Pages that link to "Q33663946"

The following pages link to Cardiac extracellular matrix remodeling: fibrillar collagens and Secreted Protein Acidic and Rich in Cysteine (SPARC) (Q33663946):

Displaying 43 items.

• Defects in tendon, ligament, and enthesis in response to genetic alterations in key proteoglycans and glycoproteins: a review (Q21285116) (← links)
• Proteomics analysis of cardiac extracellular matrix remodeling in a porcine model of ischemia/reperfusion injury (Q24302184) (← links)
• Control of whole heart geometry by intramyocardial mechano-feedback: a model study (Q28480580) (← links)
• Cellular mechanisms of tissue fibrosis. 6. Purinergic signaling and response in fibroblasts and tissue fibrosis (Q33569405) (← links)
• Silencing SMOC2 ameliorates kidney fibrosis by inhibiting fibroblast to myofibroblast transformation (Q33578992) (← links)
• AMP-activated protein kinase regulates intraocular pressure, extracellular matrix, and cytoskeleton in trabecular meshwork (Q33621882) (← links)
• Age-dependent alterations in fibrillar collagen content and myocardial diastolic function: role of SPARC in post-synthetic procollagen processing. (Q33655983) (← links)
• SPARC/osteonectin functions to maintain homeostasis of the collagenous extracellular matrix in the periodontal ligament (Q34141724) (← links)
• Novel therapeutic strategies targeting fibroblasts and fibrosis in heart disease (Q34531894) (← links)
• The expression of SPARC in human intracranial aneurysms and its relationship with MMP-2/-9. (Q34629656) (← links)
• Anti-remodeling and anti-fibrotic effects of the neuregulin-1β glial growth factor 2 in a large animal model of heart failure (Q35070764) (← links)
• LPS induces greater bone and PDL loss in SPARC-null mice (Q35129266) (← links)
• Quantitative N-linked glycoproteomics of myocardial ischemia and reperfusion injury reveals early remodeling in the extracellular environment (Q35144131) (← links)
• SPARC mediates early extracellular matrix remodeling following myocardial infarction. (Q35159891) (← links)
• SPARC regulates collagen interaction with cardiac fibroblast cell surfaces (Q35315137) (← links)
• Endogenous Thrombospondin 1 Protects the Pressure-Overloaded Myocardium by Modulating Fibroblast Phenotype and Matrix Metabolism (Q35421651) (← links)
• Conditioned medium from human amniotic mesenchymal stromal cells limits infarct size and enhances angiogenesis (Q35552105) (← links)
• Changes in the myocardial interstitium and contribution to the progression of heart failure (Q35582221) (← links)
• The Influence of a Biologically Relevant Substratum Topography on Human Aortic and Umbilical Vein Endothelial Cells (Q35810059) (← links)
• The Function of SPARC as a Mediator of Fibrosis (Q36092348) (← links)
• Time course of right ventricular pressure-overload induced myocardial fibrosis: relationship to changes in fibroblast postsynthetic procollagen processing (Q36454243) (← links)
• Extracellular matrix and fibroblast communication following myocardial infarction (Q36457257) (← links)
• Overexpression of SPARC in human trabecular meshwork increases intraocular pressure and alters extracellular matrix (Q36827557) (← links)
• Aging and the cardiac collagen matrix: Novel mediators of fibrotic remodelling (Q37096257) (← links)
• The mineralocorticoid receptor promotes fibrotic remodeling in atrial fibrillation (Q37622571) (← links)
• Age and SPARC change the extracellular matrix composition of the left ventricle (Q37695656) (← links)
• SPARC: a key player in the pathologies associated with obesity and diabetes. (Q37701264) (← links)
• The myofibroblast matrix: implications for tissue repair and fibrosis. (Q37733670) (← links)
• Myocardial fibroblast-matrix interactions and potential therapeutic targets (Q37734270) (← links)
• Extracellular matrix roles during cardiac repair. (Q37776560) (← links)
• Role of matricellular proteins in cardiac tissue remodeling after myocardial infarction (Q37871839) (← links)
• Engineering cell platforms for myocardial regeneration. (Q37872006) (← links)
• Matricellular Proteins: New Molecular Targets To Prevent Heart Failure (Q37925022) (← links)
• From stem cells to cardiomyocytes: the role of forces in cardiac maturation, aging, and disease. (Q38235819) (← links)
• Cardiac Physiology of Aging: Extracellular Considerations. (Q38542448) (← links)
• Understanding the role of the extracellular matrix in cardiovascular development and disease: where do we go from here? (Q42065319) (← links)
• Atorvastatin Improves Ventricular Remodeling after Myocardial Infarction by Interfering with Collagen Metabolism (Q42364213) (← links)
• Osteonectin expression in surrounding stroma of craniopharyngiomas: association with recurrence rate and brain infiltration (Q44641509) (← links)
• Integration properties of Wharton's jelly-derived novel mesenchymal stem cells into ventricular slices of murine hearts (Q50673940) (← links)
• Differential impact of mechanical unloading on structural and nonstructural components of the extracellular matrix in advanced human heart failure (Q51453829) (← links)
• Predictive value of circulating osteonectin in patients with ischemic symptomatic chronic heart failure (Q52144083) (← links)
• Integration of Metabonomics and Transcriptomics Reveals the Therapeutic Effects and Mechanisms of Baoyuan Decoction for Myocardial Ischemia. (Q55017834) (← links)
• Recent advances in understanding the roles of T cells in pressure overload-induced cardiac hypertrophy and remodeling (Q91037375) (← links)