Pages that link to "Q34149775"
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The following pages link to Lipopolysaccharides directly decrease Ca2 oscillations and the hyperpolarization-activated nonselective cation current If in immortalized HL-1 cardiomyocytes (Q34149775):
Displaying 14 items.
- Inhibition of cardiac pacemaker channel hHCN2 depends on intercalation of lipopolysaccharide into channel-containing membrane microdomains (Q30317615) (← links)
- Enhanced monocyte chemoattractant protein-1 production in aging mice exaggerates cardiac depression during endotoxemia (Q34233617) (← links)
- Long-term culture of HL-1 cardiomyocytes in modular poly(ethylene glycol) microsphere-based scaffolds crosslinked in the phase-separated state (Q35581125) (← links)
- Comparison of mouse urinary metabolic profiles after exposure to the inflammatory stressors γ radiation and lipopolysaccharide (Q35775904) (← links)
- Lipopolysaccharide prolongs action potential duration in HL-1 mouse cardiomyocytes (Q36312795) (← links)
- The TRPM4 channel inhibitor 9-phenanthrol (Q37665584) (← links)
- Lipopolysaccharide and sepsis-associated myocardial dysfunction (Q37850260) (← links)
- Intracardiac origin of heart rate variability, pacemaker funny current and their possible association with critical illness. (Q38037663) (← links)
- Endoplasmic reticulum stress-mediated autophagy protects against lipopolysaccharide-induced apoptosis in HL-1 cardiomyocytes (Q38982529) (← links)
- I(f) blocking potency of ivabradine is preserved under elevated endotoxin levels in human atrial myocytes (Q40333918) (← links)
- Phosphoinositide-3-kinase/akt - dependent signaling is required for maintenance of [Ca(2 )](i), I(Ca), and Ca(2 ) transients in HL-1 cardiomyocytes (Q42202030) (← links)
- 9-Phenanthrol and flufenamic acid inhibit calcium oscillations in HL-1 mouse cardiomyocytes (Q42280458) (← links)
- Escherichia coli outer membrane vesicles can contribute to sepsis induced cardiac dysfunction (Q47151936) (← links)
- Toll-like receptor 4 activation promotes cardiac arrhythmias by decreasing the transient outward potassium current (Ito) through an IRF3-dependent and MyD88-independent pathway. (Q54326472) (← links)