Pages that link to "Q33673823"
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The following pages link to Spontaneous development of endoplasmic reticulum stress that can lead to diabetes mellitus is associated with higher calcium-independent phospholipase A2 expression: a role for regulation by SREBP-1 (Q33673823):
Displaying 34 items.
- Roles and regulation of neutral sphingomyelinase-2 in cellular and pathological processes (Q27022944) (← links)
- Effects of Endoplasmic Reticulum Stress on Group VIA Phospholipase A2 in Beta Cells Include Tyrosine Phosphorylation and Increased Association with Calnexin* (Q28384113) (← links)
- Stimulation of autophagy improves endoplasmic reticulum stress-induced diabetes (Q28506554) (← links)
- Characterization of FKGK18 as inhibitor of group VIA Ca2 -independent phospholipase A2 (iPLA2β): candidate drug for preventing beta-cell apoptosis and diabetes (Q28535466) (← links)
- Group VIA Ca2 -independent phospholipase A2 (iPLA2beta) and its role in beta-cell programmed cell death (Q33886495) (← links)
- Evidence of contribution of iPLA2β-mediated events during islet β-cell apoptosis due to proinflammatory cytokines suggests a role for iPLA2β in T1D development (Q34068396) (← links)
- iPLA2β overexpression in smooth muscle exacerbates angiotensin II-induced hypertension and vascular remodeling (Q34171442) (← links)
- Inhibition of Ca2 -independent phospholipase A2β (iPLA2β) ameliorates islet infiltration and incidence of diabetes in NOD mice (Q35004275) (← links)
- Oxidative stress and redox modulation potential in type 1 diabetes. (Q35008614) (← links)
- Toxoplasma gondii prevalent in China induce weaker apoptosis of neural stem cells C17.2 via endoplasmic reticulum stress (ERS) signaling pathways (Q35066835) (← links)
- Group VIA Phospholipase A2 (iPLA2β) Modulates Bcl-x 5'-Splice Site Selection and Suppresses Anti-apoptotic Bcl-x(L) in β-Cells (Q35536316) (← links)
- Group VIA PLA2 (iPLA2β) is activated upstream of p38 mitogen-activated protein kinase (MAPK) in pancreatic islet β-cell signaling (Q35773997) (← links)
- Calcium-independent phospholipases A2 and their roles in biological processes and diseases. (Q35994344) (← links)
- Smooth muscle-specific expression of calcium-independent phospholipase A2β (iPLA2β) participates in the initiation and early progression of vascular inflammation and neointima formation (Q36098088) (← links)
- Hyperosmolarity-induced lipid droplet formation depends on ceramide production by neutral sphingomyelinase 2. (Q36302713) (← links)
- Group VIB Phospholipase A(2) promotes proliferation of INS-1 insulinoma cells and attenuates lipid peroxidation and apoptosis induced by inflammatory cytokines and oxidant agents (Q36415597) (← links)
- Genetic modulation of islet β-cell iPLA₂β expression provides evidence for its impact on β-cell apoptosis and autophagy (Q36870367) (← links)
- Eicosanoids in metabolic syndrome. (Q36911098) (← links)
- Hepatitis C virus infection induces inflammatory cytokines and chemokines mediated by the cross talk between hepatocytes and stellate cells (Q36978634) (← links)
- A link between endoplasmic reticulum stress‐induced β‐cell apoptosis and the group VIA Ca2 ‐independent phospholipase A2 (iPLA2β) (Q37019836) (← links)
- Role of calcium-independent phospholipase A(2)β in human pancreatic islet β-cell apoptosis (Q37174156) (← links)
- Recent progress in phospholipase A2 research: From cells to animals to humans (Q37824075) (← links)
- Ceramide formation as a target in beta-cell survival and function (Q37884074) (← links)
- Dysfunctional mitochondrial bioenergetics and oxidative stress in Akita( /Ins2)-derived β-cells (Q39131463) (← links)
- Unique sphingomyelin patches are targets of a beta-cell-specific antibody. (Q39508703) (← links)
- Polarization of Macrophages toward M2 Phenotype Is Favored by Reduction in iPLA2β (Group VIA Phospholipase A2) (Q42725788) (← links)
- Modeling Acute ER Stress in Vivo and in Vitro (Q42806718) (← links)
- Protective effects of dipeptidyl peptidase-4 (DPP-4) inhibitor against increased β cell apoptosis induced by dietary sucrose and linoleic acid in mice with diabetes (Q42821880) (← links)
- n-3 Polyunsaturated fatty acids protect against pancreatic β-cell damage due to ER stress and prevent diabetes development. (Q53837718) (← links)
- Toxoplasma gondii induce apoptosis of neural stem cells via endoplasmic reticulum stress pathway. (Q54369927) (← links)
- iPLAβ and its role in male fertility, neurological disorders, metabolic disorders, and inflammation (Q58609392) (← links)
- β-Lactones: A Novel Class of Ca2 -Independent Phospholipase A2 (Group VIA iPLA2) Inhibitors with the Ability To Inhibit β-Cell Apoptosis (Q91862174) (← links)
- Amorphous solid dispersion of Berberine mitigates apoptosis via iPLA2β/Cardiolipin/Opa1 pathway in db/db mice and in Palmitate-treated MIN6 β-cells (Q92135413) (← links)
- Thapsigargin induces apoptosis in adrenocortical carcinoma by activating endoplasmic reticulum stress and the JNK signaling pathway: an in vitro and in vivo study (Q93184661) (← links)