Pages that link to "Q30557138"
Jump to navigation
Jump to search
The following pages link to Dysapoptosis of osteoblasts and osteocytes increases cancellous bone formation but exaggerates cortical porosity with age. (Q30557138):
Displaying 24 items.
- Apoptotic osteocytes and the control of targeted bone resorption (Q27023670) (← links)
- Cell Death in Chondrocytes, Osteoblasts, and Osteocytes (Q28067045) (← links)
- Osteocyte apoptosis is required for production of osteoclastogenic signals following bone fatigue in vivo. (Q33803842) (← links)
- Estrogens and Androgens in Skeletal Physiology and Pathophysiology (Q34543980) (← links)
- Glycation of human cortical and cancellous bone captures differences in the formation of Maillard reaction products between glucose and ribose (Q35101624) (← links)
- Cyclic Compressive Stress Regulates Apoptosis in Rat Osteoblasts: Involvement of PI3K/Akt and JNK MAPK Signaling Pathways (Q36180524) (← links)
- The Effects of Aging and Sex Steroid Deficiency on the Murine Skeleton Are Independent and Mechanistically Distinct (Q37685177) (← links)
- Fate of growth plate hypertrophic chondrocytes: death or lineage extension? (Q38365059) (← links)
- A look behind the scenes: the risk and pathogenesis of primary osteoporosis (Q38434317) (← links)
- The Role of Osteocytes in Age-Related Bone Loss (Q38747978) (← links)
- Fluid shear stress suppresses TNF-α-induced apoptosis in MC3T3-E1 cells: Involvement of ERK5-AKT-FoxO3a-Bim/FasL signaling pathways (Q38779699) (← links)
- Childhood cancer chemotherapy-induced bone damage: pathobiology and protective effects of resveratrol and other nutraceuticals. (Q39405101) (← links)
- DNA damage and senescence in osteoprogenitors expressing Osx1 may cause their decrease with age. (Q40988880) (← links)
- Old age causes de novo intracortical bone remodeling and porosity in mice. (Q42281500) (← links)
- Fenofibrate decreases the bone quality by down regulating Runx2 in high-fat-diet induced Type 2 diabetes mellitus mouse model. (Q42377473) (← links)
- Preclinical Models for Skeletal Research: How Commonly Used Species Mimic (or Don't) Aspects of Human Bone (Q46300849) (← links)
- Autophagy relieves the function inhibition and apoptosis‑promoting effects on osteoblast induced by glucocorticoid (Q47329175) (← links)
- Fas- and Mitochondria-Mediated Signaling Pathway Involved in Osteoblast Apoptosis Induced by AlCl3. (Q47659384) (← links)
- Skeletal inflammation and attenuation of Wnt signaling, Wnt ligand expression, and bone formation in atherosclerotic ApoE-null mice. (Q48061664) (← links)
- The quest for osteoporosis mechanisms and rational therapies: How far we've come, how much further we need to go. (Q48499773) (← links)
- Fangchinoline Promotes Autophagy and Inhibits Apoptosis in Osteoporotic Rats (Q61449285) (← links)
- The impact of inhibiting apoptosis in osteoblasts and osteocytes (Q87094001) (← links)
- Multiple Modes of Vitamin K Actions in Aging-Related Musculoskeletal Disorders (Q92853434) (← links)
- Estrogens decrease osteoclast number by attenuating mitochondria oxidative phosphorylation and ATP production in early osteoclast precursors (Q97595722) (← links)