Pages that link to "Q28589880"

The following pages link to Neural network of structures in which GABAB receptors regulate absence seizures in the lethargic (lh/lh) mouse model (Q28589880):

Displaying 27 items.

• Calcium channel, voltage-dependent, beta 4 subunit (Q21497160) (← links)
• A brief history on the oscillating roles of thalamus and cortex in absence seizures (Q26823638) (← links)
• Decreased (45)Ca(2)( ) uptake in P/Q-type calcium channels in homozygous lethargic (Cacnb4lh) mice is associated with increased beta3 and decreased beta4 calcium channel subunit mRNA expression (Q28504580) (← links)
• Characterization of absence seizure-dependent cyclic AMP responsive element-and activator protein 1 DNA-binding activities in lethargic (lh/lh) mice (Q28505098) (← links)
• A targeted mutation in Cacng4 exacerbates spike-wave seizures in stargazer (Cacng2) mice (Q28511539) (← links)
• Paroxysmal dyskinesias in the lethargic mouse mutant (Q28587488) (← links)
• Ducky mouse phenotype of epilepsy and ataxia is associated with mutations in the Cacna2d2 gene and decreased calcium channel current in cerebellar Purkinje cells (Q28591132) (← links)
• Childhood absence epilepsy: genes, channels, neurons and networks. (Q30835420) (← links)
• Repeated administration of CGP 46381, a gamma-aminobutyric acidB antagonist, and ethosuximide suppresses seizure-associated cyclic adenosine 3'5' monophosphate response element- and activator protein-1 DNA-binding activities in lethargic (lh/lh) mic (Q31804033) (← links)
• Contribution of GABA(A) and GABA(B) receptors to thalamic neuronal activity during spontaneous absence seizures in rats. (Q31840236) (← links)
• GABABR1 receptor protein expression in human mesial temporal cortex: changes in temporal lobe epilepsy (Q34138555) (← links)
• The interface of preclinical evaluation with clinical testing of antiepileptic drugs: role of pharmacogenomics and pharmacogenetics (Q34768788) (← links)
• Neurochemical and behavioral features in genetic absence epilepsy and in acutely induced absence seizures (Q36877182) (← links)
• Maternal epileptic seizure induced by pentylenetetrazol: apoptotic neurodegeneration and decreased GABAB1 receptor expression in prenatal rat brain (Q37252981) (← links)
• A thalamo-hippocampal-ventral tegmental area loop may produce the positive feedback that underlies the psychotic break in schizophrenia (Q37765676) (← links)
• The Role of Calcium Channels in Epilepsy (Q38686593) (← links)
• The role of GABAB mechanisms in animal models of absence seizures (Q41189029) (← links)
• Altered expression of GABAB receptors in the hippocampus after kainic-acid-induced seizures in rats (Q42441293) (← links)
• Targeting thalamic nuclei is not sufficient for the full anti-absence action of ethosuximide in a rat model of absence epilepsy. (Q44499237) (← links)
• Treatment of spontaneous and chemically induced EEG paroxysms in the Fischer-344 rat with traditional antiepileptic drugs or AED CGP 35348 polytherapy (Q44551788) (← links)
• Development of a new genetic model for absence epilepsy: spike-wave seizures in C3H/He and backcross mice. (Q46412714) (← links)
• Age-related relationship between mRNA expression of GABA(B) receptors and calcium channel beta4 subunits in cacnb4lh mice (Q48161861) (← links)
• Pathophysiological mechanisms of genetic absence epilepsy in the rat. (Q48461890) (← links)
• Opposite effects of GABAB receptor antagonists on absences and convulsive seizures (Q48637716) (← links)
• Phenotypic heterogeneity in the stargazin allelic series. (Q52551602) (← links)
• The gene encoding GABBR1 is not associated with childhood absence epilepsy in the Chinese Han population (Q73443703) (← links)
• Effects of compounds acting on GABA(B) receptors in the pentylenetetrazole kindling model of epilepsy in mice (Q74238283) (← links)