Pages that link to "Q30536405"
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The following pages link to Common genetic variants, acting additively, are a major source of risk for autism (Q30536405):
Displaying 50 items.
- Partitioning the heritability of Tourette syndrome and obsessive compulsive disorder reveals differences in genetic architecture (Q21090200) (← links)
- Gluten- and casein-free dietary intervention for autism spectrum conditions (Q21129398) (← links)
- Top 10 Replicated Findings From Behavioral Genetics (Q24288977) (← links)
- The ENIGMA Consortium: large-scale collaborative analyses of neuroimaging and genetic data (Q24289484) (← links)
- A novel computational biostatistics approach implies impaired dephosphorylation of growth factor receptors as associated with severity of autism (Q24612710) (← links)
- The Evolving Diagnostic and Genetic Landscapes of Autism Spectrum Disorder (Q26747333) (← links)
- Genetics of Autism Spectrum Disorder: Current Status and Possible Clinical Applications (Q26771770) (← links)
- Moving from capstones toward cornerstones: successes and challenges in applying systems biology to identify mechanisms of autism spectrum disorders (Q26779153) (← links)
- The developmental transcriptome of the human brain: implications for neurodevelopmental disorders (Q26824947) (← links)
- A genome-wide association study of autism using the Simons Simplex Collection: Does reducing phenotypic heterogeneity in autism increase genetic homogeneity? (Q27326701) (← links)
- Advancing the understanding of autism disease mechanisms through genetics (Q28069757) (← links)
- The role of sex-differential biology in risk for autism spectrum disorder (Q28077103) (← links)
- Synaptic, transcriptional and chromatin genes disrupted in autism (Q28250800) (← links)
- Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs (Q28296286) (← links)
- A comprehensive meta-analysis of common genetic variants in autism spectrum conditions (Q28608366) (← links)
- An Overview of Autism Spectrum Disorder, Heterogeneity and Treatment Options. (Q30235878) (← links)
- Folic acid and autism: What do we know? (Q30252184) (← links)
- Environmental Enrichment Therapy for Autism: Outcomes with Increased Access. (Q30372647) (← links)
- Copy number variation at 22q11.2: from rare variants to common mechanisms of developmental neuropsychiatric disorders (Q30439224) (← links)
- The autism sequencing consortium: large-scale, high-throughput sequencing in autism spectrum disorders (Q30586375) (← links)
- Dopaminergic variants in siblings at high risk for autism: Associations with initiating joint attention (Q30570774) (← links)
- Neural signatures of autism spectrum disorders: insights into brain network dynamics (Q30835950) (← links)
- DAWN: a framework to identify autism genes and subnetworks using gene expression and genetics (Q33592044) (← links)
- Genomic diagnosis for children with intellectual disability and/or developmental delay (Q33737759) (← links)
- A de novo convergence of autism genetics and molecular neuroscience (Q33830989) (← links)
- Prioritization of neurodevelopmental disease genes by discovery of new mutations (Q33830995) (← links)
- Age of onset and family history as indicators of polygenic risk for major depression. (Q33885379) (← links)
- Measuring shared variants in cohorts of discordant siblings with applications to autism (Q33886991) (← links)
- Bio-collections in autism research (Q33892169) (← links)
- Gene × Environment interactions in autism spectrum disorders: role of epigenetic mechanisms. (Q33997473) (← links)
- Most genetic risk for autism resides with common variation (Q34063817) (← links)
- A candidate gene association study further corroborates involvement of contactin genes in autism (Q34299841) (← links)
- Research review: Polygenic methods and their application to psychiatric traits (Q34434004) (← links)
- Increasing our understanding of human cognition through the study of Fragile X Syndrome (Q34435526) (← links)
- Peptide sharing between influenza A H1N1 hemagglutinin and human axon guidance proteins (Q34573339) (← links)
- Analysis of rare, exonic variation amongst subjects with autism spectrum disorders and population controls (Q34672061) (← links)
- The evidence for association of ATP2B2 polymorphisms with autism in Chinese Han population (Q34688669) (← links)
- Integrated model of de novo and inherited genetic variants yields greater power to identify risk genes (Q34961828) (← links)
- Performance comparison of bench-top next generation sequencers using microdroplet PCR-based enrichment for targeted sequencing in patients with autism spectrum disorder (Q34997249) (← links)
- Synergistic interactions between Drosophila orthologues of genes spanned by de novo human CNVs support multiple-hit models of autism (Q35225285) (← links)
- High-resolution chromosome ideogram representation of currently recognized genes for autism spectrum disorders (Q35381777) (← links)
- The female protective effect in autism spectrum disorder is not mediated by a single genetic locus. (Q35599644) (← links)
- Early sex differences are not autism-specific: A Baby Siblings Research Consortium (BSRC) study (Q35684371) (← links)
- A Pooled Genome-Wide Association Study of Asperger Syndrome (Q35692048) (← links)
- Schizophrenia Related Variants in CACNA1C also Confer Risk of Autism. (Q35708361) (← links)
- Genome-Wide Association Study for Autism Spectrum Disorder in Taiwanese Han Population (Q35784947) (← links)
- A Common CYFIP1 Variant at the 15q11.2 Disease Locus Is Associated with Structural Variation at the Language-Related Left Supramarginal Gyrus (Q36062997) (← links)
- Autism spectrum disorders: from genes to neurobiology (Q36101188) (← links)
- The human clinical phenotypes of altered CHRNA7 copy number (Q36144791) (← links)
- Pleiotropic Mechanisms Indicated for Sex Differences in Autism (Q36192641) (← links)