Pages that link to "Q30465622"

The following pages link to Control of neuronal ion channel function by glycogen synthase kinase-3: new prospective for an old kinase (Q30465622):

Displaying 24 items.

• Intraneuronal Aβ accumulation induces hippocampal neuron hyperexcitability through A-type K( ) current inhibition mediated by activation of caspases and GSK-3. (Q27307787) (← links)
• Gsk3 Signalling and Redox Status in Bipolar Disorder: Evidence from Lithium Efficacy (Q28078551) (← links)
• Role of the axonal initial segment in psychiatric disorders: function, dysfunction, and intervention (Q34071914) (← links)
• The Fibroblast Growth Factor signaling pathway (Q34467171) (← links)
• The fibroblast growth factor 14·voltage-gated sodium channel complex is a new target of glycogen synthase kinase 3 (GSK3). (Q34703974) (← links)
• Activation of GSK-3β and caspase-3 occurs in Nigral dopamine neurons during the development of apoptosis activated by a striatal injection of 6-hydroxydopamine (Q34936413) (← links)
• Quantitative proteomics reveals protein-protein interactions with fibroblast growth factor 12 as a component of the voltage-gated sodium channel 1.2 (nav1.2) macromolecular complex in Mammalian brain. (Q35583345) (← links)
• Herpes Simplex Virus type-1 infection induces synaptic dysfunction in cultured cortical neurons via GSK-3 activation and intraneuronal amyloid-β protein accumulation (Q36187501) (← links)
• CK2 activity is required for the interaction of FGF14 with voltage-gated sodium channels and neuronal excitability (Q36913792) (← links)
• Wnt signaling: role in Alzheimer disease and schizophrenia (Q38060562) (← links)
• Wnt and lithium: a common destiny in the therapy of nervous system pathologies? (Q38113139) (← links)
• PPARgamma agonists rescue increased phosphorylation of FGF14 at S226 in the Tg2576 mouse model of Alzheimer's disease. (Q38776236) (← links)
• Modulation of Kv7 channels and excitability in the brain (Q38958968) (← links)
• The Nav1.2 channel is regulated by GSK3. (Q39756006) (← links)
• Glycogen synthase kinase 3 beta alters anxiety-, depression-, and addiction-related behaviors and neuronal activity in the nucleus accumbens shell (Q40362890) (← links)
• Membrane-tethered AKT kinase regulates basal synaptic transmission and early phase LTP expression by modulation of post-synaptic AMPA receptor level. (Q42491700) (← links)
• Hippocampal neurophysiology is modified by a disease-associated C-terminal fragment of tau protein. (Q45927343) (← links)
• Disparate Effects of Lithium and a GSK-3 Inhibitor on Neuronal Oscillatory Activity in Prefrontal Cortex and Hippocampus (Q49333511) (← links)
• GSK3β Modulates Timing-Dependent Long-Term Depression Through Direct Phosphorylation of Kv4.2 Channels. (Q54107591) (← links)
• Juvenile treatment with mGluR2/3 agonist prevents schizophrenia-like phenotypes in adult by acting through GSK3β. (Q54978405) (← links)
• Sex-Specific Proteomic Changes Induced by Genetic Deletion of Fibroblast Growth Factor 14 (FGF14), a Regulator of Neuronal Ion Channels (Q64232177) (← links)
• The Anti-Tumor Agent Sodium Selenate Decreases Methylated PP2A, Increases GSK3βY216 Phosphorylation, Including Tau Disease Epitopes and Reduces Neuronal Excitability in SHSY-5Y Neurons (Q64257020) (← links)
• Differential Dorsolateral Prefrontal Cortex Proteomic Profiles of Suicide Victims with Mood Disorders (Q89976029) (← links)
• Single amyloid-beta injection exacerbates 4-aminopyridine-induced seizures and changes synaptic coupling in the hippocampus (Q92414401) (← links)