)
• Cardiac mTOR complex 2 preserves ventricular function in pressure-overload hypertrophy (Q40284379) (← links)
• Impact of caloric restriction on myocardial ischaemia/reperfusion injury and new therapeutic options to mimic its effects (Q40630534) (← links)
• The mammalian target of rapamycin modulates the immunoproteasome system in the heart (Q40664320) (← links)
• The Cardiomyocyte as a Source of Cytokines in Cardiac Injury (Q41256651) (← links)
• High-density lipoprotein protects cardiomyocytes from oxidative stress via the PI3K/mTOR signaling pathway (Q41627722) (← links)
• Rheb is a critical regulator of autophagy during myocardial ischemia: pathophysiological implications in obesity and metabolic syndrome. (Q41820920) (← links)
• Roles and controls of mTOR in the heart. Focus on "mTOR attenuates the inflammatory response in cardiomyocytes and prevents cardiac dysfunction in pathological hypertrophy". (Q42868139) (← links)
• Protective Effects of the Mechanistic Target of Rapamycin against Excess Iron and Ferroptosis in Cardiomyocytes (Q46263621) (← links)
• New Insights Into the Role of mTOR Signaling in the Cardiovascular System. (Q48506350) (← links)
• Effect of an acute mechanical stimulus on aortic structure in the transverse aortic constriction mouse model. (Q50535165) (← links)
• The activation of mTOR is required for monocyte pro-inflammatory response in patients with coronary artery disease (Q51312253) (← links)