Pages that link to "Q33920309"
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The following pages link to Loading-related regulation of gene expression in bone in the contexts of estrogen deficiency, lack of estrogen receptor alpha and disuse (Q33920309):
Displaying 43 items.
- Sost down-regulation by mechanical strain in human osteoblastic cells involves PGE2 signaling via EP4 (Q24309550) (← links)
- Distinct modes of inhibition by sclerostin on bone morphogenetic protein and Wnt signaling pathways (Q24621990) (← links)
- Activation of Wnt Signaling by Mechanical Loading Is Impaired in the Bone of Old Mice (Q33580683) (← links)
- Differential Gene Expression from Microarray Analysis Distinguishes Woven and Lamellar Bone Formation in the Rat Ulna following Mechanical Loading (Q34117489) (← links)
- Tibial loading increases osteogenic gene expression and cortical bone volume in mature and middle-aged mice (Q34238823) (← links)
- The Contribution of Experimental in vivo Models to Understanding the Mechanisms of Adaptation to Mechanical Loading in Bone (Q34272461) (← links)
- Sex steroid actions in male bone (Q34437537) (← links)
- Age-related impairment of bones' adaptive response to loading in mice is associated with sex-related deficiencies in osteoblasts but no change in osteocytes (Q34643886) (← links)
- A method for isolating high quality RNA from mouse cortical and cancellous bone (Q34802787) (← links)
- The impact of low-magnitude high-frequency vibration on fracture healing is profoundly influenced by the oestrogen status in mice (Q34818364) (← links)
- Ovariectomy enhances mechanical load-induced solute transport around osteocytes in rat cancellous bone (Q35174737) (← links)
- Conditional disruption of IGF-I gene in type 1α collagen-expressing cells shows an essential role of IGF-I in skeletal anabolic response to loading (Q35601229) (← links)
- Loading-related regulation of transcription factor EGR2/Krox-20 in bone cells is ERK1/2 protein-mediated and prostaglandin, Wnt signaling pathway-, and insulin-like growth factor-I axis-dependent (Q35763278) (← links)
- Mechanical loading-related changes in osteocyte sclerostin expression in mice are more closely associated with the subsequent osteogenic response than the peak strains engendered (Q35831180) (← links)
- Mechanical load increases in bone formation via a sclerostin-independent pathway. (Q35850660) (← links)
- Molecular genetic studies of gene identification for osteoporosis: the 2009 update (Q36003387) (← links)
- Mechanical regulation of signaling pathways in bone (Q36019769) (← links)
- A specific role for phosphoinositide 3-kinase and AKT in osteoblasts? (Q36105778) (← links)
- The importance of foetal movement for co-ordinated cartilage and bone development in utero : clinical consequences and potential for therapy. (Q36149873) (← links)
- Disuse rescues the age-impaired adaptive response to external loading in mice (Q36162301) (← links)
- Estrogen receptor-α is required for the osteogenic response to mechanical loading in a ligand-independent manner involving its activation function 1 but not 2. (Q36619427) (← links)
- Estrogen receptor α mediates proliferation of osteoblastic cells stimulated by estrogen and mechanical strain, but their acute down-regulation of the Wnt antagonist Sost is mediated by estrogen receptor β. (Q36724963) (← links)
- Transcriptional profiling of cortical versus cancellous bone from mechanically-loaded murine tibiae reveals differential gene expression (Q36804459) (← links)
- Screening of osteoprotegerin-related feature genes in osteoporosis and functional analysis with DNA microarray (Q37075242) (← links)
- The molecular basis of bone mechanotransduction (Q37421319) (← links)
- Old age and the associated impairment of bones' adaptation to loading are associated with transcriptomic changes in cellular metabolism, cell-matrix interactions and the cell cycle. (Q37477767) (← links)
- Disrupted mitochondrial function in the Opa3L122P mouse model for Costeff Syndrome impairs skeletal integrity (Q37530214) (← links)
- Sclerostin's role in bone's adaptive response to mechanical loading (Q37684437) (← links)
- Role of endocrine and paracrine factors in the adaptation of bone to mechanical loading (Q37850710) (← links)
- Is interaction between age-dependent decline in mechanical stimulation and osteocyte-estrogen receptor levels the culprit for postmenopausal-impaired bone formation? (Q38066386) (← links)
- Adaptive and Injury Response of Bone to Mechanical Loading (Q38090608) (← links)
- Nitric oxide signaling in mechanical adaptation of bone (Q38169910) (← links)
- Microarray gene expression during early healing of GBR-treated calvarial critical size defects. (Q38442845) (← links)
- Bone Homeostasis and Repair: Forced Into Shape. (Q38559011) (← links)
- Expression of muscle anabolic and metabolic factors in mechanically loaded MLO-Y4 osteocytes (Q39437832) (← links)
- Comparing histological, vascular and molecular responses associated with woven and lamellar bone formation induced by mechanical loading in the rat ulna (Q42572957) (← links)
- Bone mechanobiology in mice: toward single-cell in vivo mechanomics (Q48361504) (← links)
- Changes of serum sclerostin and Dickkopf-1 levels during the menstrual cycle. A pilot study. (Q48762542) (← links)
- Genetic loci that control the loss and regain of trabecular bone during unloading and reambulation. (Q51274396) (← links)
- Prostaglandin dependent control of an endogenous estrogen receptor agonist by osteoblasts. (Q55070816) (← links)
- Mechanically-stimulated ATP release from murine bone cells is regulated by a balance of injury and repair (Q57456946) (← links)
- The Bromodomain Inhibitor -Methyl pyrrolidone Prevents Osteoporosis and BMP-Triggered Sclerostin Expression in Osteocytes (Q58117027) (← links)
- NOTCH Signaling Is Activated through Mechanical Strain in Human Bone Marrow-Derived Mesenchymal Stromal Cells (Q64103459) (← links)