Pages that link to "Q33793485"
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The following pages link to Treatment with a C5aR antagonist decreases pathology and enhances behavioral performance in murine models of Alzheimer's disease (Q33793485):
Displaying 50 items.
- Complement component 5a receptor 1 (Q21496159) (← links)
- The C5a receptor antagonist PMX205 ameliorates experimentally induced colitis associated with increased IL-4 and IL-10 (Q24620635) (← links)
- Complement activation in the context of stem cells and tissue repair (Q26782203) (← links)
- Apolipoprotein E isoforms and regulation of the innate immune response in brain of patients with Alzheimer's disease (Q26822534) (← links)
- Antimicrobial peptides and complement in neonatal hypoxia-ischemia induced brain damage (Q26850135) (← links)
- Inflammatory process in Alzheimer's Disease (Q27500412) (← links)
- Treatment with the C5a receptor/CD88 antagonist PMX205 reduces inflammation in a murine model of allergic asthma (Q28390806) (← links)
- Complement: a key system for immune surveillance and homeostasis (Q29615484) (← links)
- The reemergence of long-term potentiation in aged Alzheimer's disease mouse model. (Q30379588) (← links)
- C1q enhances microglial clearance of apoptotic neurons and neuronal blebs, and modulates subsequent inflammatory cytokine production (Q33601747) (← links)
- New milestones ahead in complement-targeted therapy (Q33602320) (← links)
- Inhibition of C5a receptor alleviates experimental CNS lupus (Q33985628) (← links)
- A Novel Role for the Fifth Component of Complement (C5) in Cardiac Physiology (Q33988617) (← links)
- Retrieving novel C5aR antagonists using a hybrid ligand-based virtual screening protocol based on SVM classification and pharmacophore models. (Q34351611) (← links)
- CNS-specific expression of C3a and C5a exacerbate demyelination severity in the cuprizone model (Q34361334) (← links)
- Versatility of the complement system in neuroinflammation, neurodegeneration and brain homeostasis (Q34471200) (← links)
- Complementing the inflammasome. (Q34501875) (← links)
- Contribution of complement activation pathways to neuropathology differs among mouse models of Alzheimer's disease (Q34550347) (← links)
- Long-term treatment of thalidomide ameliorates amyloid-like pathology through inhibition of β-secretase in a mouse model of Alzheimer's disease (Q34584102) (← links)
- Apolipoprotein E isoform-dependent microglia migration (Q35003241) (← links)
- White matter injury due to experimental chronic cerebral hypoperfusion is associated with C5 deposition (Q35081055) (← links)
- Upregulation of glycolytic enzymes, mitochondrial dysfunction and increased cytotoxicity in glial cells treated with Alzheimer's disease plasma (Q35191570) (← links)
- Complement component C3 and butyrylcholinesterase activity are associated with neurodegeneration and clinical disability in multiple sclerosis. (Q35250966) (← links)
- C5a/CD88 signaling alters blood–brain barrier integrity in lupus through nuclear factor‐κB (Q35552398) (← links)
- Association of C5aR1genetic polymorphisms with coronary artery disease in a Han population in Xinjiang, China (Q35552902) (← links)
- Neuroinflammatory Cytokines-The Common Thread in Alzheimer's Pathogenesis (Q35815212) (← links)
- Active immunization against complement factor C5a: a new therapeutic approach for Alzheimer's disease (Q35957099) (← links)
- Improving the Fmoc Solid Phase Synthesis of the Cyclic Hexapeptide Complement C5a Antagonist, PMX205. (Q36031488) (← links)
- Tumor necrosis factor-α synthesis inhibitor 3,6'-dithiothalidomide attenuates markers of inflammation, Alzheimer pathology and behavioral deficits in animal models of neuroinflammation and Alzheimer's disease (Q36118204) (← links)
- Extensive innate immune gene activation accompanies brain aging, increasing vulnerability to cognitive decline and neurodegeneration: a microarray study (Q36164195) (← links)
- Targeting TNF-Alpha to Elucidate and Ameliorate Neuroinflammation in Neurodegenerative Diseases (Q36330960) (← links)
- Progress and Trends in Complement Therapeutics (Q36479278) (← links)
- Signaling pathways regulating neuron-glia interaction and their implications in Alzheimer's disease (Q36483870) (← links)
- Analysis of the Putative Role of CR1 in Alzheimer's Disease: Genetic Association, Expression and Function (Q36620401) (← links)
- Complement activation fragment C5a receptors, CD88 and C5L2, are associated with neurofibrillary pathology (Q36707253) (← links)
- Complement in immune and inflammatory disorders: pathophysiological mechanisms (Q36756850) (← links)
- Complement in immune and inflammatory disorders: therapeutic interventions (Q36756854) (← links)
- Similar connotation in chronic hepatitis B and nonalcoholic Fatty liver patients with dampness-heat syndrome (Q36801767) (← links)
- Dysregulation of the complement cascade in the hSOD1G93A transgenic mouse model of amyotrophic lateral sclerosis (Q37363769) (← links)
- The C5a anaphylatoxin receptor CD88 is expressed in presynaptic terminals of hippocampal mossy fibres. (Q37428162) (← links)
- Expression of complement system components during aging and amyloid deposition in APP transgenic mice (Q37443394) (← links)
- Recent developments in low molecular weight complement inhibitors (Q37456835) (← links)
- Protective effects of decay-accelerating factor on blast-induced neurotrauma in rats (Q37485157) (← links)
- Association of immune and metabolic receptors C5aR and C5L2 with adiposity in women (Q37553814) (← links)
- Complement expression in the retina is not influenced by short-term pressure elevation (Q37553920) (← links)
- The role of the complement system and the activation fragment C5a in the central nervous system (Q37599686) (← links)
- Ganglioside deficiency causes inflammation and neurodegeneration via the activation of complement system in the spinal cord (Q37702966) (← links)
- Microglia and inflammation in Alzheimer's disease (Q37703295) (← links)
- The role of tau in Alzheimer's disease and related disorders (Q37765632) (← links)
- Neuroinflammatory processes in Alzheimer's disease (Q37772809) (← links)