Pages that link to "Q28593244"
Jump to navigation
Jump to search
The following pages link to Genetic analysis of Pten and Tsc2 functional interactions in the mouse reveals asymmetrical haploinsufficiency in tumor suppression (Q28593244):
Displaying 50 items.
- Phosphatase and tensin homolog (Q14878408) (← links)
- tuberin (Q21992503) (← links)
- Ubiquitination regulates PTEN nuclear import and tumor suppression (Q24292946) (← links)
- Turnover of the active fraction of IRS1 involves raptor-mTOR- and S6K1-dependent serine phosphorylation in cell culture models of tuberous sclerosis (Q24670558) (← links)
- Critical role of PICT-1, a tumor suppressor candidate, in phosphatidylinositol 3,4,5-trisphosphate signals and tumorigenic transformation (Q24673801) (← links)
- Role of PI3K, mTOR and Akt2 signalling in hepatic tumorigenesis via the control of PKM2 expression (Q28294651) (← links)
- Feedback inhibition of Akt signaling limits the growth of tumors lacking Tsc2 (Q28509303) (← links)
- mTOR and cancer: insights into a complex relationship (Q29617779) (← links)
- Inhibition of mTORC1 leads to MAPK pathway activation through a PI3K-dependent feedback loop in human cancer (Q29618030) (← links)
- Novel interactions between actin and the proteasome revealed by complex haploinsufficiency (Q30430569) (← links)
- mTOR and cancer: many loops in one pathway. (Q33348248) (← links)
- Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling (Q33619056) (← links)
- Simultaneous haploinsufficiency of Pten and Trp53 tumor suppressor genes accelerates tumorigenesis in a mouse model of prostate cancer. (Q33687282) (← links)
- Compound haploinsufficiencies of Ebf1 and Runx1 genes impede B cell lineage progression (Q33842740) (← links)
- S100B and neurofibromin immunostaining and X-inactivation patterns of laser-microdissected cells indicate a multicellular origin of some NF1-associated neurofibromas (Q33933299) (← links)
- Linking genetics to structural biology: complex heterozygosity screening with actin alanine scan alleles identifies functionally related surfaces on yeast actin (Q34042411) (← links)
- Molecular genetics of prostate cancer: new prospects for old challenges (Q34127462) (← links)
- Akt-dependent activation of mTORC1 complex involves phosphorylation of mTOR (mammalian target of rapamycin) by IκB kinase α (IKKα). (Q34139142) (← links)
- TSC2 deficiency increases PTEN via HIF1alpha (Q34658958) (← links)
- Tuberous Sclerosis Complex 1: An Epithelial Tumor Suppressor Essential to Prevent Spontaneous Prostate Cancer in Aged Mice (Q34724225) (← links)
- The PTEN-PI3K pathway: of feedbacks and cross-talks (Q34828662) (← links)
- Pancreatic endocrine tumors: expression profiling evidences a role for AKT-mTOR pathway (Q34887795) (← links)
- Subtle variations in Pten dose determine cancer susceptibility (Q35055350) (← links)
- Determinants of sensitivity and resistance to rapamycin-chemotherapy drug combinations in vivo (Q36100451) (← links)
- Igf2 ligand dependency of Pten /− developmental and tumour phenotypes in the mouse (Q36166920) (← links)
- Tumor suppressor genetics (Q36252762) (← links)
- Akt-regulated pathways in prostate cancer (Q36313185) (← links)
- Beyond PTEN mutations: the PI3K pathway as an integrator of multiple inputs during tumorigenesis (Q36384467) (← links)
- Differential IKK/NF-κB Activity Is Mediated by TSC2 through mTORC1 in PTEN-Null Prostate Cancer and Tuberous Sclerosis Complex Tumor Cells (Q36402424) (← links)
- FOXO-regulated transcription restricts overgrowth of Tsc mutant organs (Q36492071) (← links)
- Aberrant Rheb-mediated mTORC1 activation and Pten haploinsufficiency are cooperative oncogenic events (Q36843531) (← links)
- Tumorigenic activity and therapeutic inhibition of Rheb GTPase (Q36843535) (← links)
- Identification of structural aberrations in cancer by SNP array analysis (Q36897470) (← links)
- Possible mechanisms of disease development in tuberous sclerosis (Q37051667) (← links)
- PTEN deficiency is fully penetrant for prostate adenocarcinoma in C57BL/6 mice via mTOR-dependent growth (Q37164248) (← links)
- PTEN and the PI3-kinase pathway in cancer (Q37259680) (← links)
- The role of PTEN signaling perturbations in cancer and in targeted therapy (Q37270446) (← links)
- Deconstructing feedback-signaling networks to improve anticancer therapy with mTORC1 inhibitors (Q37355543) (← links)
- The Par-4/PTEN connection in tumor suppression (Q37559086) (← links)
- The pathologist's slide reveals more than meets the eye: loss of heterozygosity and cancer biology (Q37791305) (← links)
- Haplo-insufficiency: a driving force in cancer (Q37815124) (← links)
- The Role of Phosphatidylinositol 3-Kinase Signaling Pathways in Pancreatic Cancer (Q37882389) (← links)
- Mouse hospital and co-clinical trial project--from bench to bedside. (Q38431277) (← links)
- Modeling complex genetic interactions in a simple eukaryotic genome: actin displays a rich spectrum of complex haploinsufficiencies (Q38517169) (← links)
- PTEN is destabilized by phosphorylation on Thr366. (Q40143795) (← links)
- BRSK2 induced by nutrient deprivation promotes Akt activity in pancreatic cancer via downregulation of mTOR activity. (Q41280277) (← links)
- Mesenchymal Tumorigenesis Driven by TSC2 Haploinsufficiency Requires HMGA2 and Is Independent of mTOR Pathway Activation. (Q41365464) (← links)
- Constitutively active Akt1 expression in mouse pancreas requires S6 kinase 1 for insulinoma formation (Q41891152) (← links)
- Pml represses tumour progression through inhibition of mTOR. (Q42774421) (← links)
- Analysis of protein-protein interactions and proteomic profiles of normal human lenses (Q42992165) (← links)