Pages that link to "Q34006762"
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The following pages link to Paradoxical suppression of cellular senescence by p53 (Q34006762):
Displaying 50 items.
- Nutlin-3a, an MDM2 antagonist and p53 activator, helps to preserve the replicative potential of cancer cells treated with a genotoxic dose of resveratrol (Q24595171) (← links)
- Regulation of glucose metabolism by p53: emerging new roles for the tumor suppressor (Q24631421) (← links)
- Sestrins at the crossroad between stress and aging (Q24633564) (← links)
- Ribosomal proteins as unrevealed caretakers for cellular stress and genomic instability (Q26829490) (← links)
- Rapalogs in cancer prevention: anti-aging or anticancer? (Q26866151) (← links)
- Aberrant autolysosomal regulation is linked to the induction of embryonic senescence: differential roles of Beclin 1 and p53 in vertebrate Spns1 deficiency (Q27315143) (← links)
- NADPH oxidases: key modulators in aging and age-related cardiovascular diseases? (Q28072348) (← links)
- Redox control of senescence and age-related disease (Q28074230) (← links)
- P53-MDM2 Pathway: Evidences for A New Targeted Therapeutic Approach in B-Acute Lymphoblastic Leukemia (Q28078609) (← links)
- Senescence from G2 arrest, revisited (Q28088672) (← links)
- REDD1 Protects Osteoblast Cells from Gamma Radiation-Induced Premature Senescence (Q28384621) (← links)
- Activation of p53 by nutlin-3a induces apoptosis and cellular senescence in human glioblastoma multiforme (Q28477660) (← links)
- Suppression of replicative senescence by rapamycin in rodent embryonic cells (Q28571864) (← links)
- Rapamycin induces pluripotent genes associated with avoidance of replicative senescence (Q28572137) (← links)
- Big mice die young but large animals live longer (Q28704146) (← links)
- Elimination of proliferating cells unmasks the shift from senescence to quiescence caused by rapamycin (Q28744036) (← links)
- DNA damaging agents and p53 do not cause senescence in quiescent cells, while consecutive re-activation of mTOR is associated with conversion to senescence (Q28972513) (← links)
- Contact inhibition and high cell density deactivate the mammalian target of rapamycin pathway, thus suppressing the senescence program (Q28972583) (← links)
- Hyper-mitogenic drive coexists with mitotic incompetence in senescent cells (Q30533934) (← links)
- DNA damage causes TP53-dependent coupling of self-renewal and senescence pathways in embryonal carcinoma cells (Q30537160) (← links)
- Energy metabolism and metabolic sensors in stem cells: the metabostem crossroads of aging and cancer (Q30837218) (← links)
- New biomarkers probing depth of cell senescence assessed by laser scanning cytometry (Q33715537) (← links)
- Sustained activation of DNA damage response in irradiated apoptosis-resistant cells induces reversible senescence associated with mTOR downregulation and expression of stem cell markers (Q33728894) (← links)
- p53 Upregulation Is a Frequent Response to Deficiency of Cell-Essential Genes (Q33786749) (← links)
- From rapalogs to anti-aging formula (Q33829205) (← links)
- Avian reovirus p17 and σA act cooperatively to downregulate Akt by suppressing mTORC2 and CDK2/cyclin A2 and upregulating proteasome PSMB6. (Q33899583) (← links)
- Age-related susceptibility to apoptosis in human retinal pigment epithelial cells is triggered by disruption of p53-Mdm2 association (Q33961165) (← links)
- Targeting Phosphatidylinositide3-Kinase/Akt pathway by BKM120 for radiosensitization in hepatocellular carcinoma (Q33977001) (← links)
- mTOR favors senescence over quiescence in p53-arrested cells (Q34056457) (← links)
- Sodium dichloroacetate exhibits anti-leukemic activity in B-chronic lymphocytic leukemia (B-CLL) and synergizes with the p53 activator Nutlin-3. (Q34100963) (← links)
- Attenuation of replication stress-induced premature cellular senescence to assess anti-aging modalities (Q34145091) (← links)
- Androgen Receptor Drives Cellular Senescence (Q34189267) (← links)
- Immortalization of MEF is characterized by the deregulation of specific miRNAs with potential tumor suppressor activity (Q34201222) (← links)
- Hormesis, cell death and aging (Q34217654) (← links)
- Interaction between the Cockayne syndrome B and p53 proteins: implications for aging (Q34258129) (← links)
- Hypoxia suppresses conversion from proliferative arrest to cellular senescence (Q34291377) (← links)
- Unfolded p53 in the pathogenesis of Alzheimer's disease: is HIPK2 the link? (Q34322386) (← links)
- Berberine suppresses gero-conversion from cell cycle arrest to senescence (Q34366519) (← links)
- TOR-centric view on insulin resistance and diabetic complications: perspective for endocrinologists and gerontologists (Q34391710) (← links)
- Selective anti-cancer agents as anti-aging drugs (Q34392470) (← links)
- Resistance to discodermolide, a microtubule-stabilizing agent and senescence inducer, is 4E-BP1–dependent (Q34471836) (← links)
- Nuclear and cytoplasmic p53 suppress cell invasion by inhibiting respiratory complex-I activity via Bcl-2 family proteins (Q34483626) (← links)
- Biomarkers of cell senescence assessed by imaging cytometry (Q34538696) (← links)
- A molecular signature of normal breast epithelial and stromal cells from Li-Fraumeni syndrome mutation carriers (Q34575966) (← links)
- HdmX overexpression inhibits oncogene induced cellular senescence (Q34583158) (← links)
- p53 status as effect modifier of the association between pre-treatment fasting glucose and breast cancer outcomes in non diabetic, HER2 positive patients treated with trastuzumab. (Q34786457) (← links)
- Roles of the Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR pathways in controlling growth and sensitivity to therapy-implications for cancer and aging (Q34966603) (← links)
- Gerosuppression in confluent cells (Q34982499) (← links)
- Blockade of Hsp90 by 17AAG antagonizes MDMX and synergizes with Nutlin to induce p53-mediated apoptosis in solid tumors (Q35064987) (← links)
- Weekly administration of rapamycin improves survival and biomarkers in obese male mice on high-fat diet (Q35080619) (← links)