Pages that link to "Q33971060"
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The following pages link to Long-term persistance of the pathophysiologic response to severe burn injury (Q33971060):
Displaying 50 items.
- Beta adrenergic antagonists for hospitalized burned patients (Q24187666) (← links)
- Burns: an update on current pharmacotherapy (Q24618730) (← links)
- The Therapeutic Potential of Brown Adipocytes in Humans (Q26777229) (← links)
- Neuroendocrine regulation of inflammation (Q27001639) (← links)
- The impact of severe burns on skeletal muscle mitochondrial function (Q27026797) (← links)
- Time-Dependent and Organ-Specific Changes in Mitochondrial Function, Mitochondrial DNA Integrity, Oxidative Stress and Mononuclear Cell Infiltration in a Mouse Model of Burn Injury (Q28551267) (← links)
- Human herpes viruses in burn patients: A systematic review. (Q30248520) (← links)
- Burn injury, gender and cancer risk: population-based cohort study using data from Scotland and Western Australia (Q30736937) (← links)
- Screening of Key Genes in Severe Burn Injury at Different Stages via Analyzing Gene Expression Data (Q30870727) (← links)
- Nutrition and metabolism in burn patients (Q33568722) (← links)
- Catecholamine stress alters neutrophil trafficking and impairs wound healing by β2-adrenergic receptor-mediated upregulation of IL-6. (Q33579231) (← links)
- Impact of stress-induced diabetes on outcomes in severely burned children (Q33709684) (← links)
- Insulin effects on glucose tolerance, hypermetabolic response, and circadian-metabolic protein expression in a rat burn and disuse model (Q33840333) (← links)
- Effects of a caspase and a calpain inhibitor on resting energy expenditures in normal and hypermetabolic rats: a pilot study (Q33864903) (← links)
- Early rehabilitative exercise training in the recovery from pediatric burn (Q34005971) (← links)
- Animal models in burn research (Q34051592) (← links)
- Uncoupled skeletal muscle mitochondria contribute to hypermetabolism in severely burned adults. (Q34130829) (← links)
- Predictors of insulin resistance in pediatric burn injury survivors 24 to 36 months postburn (Q34173618) (← links)
- Leukocyte infiltration and activation of the NLRP3 inflammasome in white adipose tissue following thermal injury (Q34200054) (← links)
- Hepatic autophagy after severe burn in response to endoplasmic reticulum stress (Q34212838) (← links)
- Five-Year Outcomes after Oxandrolone Administration in Severely Burned Children: A Randomized Clinical Trial of Safety and Efficacy (Q34215250) (← links)
- Intravenous phage display identifies peptide sequences that target the burn-injured intestine (Q34409138) (← links)
- The role of exercise in the rehabilitation of patients with severe burns. (Q34747041) (← links)
- Inflammatory cytokines and their prognostic ability in cases of major burn injury (Q34749568) (← links)
- Peripherally inserted central venous catheter safety in burn care: a single-center retrospective cohort review (Q34860222) (← links)
- Endoplasmic reticulum stress in adipose tissue augments lipolysis (Q34885351) (← links)
- Changes in fat distribution in children following severe burn injury (Q34920165) (← links)
- Is the heat surrounding adipose tissue mitochondria warranted? (Q34993377) (← links)
- Predictors of muscle protein synthesis after severe pediatric burns (Q35222745) (← links)
- Activities of nonlysosomal proteolytic systems in skeletal and cardiac muscle during burn-induced hypermetabolism (Q35427527) (← links)
- Effects of pharmacological interventions on muscle protein synthesis and breakdown in recovery from burns. (Q35561653) (← links)
- Propranolol attenuates hemorrhage and accelerates wound healing in severely burned adults (Q35609600) (← links)
- Long-term mortality among older adults with burn injury: a population-based study in Australia (Q35670120) (← links)
- FIVE-YEAR OUTCOMES AFTER LONG-TERM OXANDROLONE ADMINISTRATION IN SEVERELY BURNED CHILDREN: A RANDOMIZED CLINICAL TRIAL. (Q35822989) (← links)
- Burn injury induces histopathological changes and cell proliferation in liver of rats. (Q35951997) (← links)
- Severe Burn Injury Induces Thermogenically Functional Mitochondria in Murine White Adipose Tissue. (Q35957477) (← links)
- Proteomics improves the prediction of burns mortality: results from regression spline modeling (Q36033465) (← links)
- Decreased skin-mediated detoxification contributes to oxidative stress and insulin resistance (Q36152041) (← links)
- Inflammation, organomegaly, and muscle wasting despite hyperphagia in a mouse model of burn cachexia. (Q36179118) (← links)
- Skeletal Muscle Loss is Associated with TNF Mediated Insufficient Skeletal Myogenic Activation After Burn (Q36190823) (← links)
- Skeletal Muscle Protein Breakdown Remains Elevated in Pediatric Burn Survivors up to One-Year Post-Injury. (Q36190868) (← links)
- β-Adrenergic blockade does not impair the skin blood flow sensitivity to local heating in burned and nonburned skin under neutral and hot environments in children. (Q36245313) (← links)
- Pathophysiologic Response to Burns in the Elderly (Q36251408) (← links)
- Burn plus lipopolysaccharide augments endoplasmic reticulum stress and NLRP3 inflammasome activation and reduces PGC-1α in liver (Q36254997) (← links)
- Burn Induces Browning of the Subcutaneous White Adipose Tissue in Mice and Humans. (Q36327596) (← links)
- Long-Term Skeletal Muscle Mitochondrial Dysfunction is Associated with Hypermetabolism in Severely Burned Children (Q36404885) (← links)
- Postburn Hypermetabolism: Past, Present, and Future (Q36405112) (← links)
- Long-term propranolol use in severely burned pediatric patients: a randomized controlled study. (Q36422015) (← links)
- Proteasome Inhibition After Burn Injury (Q36487994) (← links)
- 17β-Estradiol reappropriates mass lost to the hypermetabolic state in thermally injured rats (Q36492315) (← links)