Pages that link to "Q30633722"
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The following pages link to Caspase-cleavage of tau is an early event in Alzheimer disease tangle pathology (Q30633722):
Displaying 50 items.
- Accumulation of transactive response DNA binding protein 43 in mild cognitive impairment and Alzheimer disease (Q24634907) (← links)
- Tau fragmentation, aggregation and clearance: the dual role of lysosomal processing (Q24656204) (← links)
- Intracellular Calcium Dysregulation: Implications for Alzheimer's Disease (Q26747203) (← links)
- Chronic Stress and Glucocorticoids: From Neuronal Plasticity to Neurodegeneration (Q26748974) (← links)
- The Metamorphic Nature of the Tau Protein: Dynamic Flexibility Comes at a Cost (Q26772846) (← links)
- Insulin in the brain: its pathophysiological implications for States related with central insulin resistance, type 2 diabetes and Alzheimer's disease (Q26866392) (← links)
- Tau clearance mechanisms and their possible role in the pathogenesis of Alzheimer disease (Q26995276) (← links)
- Tangles, Toxicity, and Tau Secretion in AD - New Approaches to a Vexing Problem (Q27004724) (← links)
- The role of tau in neurodegenerative diseases and its potential as a therapeutic target (Q27005032) (← links)
- Alzheimer's disease--a panorama glimpse (Q27011749) (← links)
- Metformin promotes tau aggregation and exacerbates abnormal behavior in a mouse model of tauopathy (Q27301246) (← links)
- Lysosomal dysfunction promotes cleavage and neurotoxicity of tau in vivo (Q27346877) (← links)
- Zinc-mediated Allosteric Inhibition of Caspase-6 (Q27671521) (← links)
- Blood biomarkers for brain injury: What are we measuring? (Q28067275) (← links)
- Mitochondrial defects and oxidative stress in Alzheimer disease and Parkinson disease (Q28280353) (← links)
- Multiple signaling factors and drugs alleviate neuronal death induced by expression of human and zebrafish tau proteins in vivo (Q28386141) (← links)
- Caspase-3 activation as a bifurcation point between plasticity and cell death. (Q30446892) (← links)
- Nucleation-dependent tau filament formation: the importance of dimerization and an estimation of elementary rate constants (Q30481896) (← links)
- β-Amyloid impairs axonal BDNF retrograde trafficking (Q30498160) (← links)
- Co-expression of truncated and full-length tau induces severe neurotoxicity (Q30829185) (← links)
- Low density lipoprotein receptor-related protein 1 promotes anti-apoptotic signaling in neurons by activating Akt survival pathway (Q33553429) (← links)
- Roles of tau protein in health and disease (Q33559303) (← links)
- ER stress in Alzheimer's disease: a novel neuronal trigger for inflammation and Alzheimer's pathology (Q33589732) (← links)
- Humanized monoclonal antibody armanezumab specific to N-terminus of pathological tau: characterization and therapeutic potency (Q33640390) (← links)
- Tau phosphorylation and cleavage in ethanol-induced neurodegeneration in the developing mouse brain (Q33763862) (← links)
- Altered cell viability and proliferation activity of peripheral lymphocytes in patients with Alzheimer's disease (Q33766609) (← links)
- Caspase cleavage of the amyloid precursor protein is prevented after overexpression of bcl-2 in a triple transgenic mouse model of Alzheimer's disease (Q33799351) (← links)
- A caspase cleaved form of tau is preferentially degraded through the autophagy pathway (Q33991036) (← links)
- Relationship between tau pathology and neuroinflammation in Alzheimer's disease (Q33995490) (← links)
- Downregulation of CREB expression in Alzheimer's brain and in Aβ-treated rat hippocampal neurons (Q33997810) (← links)
- Stress-induced tau phosphorylation: functional neuroplasticity or neuronal vulnerability? (Q34003380) (← links)
- The future of blood-based biomarkers for Alzheimer's disease (Q34026317) (← links)
- Hyperphosphorylation and cleavage at D421 enhance tau secretion (Q34277477) (← links)
- Structure and pathology of tau protein in Alzheimer disease (Q34280787) (← links)
- Cleavage of tau by asparagine endopeptidase mediates the neurofibrillary pathology in Alzheimer's disease. (Q34473854) (← links)
- Increased tau phosphorylation and tau truncation, and decreased synaptophysin levels in mutant BRI2/tau transgenic mice (Q34590082) (← links)
- Depletion of Beclin-1 due to proteolytic cleavage by caspases in the Alzheimer's disease brain (Q34733374) (← links)
- Neuroinflammation and J2 prostaglandins: linking impairment of the ubiquitin-proteasome pathway and mitochondria to neurodegeneration (Q34933261) (← links)
- The Hsp90 kinase co-chaperone Cdc37 regulates tau stability and phosphorylation dynamics. (Q34947846) (← links)
- Caspase activation precedes and leads to tangles (Q34965124) (← links)
- NO synthase 2 (NOS2) deletion promotes multiple pathologies in a mouse model of Alzheimer's disease (Q34998995) (← links)
- Suppression of Autophagy and Activation of Glycogen Synthase Kinase 3beta Facilitate the Aggregate Formation of Tau (Q35014018) (← links)
- Caspase Cleaved Tau in Alzheimer's Disease: A Therapeutic Target Realized. (Q35020903) (← links)
- PE859, a novel tau aggregation inhibitor, reduces aggregated tau and prevents onset and progression of neural dysfunction in vivo (Q35058484) (← links)
- The relationship between truncation and phosphorylation at the C-terminus of tau protein in the paired helical filaments of Alzheimer's disease (Q35071628) (← links)
- Increased protein aggregation in Zucker diabetic fatty rat brain: identification of key mechanistic targets and the therapeutic application of hydrogen sulfide (Q35083908) (← links)
- Caspase-mediated cleavage of glial fibrillary acidic protein within degenerating astrocytes of the Alzheimer's disease brain (Q35088000) (← links)
- Truncated tau and Aβ cooperatively impair mitochondria in primary neurons (Q35119001) (← links)
- Immunoelectron microscopic and biochemical studies of caspase-cleaved tau in a mouse model of tauopathy (Q35183072) (← links)
- Astrocytes are important mediators of Aβ-induced neurotoxicity and tau phosphorylation in primary culture. (Q35202286) (← links)