Pages that link to "Q30327817"

The following pages link to Amyloid beta-peptide disrupts mitochondrial membrane lipid and protein structure: protective role of tauroursodeoxycholate. (Q30327817):

Displaying 28 items.

• Bile acids in treatment of ocular disease (Q24653326) (← links)
• Bile acids: regulation of apoptosis by ursodeoxycholic acid (Q24653969) (← links)
• The Unexpected Uses of Urso- and Tauroursodeoxycholic Acid in the Treatment of Non-liver Diseases (Q27026087) (← links)
• Melatonin in Alzheimer's disease and other neurodegenerative disorders (Q28217483) (← links)
• Changes in hepatic gene expression upon oral administration of taurine-conjugated ursodeoxycholic acid in ob/ob mice (Q28476015) (← links)
• Role of endocytic inhibitory drugs on internalization of amyloidogenic light chains by cardiac fibroblasts (Q30478679) (← links)
• Unlocking the Door to Neuronal Woes in Alzheimer's Disease: Aβ and Mitochondrial Permeability Transition Pore (Q33664175) (← links)
• Tauroursodeoxycholic acid, a bile acid, is neuroprotective in a transgenic animal model of Huntington's disease. (Q34075502) (← links)
• Tauroursodeoxycholic acid prevents amyloid-beta peptide-induced neuronal death via a phosphatidylinositol 3-kinase-dependent signaling pathway (Q34520873) (← links)
• Tauroursodeoxycholic acid reduces apoptosis and protects against neurological injury after acute hemorrhagic stroke in rats (Q35022852) (← links)
• Hydrophilic bile acids protect human blood-brain barrier endothelial cells from disruption by unconjugated bilirubin: an in vitro study (Q35172521) (← links)
• The use of the R6 transgenic mouse models of Huntington's disease in attempts to develop novel therapeutic strategies (Q36358871) (← links)
• Alzheimer's disease protein Abeta1-42 does not disrupt isolated synaptic vesicles (Q36713885) (← links)
• Natural secretory products of human neural and microvessel endothelial cells: Implications in pathogenic "spreading" and Alzheimer's disease (Q36739417) (← links)
• The mitochondrial permeability transition in neurologic disease (Q36775895) (← links)
• Oxidative stress and the pathogenesis of Alzheimer's disease (Q37101305) (← links)
• Alzheimer’s disease: pathological mechanisms and the beneficial role of melatonin (Q37959556) (← links)
• Emerging Advances in the Management of Cardiac Amyloidosis (Q38587648) (← links)
• Mitochondrial permeability transition pore: a promising target for the treatment of Parkinson's disease. (Q38716156) (← links)
• Misfolded BRICHOS SP-C mutant proteins induce apoptosis via caspase-4- and cytochrome c-related mechanisms (Q40116328) (← links)
• Chemical chaperone TUDCA preserves cone photoreceptors in a mouse model of Leber congenital amaurosis (Q41824260) (← links)
• Cholesterol attenuates the membrane perturbing properties of beta-amyloid peptides (Q44199468) (← links)
• Red cell perturbations by amyloid beta-protein (Q44493150) (← links)
• Lipid peroxidation is not the primary mechanism of bilirubin-induced neurologic dysfunction in jaundiced Gunn rat pups (Q48405759) (← links)
• Alzheimer's disease: soluble oligomeric Abeta(1-40) peptide in membrane mimic environment from solution NMR and circular dichroism studies (Q49110897) (← links)
• Phospholipases A2 mediate amyloid-beta peptide-induced mitochondrial dysfunction. (Q51113387) (← links)
• Translating the biology of aging into novel therapeutics for Alzheimer disease (Q61450425) (← links)
• Oxidative stress in Alzheimer's disease (Q87512835) (← links)