Pages that link to "Q34105144"
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The following pages link to Hyperactive neuroendocrine secretion causes size, feeding, and metabolic defects of C. elegans Bardet-Biedl syndrome mutants (Q34105144):
Displaying 23 items.
- Conserved Genetic Interactions between Ciliopathy Complexes Cooperatively Support Ciliogenesis and Ciliary Signaling (Q27310130) (← links)
- Loss of a neural AMP-activated kinase mimics the effects of elevated serotonin on fat, movement, and hormonal secretions (Q27315375) (← links)
- Primary cilia in pancreatic development and disease (Q34421859) (← links)
- Identification and characterization of a novel allele of Caenorhabditis elegans bbs-7. (Q34653584) (← links)
- QTL analysis of dietary obesity in C57BL/6byj X 129P3/J F2 mice: diet- and sex-dependent effects (Q34901756) (← links)
- Kynurenic acid is a nutritional cue that enables behavioral plasticity (Q35101846) (← links)
- Whole organism transcriptome analysis of zebrafish models of Bardet-Biedl Syndrome and Alström Syndrome provides mechanistic insight into shared and divergent phenotypes (Q36008079) (← links)
- Differential effects on β-cell mass by disruption of Bardet-Biedl syndrome or Alstrom syndrome genes (Q36402564) (← links)
- Cell-cycle quiescence maintains Caenorhabditis elegans germline stem cells independent of GLP-1/Notch (Q36479362) (← links)
- C. elegans Body Cavity Neurons Are Homeostatic Sensors that Integrate Fluctuations in Oxygen Availability and Internal Nutrient Reserves (Q36618049) (← links)
- BBS7 is required for BBSome formation and its absence in mice results in Bardet-Biedl syndrome phenotypes and selective abnormalities in membrane protein trafficking (Q36920886) (← links)
- Insights and challenges in using C. elegans for investigation of fat metabolism (Q38250236) (← links)
- Metabolic regulation and energy homeostasis through the primary Cilium (Q38301656) (← links)
- From phenologs to silent suppressors: Identifying potential therapeutic targets for human disease. (Q38611619) (← links)
- Investigating Connections between Metabolism, Longevity, and Behavior in Caenorhabditis elegans (Q38862292) (← links)
- Genetic kidney diseases: Caenorhabditis elegans as model system (Q39292822) (← links)
- A new tool in C. elegans reveals changes in secretory protein metabolism in ire-1-deficient animals (Q42044312) (← links)
- Ciliary dysfunction impairs beta-cell insulin secretion and promotes development of type 2 diabetes in rodents (Q42468160) (← links)
- UNC-18 and Tomosyn Antagonistically Control Synaptic Vesicle Priming Downstream of UNC-13 in Caenorhabditis elegans (Q47933065) (← links)
- Kynurenic acid accumulation underlies learning and memory impairment associated with aging (Q49545472) (← links)
- Trafficking of ciliary membrane proteins by the intraflagellar transport/BBSome machinery (Q57026559) (← links)
- Primary Cilium, An Unsung Hero in Maintaining Functional β-cell Population (Q90206038) (← links)
- Global Transcriptome Changes That Accompany Alterations in Serotonin Levels in Caenorhabditis elegans (Q93036961) (← links)