Pages that link to "Q34156605"
Jump to navigation
Jump to search
The following pages link to Niemann-Pick type C mutations cause lipid traffic jam. (Q34156605):
Displaying 50 items.
- The Wilson disease protein ATP7B resides in the late endosomes with Rab7 and the Niemann-Pick C1 protein (Q24293217) (← links)
- Cholesterol trafficking is required for mTOR activation in endothelial cells (Q24620236) (← links)
- Deficiency of niemann-pick type C-1 protein impairs release of human immunodeficiency virus type 1 and results in Gag accumulation in late endosomal/lysosomal compartments (Q24644759) (← links)
- Rab8-dependent recycling promotes endosomal cholesterol removal in normal and sphingolipidosis cells (Q24676084) (← links)
- Saccharomyces cerevisiae Npc2p is a functionally conserved homologue of the human Niemann-Pick disease type C 2 protein, hNPC2. (Q27937861) (← links)
- Drug-induced phospholipidosis (Q28264054) (← links)
- MLN64 mediates egress of cholesterol from endosomes to mitochondria in the absence of functional Niemann-Pick Type C1 protein (Q28266767) (← links)
- Bis(monoacylglycerol)phosphate as a non-invasive biomarker to monitor the onset and time-course of phospholipidosis with drug-induced toxicities (Q28278771) (← links)
- Different cellular traffic of LDL-cholesterol and acetylated LDL-cholesterol leads to distinct reverse cholesterol transport pathways (Q28505513) (← links)
- Cholesterol accumulation increases insulin granule size and impairs membrane trafficking (Q30525535) (← links)
- Tauists, Baptists, Syners, Apostates, and new data (Q30717885) (← links)
- Identification of a pharmaceutical compound that partially corrects the Niemann-Pick C phenotype in cultured cells (Q33184779) (← links)
- Automated microscopy screening for compounds that partially revert cholesterol accumulation in Niemann-Pick C cells (Q33227195) (← links)
- Itraconazole, a commonly used antifungal, inhibits Fcγ receptor-mediated phagocytosis: alteration of Fcγ receptor glycosylation and gene expression (Q33810233) (← links)
- Traffic jam: a compendium of human diseases that affect intracellular transport processes (Q34156525) (← links)
- Role of membrane organization and membrane domains in endocytic lipid trafficking (Q34156593) (← links)
- Roles of lipid rafts in membrane transport (Q34309076) (← links)
- A human genome-wide screen for regulators of clathrin-coated vesicle formation reveals an unexpected role for the V-ATPase (Q34319285) (← links)
- Late endosomes: sorting and partitioning in multivesicular bodies. (Q34368521) (← links)
- Cholesterol in brain disease: sometimes determinant and frequently implicated (Q34621446) (← links)
- Niemann-Pick disease type C1 is a sphingosine storage disease that causes deregulation of lysosomal calcium (Q34866369) (← links)
- Niemann-Pick disease type C. (Q35219271) (← links)
- Dynamics of endosomal sorting (Q35623173) (← links)
- Cholesterol accumulation is associated with lysosomal dysfunction and autophagic stress in Npc1 -/- mouse brain (Q35963203) (← links)
- Host but not parasite cholesterol controls Toxoplasma cell entry by modulating organelle discharge. (Q35973591) (← links)
- A caveolin dominant negative mutant associates with lipid bodies and induces intracellular cholesterol imbalance (Q36379994) (← links)
- Trafficking of endogenous smooth muscle cell cholesterol: a role for serum amyloid A and interleukin-1β (Q36574567) (← links)
- Mechanisms for cellular cholesterol transport: defects and human disease (Q36610465) (← links)
- Leishmania adaptor protein-1 subunits are required for normal lysosome traffic, flagellum biogenesis, lipid homeostasis, and adaptation to temperatures encountered in the mammalian host. (Q36846221) (← links)
- Intracellular sterol dynamics (Q37228012) (← links)
- A novel approach to analyze lysosomal dysfunctions through subcellular proteomics and lipidomics: the case of NPC1 deficiency (Q37612415) (← links)
- Sequestration of cholesterol within the host late endocytic pathway restricts liver-stage Plasmodium development (Q37699010) (← links)
- Invertebrate models of lysosomal storage disease: what have we learned so far? (Q37950897) (← links)
- Yeast lipid metabolism at a glance (Q38187442) (← links)
- Cognitive impairment in "Other" movement disorders: hidden defects and valuable clues (Q38206502) (← links)
- Sphingolipid lysosomal storage disorders (Q38217766) (← links)
- Liver X receptor agonist T0901317 reduces atherosclerotic lesions in apoE-/- mice by up-regulating NPC1 expression (Q38287462) (← links)
- Physiological and pathological roles of exosomes in the nervous system (Q38712290) (← links)
- Intracellular accumulation of amyloidogenic fragments of amyloid-beta precursor protein in neurons with Niemann-Pick type C defects is associated with endosomal abnormalities (Q38722206) (← links)
- The life cycle of phagosomes: formation, maturation, and resolution (Q38936522) (← links)
- Defective insulin receptor activation and altered lipid rafts in Niemann-Pick type C disease hepatocytes (Q39498801) (← links)
- Endocytic trafficking of sphingomyelin depends on its acyl chain length (Q40066539) (← links)
- Mucolipin-1 is a lysosomal membrane protein required for intracellular lactosylceramide traffic (Q40231477) (← links)
- Deficiency in ethanolamine plasmalogen leads to altered cholesterol transport (Q40611683) (← links)
- Overexpression of the myelin proteolipid protein leads to accumulation of cholesterol and proteolipid protein in endosomes/lysosomes: implications for Pelizaeus-Merzbacher disease (Q40737791) (← links)
- Intra-endosomal trafficking mediated by lysobisphosphatidic acid contributes to intracellular release of phosphorothioate-modified antisense oligonucleotides (Q41693957) (← links)
- Distribution and trafficking of MPR300 is normal in cells with cholesterol accumulated in late endocytic compartments: evidence for early endosome-to-TGN trafficking of MPR300. (Q41915571) (← links)
- Lysosomes, cholesterol and atherosclerosis (Q42122586) (← links)
- The transport of low density lipoprotein-derived cholesterol to the plasma membrane is defective in NPC1 cells (Q44319458) (← links)
- Concurrent increase of cholesterol, sphingomyelin and glucosylceramide in the spleen from non-neurologic Niemann-Pick type C patients but also patients possibly affected with other lipid trafficking disorders (Q44328450) (← links)